675 results on '"Dmitrovsky, Ethan"'
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2. The Influence of Body Mass Index on Overall Survival Following Surgical Resection of Non–Small Cell Lung Cancer
3. Retinoids and rexinoids in cancer prevention: from laboratory to clinic
4. REPLY TO OEGEMA ET AL. : CFI-400945 and Polo-like kinase 4 inhibition
5. Polo-like kinase 4 inhibition produces polyploidy and apoptotic death of lung cancers
6. Human Teratocarcinomas and Retinoic Acid-Mediated Tumor Differentiation
7. Retinoids
8. Supplementary Figure S4 from Real-world Studies Link NSAID Use to Improved Overall Lung Cancer Survival
9. Data from Real-world Studies Link NSAID Use to Improved Overall Lung Cancer Survival
10. Supplementary Table S3 from Real-world Studies Link NSAID Use to Improved Overall Lung Cancer Survival
11. Abstract 1836: Combining a novel retinoic acid receptor-γ agonist with immune checkpoint blockade represses lung cancer growth in vivo
12. Supplementary table from A Novel CDK2/9 Inhibitor CYC065 Causes Anaphase Catastrophe and Represses Proliferation, Tumorigenesis, and Metastasis in Aneuploid Cancers
13. Data from Bexarotene Plus Erlotinib Suppress Lung Carcinogenesis Independent of KRAS Mutations in Two Clinical Trials and Transgenic Models
14. Supplementary Table S1 from CD38-Mediated Immunosuppression as a Mechanism of Tumor Cell Escape from PD-1/PD-L1 Blockade
15. Supplementary Figures 1-6 from The Ubiquitin-Specific Peptidase USP18 Promotes Lipolysis, Fatty Acid Oxidation, and Lung Cancer Growth
16. Data from Specific CP110 Phosphorylation Sites Mediate Anaphase Catastrophe after CDK2 Inhibition: Evidence for Cooperation with USP33 Knockdown
17. supplementary figure and legend from Dinaciclib Induces Anaphase Catastrophe in Lung Cancer Cells via Inhibition of Cyclin-Dependent Kinases 1 and 2
18. Supplemental Fig. S3 from Specific CP110 Phosphorylation Sites Mediate Anaphase Catastrophe after CDK2 Inhibition: Evidence for Cooperation with USP33 Knockdown
19. Data from CD38-Mediated Immunosuppression as a Mechanism of Tumor Cell Escape from PD-1/PD-L1 Blockade
20. Data from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer
21. Supplemental Table S1 from Specific CP110 Phosphorylation Sites Mediate Anaphase Catastrophe after CDK2 Inhibition: Evidence for Cooperation with USP33 Knockdown
22. Supplemental Figures 1-10 from Deubiquitinase USP18 Loss Mislocalizes and Destabilizes KRAS in Lung Cancer
23. Supplementary Text and Legends from Deubiquitinase USP18 Loss Mislocalizes and Destabilizes KRAS in Lung Cancer
24. Video-Supplementary Fig. 2a from Dinaciclib Induces Anaphase Catastrophe in Lung Cancer Cells via Inhibition of Cyclin-Dependent Kinases 1 and 2
25. Supplementary Figure 1 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
26. Supplementary Figure S1 from Bexarotene Plus Erlotinib Suppress Lung Carcinogenesis Independent of KRAS Mutations in Two Clinical Trials and Transgenic Models
27. Supplementary Methods; Figures S1 - S22; Tables S2, S6, S9 - S12 from CD38-Mediated Immunosuppression as a Mechanism of Tumor Cell Escape from PD-1/PD-L1 Blockade
28. Data from A Novel CDK2/9 Inhibitor CYC065 Causes Anaphase Catastrophe and Represses Proliferation, Tumorigenesis, and Metastasis in Aneuploid Cancers
29. Supplementary Figure 5 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
30. Supplementary Figure 2 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer
31. Supplementary Fig. 2. from Dinaciclib Induces Anaphase Catastrophe in Lung Cancer Cells via Inhibition of Cyclin-Dependent Kinases 1 and 2
32. Data from Tyrosine Threonine Kinase Inhibition Eliminates Lung Cancers by Augmenting Apoptosis and Polyploidy
33. Supplementary Figure 1 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer
34. Data from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
35. Supplementary Figure 4 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
36. Data from The Ubiquitin-Specific Peptidase USP18 Promotes Lipolysis, Fatty Acid Oxidation, and Lung Cancer Growth
37. Supplementary Figure Legend from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
38. Data from Deubiquitinase USP18 Loss Mislocalizes and Destabilizes KRAS in Lung Cancer
39. Data from Dinaciclib Induces Anaphase Catastrophe in Lung Cancer Cells via Inhibition of Cyclin-Dependent Kinases 1 and 2
40. Supplementary Figure 6 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
41. Supplementary Figure 7 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
42. Figures S1-S6 from Tyrosine Threonine Kinase Inhibition Eliminates Lung Cancers by Augmenting Apoptosis and Polyploidy
43. Supplementary Figure 3 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
44. Supplementary figures from A Novel CDK2/9 Inhibitor CYC065 Causes Anaphase Catastrophe and Represses Proliferation, Tumorigenesis, and Metastasis in Aneuploid Cancers
45. Supplementary Figure Legend from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer
46. Supplementary Figure 3 from Comparing Histone Deacetylase Inhibitor Responses in Genetically Engineered Mouse Lung Cancer Models and a Window of Opportunity Trial in Patients with Lung Cancer
47. Supplementary Figure 2 from Evidence for the Ubiquitin Protease UBP43 as an Antineoplastic Target
48. UBE1L Is a Retinoid Target That Triggers PML/RARα Degradation and Apoptosis in Acute Promyelocytic Leukemia
49. Interpreting Patterns of Gene Expression with Self-Organizing Maps: Methods and Application to Hematopoietic Differentiation
50. FIELD REPORT: GETTING NEW FACULTY INTO THE FOLD-FAST
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