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Your search keyword '"Denise Locatelli"' showing total 17 results

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1. SMA-miRs (miR-181a-5p, -324-5p, and -451a) are overexpressed in spinal muscular atrophy skeletal muscle and serum samples

2. Continuous neurodegeneration and death pathway activation in neurons and glia in an experimental model of severe chronic epilepsy

3. Axon outgrowth and neuronal differentiation defects after a-SMN and FL-SMN silencing in primary hippocampal cultures.

4. Different Stability and Proteasome-Mediated Degradation Rate of SMN Protein Isoforms.

5. Progressive brain damage, synaptic reorganization and NMDA activation in a model of epileptogenic cortical dysplasia.

6. Selective vulnerability of spinal and cortical motor neuron subpopulations in delta7 SMA mice.

7. SMA-miRs (miR-181a-5p, -324-5p, and -451a) are overexpressed in spinal muscular atrophy skeletal muscle and serum samples

8. Targeting PSD95-nNOS interaction by Tat-N-dimer peptide during status epilepticus is neuroprotective in MAM-pilocarpine rat model

9. Axon outgrowth and neuronal differentiation defects after a-SMN and FL-SMN silencing in primary hippocampal cultures

10. Long-duration epilepsy affects cell morphology and glutamatergic synapses in type IIB focal cortical dysplasia

11. Spinal muscular atrophy pathogenic mutations impair the axonogenic properties of axonal-survival of motor neuron

12. Status epilepticus-induced pathologic plasticity in a rat model of focal cortical dysplasia

13. Different Stability and Proteasome-Mediated Degradation Rate of SMN Protein Isoforms

14. Selective Vulnerability of Spinal and Cortical Motor Neuron Subpopulations in delta7 SMA Mice

15. Human axonal survival of motor neuron (a-SMN) protein stimulates axon growth, cell motility, C-C motif ligand 2 (CCL2), and insulin-like growth factor-1 (IGF1) production

16. Axonal-SMN (a-SMN), a protein isoform of the survival motor neuron gene, is specifically involved in axonogenesis

17. PROGRESSIVE BRAIN DAMAGE, SYNAPTIC REORGANIZATION AND NMDA ACTIVATION IN A MODEL OF EPILEPTOGENIC CORTICAL DYSPLASIA

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