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1. CYP19A1 fine-mapping and Mendelian randomization: estradiol is causal for endometrial cancer

2. Meta-analysis of genome-wide association studies identifies common susceptibility polymorphisms for colorectal and endometrial cancer near SH2B3 and TSHZ1.

3. Fine-mapping of the HNF1B multicancer locus identifies candidate variants that mediate endometrial cancer risk

4. Candidate locus analysis of the TERT–CLPTM1L cancer risk region on chromosome 5p15 identifies multiple independent variants associated with endometrial cancer risk

6. Supplementary Table 4. Association of 47 waist-hip ratio (WHR) SNPs with endometrial cancer risk from Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

7. Supplementary Table 3: Association of 77 body mass index (BMI) SNPs with endometrial cancer risk and BMI in the endometrial cancer dataset from Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

8. Figure S4 from Amplification of 1q32.1 Refines the Molecular Classification of Endometrial Carcinoma

9. Supplemental Table 1. Details of cases and controls included in the endometrial cancer analyses from Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

10. Supplemental Table 2. Average BMIs in the ANECS, SEARCH and iCOGS endometrial cancer datasets. from Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

11. Supplementary Text for: Genetic risk score Mendelian randomization shows that obesity measured as body mass index, but not waist:hip ratio, is causal for endometrial cancer from Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

15. Copy number load predicts outcome of metastatic colorectal cancer patients receiving bevacizumab combination therapy

17. The tumor suppressor protein PTEN undergoes amyloid-like aggregation in tumor cells

18. Identification of nine new susceptibility loci for endometrial cancer.

19. Genetic overlap between endometriosis and endometrial cancer: Evidence from cross-disease genetic correlation and GWAS meta-analyses

20. Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

21. Identification of nine new susceptibility loci for endometrial cancer

22. Somatic POLE exonuclease domain mutations are early events in sporadic endometrial and colorectal carcinogenesis, determining driver mutational landscape, clonal neoantigen burden and immune response

23. Ischemia-Induced DNA Hypermethylation during Kidney Transplant Predicts Chronic Allograft Injury

24. Genetic overlap between endometriosis and endometrial cancer: evidence from cross‐disease genetic correlation and GWAS meta‐analyses

25. Amplification of 1q32.1 Refines the Molecular Classification of Endometrial Carcinoma

26. Integrated genome analysis of uterine leiomyosarcoma to identify novel driver genes and targetable pathways

27. Prognostic Significance of POLE Proofreading Mutations in Endometrial Cancer

28. CYP19A1 fine-mapping and Mendelian randomization

29. SO009ISCHEMIA IN KIDNEY TRANSPLANTATION CAUSES DNA HYPERMETHYLATION AND PREDICTS GRAFT FIBROSIS: AN EPIGENOME-WIDE ASSOCIATION STUDY

30. Comprehensive genetic assessment of the ESR1 locus identifies a risk region for endometrial cancer

31. Candidate locus analysis of the TERT–CLPTM1L cancer risk region on chromosome 5p15 identifies multiple independent variants associated with endometrial cancer risk

32. Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

34. Successful application of endoscopic ultrasound-guided fine needle biopsy to establish pancreatic patient-derived tumor xenografts: a pilot study

36. Characterization of patient-derived tumor xenograft models of endometrial cancer for preclinical evaluation of targeted therapies

37. Meta-analysis of genome-wide association studies identifies common susceptibility polymorphisms for colorectal and endometrial cancer near SH2B3 and TSHZ1

38. Prognostic significance of POLE proofreading mutations in endometrial cancer

39. CYP19A1 fine-mapping and Mendelian randomization: estradiol is causal for endometrial cancer

40. Comprehensive genetic assessment of the ESR1 locus identifies a risk region for endometrial cancer

41. Microsatellite Instable and Microsatellite Stable Primary Endometrial Carcinoma Cells and Their Subcutaneous and Orthotopic Xenografts Recapitulate the Characteristics of the Corresponding Primary Tumor

42. Prognostic Significance of POLE Proofreading Mutations in Endometrial Cancer

43. Successful application of endoscopic ultrasoundguided fine needle biopsy to establish pancreatic patient-derived tumor xenografts: a pilot study.

45. Candidate locus analysis of the TERT- CLPTM1L cancer risk region on chromosome 5p15 identifies multiple independent variants associated with endometrial cancer risk.

46. Five endometrial cancer risk loci identified through genome-wide association analysis

48. Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer

49. Genetic overlap between endometriosis and endometrial cancer: evidence from cross-disease genetic correlation and GWAS meta-analyses

50. Prognostic Significance of POLE Proofreading Mutations in Endometrial Cancer

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