88 results on '"De Recondo J"'
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2. Malignant sellar, parasellar and skull base tumors
- Author
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Grob, R., Chantelard, J.-V., Antoine, E.-Ch., de Recondo, J., Charbonnel, B., Khayat, D., Dupas, B., Mussini, J.-M., Gayet-Delacroix, M., Buthiau, D., Buthiau, Didier, editor, and Khayat, David, editor
- Published
- 1998
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3. Fourth meeting of the European Neurological Society 25–29 June 1994 Barcelona, Spain: Abstracts of Symposia and free communications
- Author
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Harms, L., Bock, A., JÄnisch, W., Valdueza, J., Weber, J., Link, I., De Keyser, J., Goossens, A., Wilczak, N., Vedeler, C., Bjorge, L., Uvestad, E., Conti, G., Williams, K., Ginsberg, L., Rafique, S., Rapoport, S. I., Gershfeld, N. L., De La Meilleure, G., Crevits, L., Faiss, J. H., Heye, N., Blanke, J., Sackmann, A., Kastrup, O., Doornbos, R., van der Worp, H. B., Kappelle, L. J., Bar, P. R., Davie, C. A., Barker, G. J., Brenton, D., Miller, D. H., Thompson, A. J., Block, F., Schwarz, M., Delodovici, L., Baruzzi, F., Bonaldi, G., Dario, A., Marra, A., Mercuri, A., Dworzak, F., Cavallari, P., Confalonieri, P., Zuffi, M., Antozzi, C., Cornelio, F., Baldissera, F., Chassande, B., Ameri, A., Eymard, B., Poisson, M., Vérier, A., Brunet, P., Congia, S., Murgia, P. L., Cannas, A., Borghero, G., Uselli, S., Mellino, G., Ferrai, R., Lampis, R., Massa, R., Muzzetto, B., Giannini, F., Rossi, S., Cioni, R., d'Aniello, C., Guarneri, A., Battistini, N., Ceriani, F., Del Santo, A., Poloni, M., Campo, J. F., Iglesias, F., Guitera, M. V., Farinas, C., Pascual, J., Leno, C., Berciano, J., Thorpe, I. W., Kendall, B. E., McDonald, W. I., Moulignier, A., Dromer, F., Baudrimont, M., Dupont, B., Gozlan, J., El Amrani, M., Petit, J. C., Roullet, E., Sterzi, R., Causaran, R., Protti, A., Riva, M., Erminio, F., Arena, O., Villa, F., Maccagnano, E., Miletta, M., Spinelli, F., Ben-Hur, T., Weidenfeldl, J., Rao, N. S., Chari, C. C., Laforet, P., Matheron, S., Adams, D., Chemouilli, Ph., Desi, M., Said, G., Davous, P., Lionnet, F., Pulik, M., Genet, P., Rozenberg, F., Cartier, L. M., Castillo, J. L., Cea, J. G., Villagra, R., de Saint Martin, L., Mahieux, F., Manifacier, M. J., Mattos, K., Queiros, C., Publio, L., Vinhas, V., PeÇanha-Martins, A. C., Melo, A., Liska, U., Zifko, U., Budka, H., Drlicek, M., Grisold, W., Kaufmann, R., Kaiser, R., Czygan, M., Gomes, I., Jones, N., Cunha, S., EmbiruÇu, E. Katiane, Vieira, V., Araujo, I., Alexandra, M., Ferreira, A., Goes, J., Chemouilli, P., Israel-Biet, Masson, H., Lacroix, C., Gasnault, J., Hildebrandt-Müller, B., Oschmann, P., Krack, P., Willems, W. R., Dorndorf, W., Freitas, V., Bittencourt, A., Fernandes, D., Nascimento, M. H., Severo, M., Moraes, D., Muller, M., Hasert, K., Merkelbach, S., Schimrigk, K., van Oosten, B. W., Lai, M., Polman, C. H., Bertelsmann, F. W., Hodgkinson, S., Cabre, P. H., Volpe, L., Smadja, D., Vernant, J. P., Villaroya, H., Violleau, K., Younes-Chennoufi, A. Ben, Baumann, N., Villanueva-Hemandez, P., Ballabriga, J., Basart, E., Arbizu, T. X., Perez-Serra, J., Vinuels, F., Giron, J. M., Castilla, J. M., Redondo, L., Izquierdo, G., Lauer, K., Henneberg, A., Bittmann, N., Link, D., Wollinsky, K. H., Mobner, R., Fassbender, K., Kuhnen, J., Schwartz, A., Hennerici, M., Miller, A., Lider, O., Abramsky, O., Weiner, H. L., Offner, H., Vanderbark, A. A., Paoino, E., Fainardi, E., Addonizio, M. C., Ruppi, P., Tola, M. R., Granieri, E., Carreras, M., Sazdovitch, V., Joutel, A., Verdier-taillefer, M. H., Heinzlef, O., Radder, C., Tournier-Lasserve, E., Brenner, R. E., Munro, P. M. G., Williams, S. C. R., Bell, J. D., Hawkins, C. P., Filippi, M., Campi, A., Dousset, V., Canal, N., Comi, G., Zhu, J., Weber, F., Retska, R., List, J., Zhang, L., Brock, M., Taphoorn, M. J. B., Heimans, J. J., van der Veen, E. A., Karim, A. B. M. F., Sarazin, M., Argentino, N., Delattre, J. Y., Derkinderen, P., Buchwald, B., Schroter, G., Serve, G., Franke, C. H., Conrad, B., Kitchen, N. D., Thomas, D. G. T., Forman, A. D., Ang, Kie- Kian, Price, R., Stephens, C., Salmaggi, A., Nermni, R., Silvani, A., Forno, M. G., Luksch, R., Boiardi, A., Grzelec, H., Fryze, C., Nowacki, P., Zdziarska, B., Sanson, M., Merel, P., Richard, S., Rouleau, G., Thomas, G., Olsen, N. K., Pfeiffer, P., Egund, N., Bentzen, S. M., Johannesen, L., Mondrup, K., Rose, C., Zyluk, B., Wondrusch, E., Berger, O., Fast, N., Jellinger, K., Lindner, K., Urman, A., Thibault, J. L., Duyckaerts, Ch., Strik, H., Muller, B., Richter, E., Krauseneck, P., Steinbrecher, A., Schabet, M., Hess, C., Bamberg, M., Dichgans, J., Counsell, C. E., McLeod, M., Grant, R., Creel, G. B., Claus, D., Sieber, E., Engelhardt, A., Rechlin, T., Thierauf, P., Neubauer, U., Peresson, M., Di Giovacchino, G., Romani, G. L., Di Silverio, F., Danek, A., Kuffner, M., Hoermann, R., Schopohl, J., Laska, M., Heye, B., Zangaladze, A. T., Valls-SoIè, J., Cammarota, A., Alvarez, R., Tolosa, E., Hallett, M., Ulbricht, D., Ganslandt, O., Kober, H., Vieth, J., Grummich, P., Pongratz, H., Brigel, C., Fahlbusch, R., Serra, F. P., Palma, V., Nolfe, G., Buscaino, G. A., Rothstein, T. L., Gibson J. M., Morrison P. M., Collins A. D., Eiselt, M., Wagnur, H., Zwiener, U., Schindler, T., Efendi, H., Ertekin, C., Erfas, M., Larsson, L. E., Sirin, H., AraÇ, N., Toygar, A., Demir, Y., Seddigh, S., Vogt, T. H., Hundemer, H., Visbeck, A., Pastena, L., Faralli, F., Mainardi, G., Gagliardi, R., Linden, D., Berlit, P., Lopez, O. L., Becker, J. T., Jungreis, C., Brenner, R., Rezek, D., Dekesky, S. T., Estol, C., Boller, F., Fernandez, J. M., Mederer, S., Batlle, J., Turon, A., Codina, A., Hitzenberger, P., Vila, N., Valls-SolÇ, J., Chamorro, A., Pouget, J., Schmied, A., Morin, D., Azulay, J. Ph., Vedel, J. P., Montalt, J., Escudero, J., Barona, R., Campos, A., Varli, K., Ertem, E., Uludag, B., Yagiz, A., Privorkin, Z., Steinvil, Y., Kott, E., Combarros, O., Sanchez-Pernaute, R., Orizaola, P., Mokrusch, Th., Kutluaye, E., Selcuki, D., Ertikin, C., Zettl, U., Gold, R., Harvey, G. K., Hartung, H. P., Toyka, K. V., Wokke, J. H. J., Oey, P. L., Ippel, P. F., Jansen, G. H., Franssen, H., Toyooka, K., Fujimura, H., Ueno, S., Yoshikawa, H., Yorifuji, S., Yanagihara, T., Talamon, C., Tzourio, C., Kiefer, R., Jung, S., Toyka, K., Ruolt, I., Tranchant, C., Mohr, M., Warter, J. M., Younger, D. S., Rosoklija, G., Hays, A. P., Kurita, R., Hasegawa, O., Matsumto, M., Komiyama, A., Nara, Y., Oueslati, S., Belal, S., Turki, I., Ben Hamida, C., Hentati, F., Ben Hamida, M., Kwiecinski, H., Krolicki, L., Domzal-Stryga, A., Dellemijn, P. L. I., van Deventer, P., van Moll, B., Drogendijk, T., Vecht, Ch. J., Nemni S., Amadio, Fazio, R., Galardin, G., Delodovici, M. L., Peghi, E., Monticelli, M. L., Sessa, A., Viguera, M. L., Palomar, M., Gamez, J., Cervera, C., Navarro, C., Serena, J., Duran, I., Fernandez, A. L., Comabella, M., Nos, C., Rio, J., Montalban, J., Navarro, X., Verdu, E., Darbra, S., Buti, M., Mrabet, A., Fredj, M., Gouider, R., Tounsi, H., Khalfallah, N., Haddad, A., Dbaiss, T., Ghnassia, R., Rouillet, E., Chedru, F., Porsche, H., Strenge, H., Li, S. W., Young, Y. P., Garcia, A. A., Baron, P., Scarpini, E., Bianchi, R., Conti, A., Livraghi, S., Rees, J. H., Gregson, N. A., Hughes, R. A. C., Sedano, M. J., Calleja, J., Canga, E., Bahou, Y., Biary, N., Al Deeb, S. M., Guern, E. L. E., Gugenheim, M., Tardieu, S., Aisonobe, T. M., Agid, Y., Bouche, P., Brice, A., Rautenstrauss, B., Nelis, E., Grehl, H., Van Broeckhoven, C., Pfeiffer, R. A., Liehr, T., Ganzmann, E., Gehring, C., Neundörfer, B., Geremia, L., Doronzo, R., Sacilotto, G., Sergi, P., Pastorino, G. C., Scarlato, G., Planté-Bordeneuve, V., Mantel, A., Baas, F., Moser, H., Antonini, A., Psylla, M., Günther, I., Vontobell, P., Beer, H. F., Leenders, K. L., Chaudhuri, K. Ray, Parker, J., Pye, I. F., Millac, P. A. H., Abbott, R. J., Sutter, M., Albani, C., de Rijk, M. C., Breteler, M. M. B., Graveland, G. A., van der Mechè, F. G. A., Hofman, A., Keipes, M., Hilger, Ch., Diederich, N., Metz, H., Hentges, F., Pollak, P., Benabid, A. L., Limousin, P., Hoffmann, D., Benazzouz, A., Perret, J., Laihinen, A., Rinne, J. O., Ruottinen, H., Nagren, K., Lehikoinen, P., Oikonen, V., Ruotsalainen, U., Rinne, U. K., Cocozza, S., Pizzuti, A., Cavalcanti, F., Monticelli, A., Pianese, L., Redolfi, E., Paiau, F., Di Donato, S., Pandolfo, M., Palau, F., Monros, E., De Michele, G., Smeyers, P., Lopez-ArLandis, J., Uilchez, J., Filla, A., Genis, D., Matilla, T., Volpini, V., Blanchs, M. I., Davalos, A., Molins, A., Rosell, J., Estivill, X., De Jonghe, P., Smeyers, G., Krols, L., Mercelis, R., Hazan, J., Weissenbach, J., Martin, J. J., Warner, T. A. T., Williams, L., Orb, A. S., Harding, A. E., Giunti, P., Sweeney, M. G., Spadaro, M., Jodice, C., Novelletto, A., Malaspina, P., Frontali, M., Salmon, E., Gregoire, Del Fiore, Comar, Franck, G., Scheltens, P. H., Siegfried, K., Dartigues, E., De Deyn, P., Horn, R., Nelson, I., Hanna, M. G., Morgan-Hughes, J. A., Collinge, J., Palmer, M. S., Campbell, T., Mahal, S., Sidle, K., Humphreys, C., Tavitian, B., Pappata, S., Jobert, A., Crouzel, A. M., DiGiamberardino, L., Steimetz, G., Barbanti, P., Fabbrini, G., Salvatore, M., Buzzi, M. G., Di Piero, V., Petraroli, R., Sbriccoli, A., Pocchiari, M., Macchi, G., Lenzi, G. L., Spiegel, R., Maguire, P., Schmid, W., Ott, A., Bots, M. L., Grobbe, D. E., Hofman, A., Howard, R. S., Russell, S., Losseff, N., Hirsch, N. P., Couderc, R., Bailleul, S., Nargeot, M. C., Touchon, J., Picot, M. C., Rizzo, M., Watson, G., McGehee, D., Dingus, T., Kappos, L., Radü, E. W., Haas, J., Hartard, C. H., Spuler, S., Yousry, T., Voltz, R., Scheller, A., Holler, E., Hohlfeld, R., Scolding, N. J., Sussman, J., Kolar, O. J., Farlow, M. R., Rice, P. H., Zipp, F., Sotgiu, S., Weiss, E. H., Wekerle, H., Chalmers, R., Robertson, N., Compston, D. A. S., Martino, G., Clementi, E., Brambilla, E., Moiola, L., Martinelli, V., Colombo, B., Poggi, A., Rovaris, M., Grimaldi, L. M. E., Roth, M. P., Descoins, P., Ballivet, S., Ruidavets, J. B., Waubant, E., Nogueira, L., Cambon-Thomsen, A., Clanet, M., Leppert, D., Hauser, S., Lugaresi, A., Tartaro, A., D'aurelio, P., Befalo, L. L. O., Thomas, A., Malatesta, G., Gambi, D., Benedikz, J. E. G., Magnusson, H., Poser, C. M., Guomundsson, G., Bates, T. E., Davies, S. E. C., Clark, J. B., Landon, D. N., ùther, J. R., Rautenberg, W., Overgaard, K., Sereghy, T., Pedersen, H., Boysen, G., Diez-Tejedor, E., Carceller, F., Gutierrez, M., Lopez-Pajares, R., Roda, J. M., Chandra, B., Ricart, W., Gonzalez-Huix, F., Molina, A., Rundek, T., Demarin, V., De Reuck, J., Boon, P., Decoq, D., Strijckmans, K., Goethals, P., Lemahieu, I., Nibbio, A., Chabriat, H., Vahedi, K., Nagy, T., Verin, M., Mas, J. L., Julien, J., Ducrocq, X., Iba-Zizen, M. T., Cabanis, E. A., Bousser, M. G., Rolland, Y., Landgraf, F., Bompais, B., Lemaitre, M. H., Edan, G., Vorstrup, S., Knudsen, L., Olsen, K. Skovgaard, Videbaek, C., Schroeder, T., van Gijn, J., Jansen, H. M. L., Pruim, J., Paans, A. M. J., Willemsen, A. T. M., Hew, J. M., vd Vliet, A. M., Haaxma, R., Vaalburg, W., Minderhoud, J. M., Korf, J., Soudain, S. E., Ho, T. W., Mishu, B., Li, C. Y., Nachainkin, I., Gao, C. Y., Cornblath, D. R., Griffin, J. W., Asbury, A. K., Blaser, M. J., McKhann, G. M., Ho, T., Macko, C., Xue, P., Stadlan, E. M., Ramos-Alvarez, M., Valenciano, L., Visser, L. H., van der Meché, F. G. A., van Darn, P. A., Meulstee, J., Schmitz, P. I. M., Jacobs, B., Oomes, P. G., Kleyweg, R. P., Jacobs, B. C., Endtz, H. P., van Doorn, P. A., van der Mech, F. G. A., Van den Berg, L. H., Mollee, I., Logtenberg, T., Thomas, P. K., Plant, G., Baxter, P. J., Luis, R. Santiago, Matsumoto, M., Notermans, N. C., Wokke, J. H. J., Lokhorst, H. M., van der Graaf, Y., Jennekens, F. G. I., Azulay, J. P., Bille-Turg, F., Valentin, P., Farnarier, G. G., Pellissier, J. F., Serratrice, G., Quasthoff, S., Schneider, U., Grafe, P., Hilkens, P. H. E., Moll, J. W. B., van der Burg, M. E. L., Planting, A. S. T., van Putten, W. L. J., van den Bent, M. J., Birklein, F., Spitzer, A., Lang, E., Neundorfer, B., Diehl, R. R., Lücke, D., Smith, G. D. P., Mathias, C. J., Serra, J., Campera, M., Ochoa, J. L., Ray Chaudhuri, K., Pavitt, D., Alam, M., Handwerker, H. O., Bleasdale-Barr, K., Smith, G., Murray, N. M. F., Hawkins, P., Pepys, M., Gellera, C., DiDonato, S., Taroni, F., Uncini, A., Di Muzio, A., Servidei, S., Silvestri, G., Lodi, R., Iotti, S., Barbiroli, B., Morrissey, S. P., Borruat, F. X., Francis, D., Mosely, I., Hansen, H. C., Helmke, K., Kunze, K., Sadzot, B., Maquet, P., Lemaire, Plenevaux, Damhaut, Sommer, C., Myers, R. R., Berta, E., Mantegazza, R., Argov, Z., Shapira, Y., Wirguin, I., Beuuer, J., Franke, C., Roberts, M., Willison, H., Vincent, A., Newsom-Davis, J., Morrison, K. E., Damels, R., Francis, M., Campbell, L., Davies, K. E., Kohler, W., Bucka, C., Hertel, G., Kanovsky, P., Auer, D., Ackermann, H., Klose, U., Naegele, Th., Bien, S., Voigt, K., Fink, G. R., Stephan, K. M., Wise, R. J. S., Mullatti, N., Hewer, L., Frackowiak, R. S. J., Weiller, C. S., Rijnites, M., Jueptner, M., Bauermann, T., Krams, M., Diener, H. C., van Walderveen, M. A. A., Barkhof, F., Hommes, O. R., Valk, J., Willmer, J. P., Guzman, D. A., Passingham, R. E., Silbersweig, D., Ceballos-Baumann, A., Frith, C. D., Frackowiak, R., Lucas, C. H., Goullard, L., Marchau, M. J., Godefroy, O., Rondepierre, P. H., Chamas, E., Mounier-Vehier, F., Leys, D., Renato, J., Verdugo, M. S. C., Campero, M., Jose, L., Ochoa, D. S. C., Vivancos, F., Tejedor, E. Diez, Martinez, N., Roda, J., Frank, A., Barreiro, P., Satoh, Y., Nagata, K., Maeda, T., Hirata, Y., YalÇinerner, B., Ozkara, C., Ozer, F., Ozer, S., Hanoglu, L., Zunker, P., Pozo, J. L., Oberwittler, C., Schick, A., Buschmann, H. -Ch., Ringelstein, E. Bernd, Lara, M., Anzola, G. P., Magoni, M., Volta, G. Dalla, Tarasov, A., Feigin, V., Beaudry, M. G., Carrier, S., Chicoutimi, Henriques, I. L., Bogoussslavsky, J., van Melle, G., Mathieu, J., Perusse, L., Allard, P., Prevost, C., Cantin, L., Bouchard, J. M., De Braekeleer, M., Agbo, C., Neau, J. P., Tantot, A. M., Dary-Auriol, M., Ingrand, P., Gil, R., Baltadjiev, D., Zekin, D., Sabey, K., Gennaula, C. P., Pope, B. A., Caparros-Lefebvre, D., Girard-Buttaz, I., Pruvo, J. P., Petit, H., Hipola, D., Martin, M., Giménez-Roldan, S., Ivanez, V., Japaridze, G., Carrasco, J. L., Picomell, I., Herranz, J. L., Macias, J. A., Nieto, M., Noya, M., Oller, L., Kiteva-Trencevska, G., Delgado, M. R., Liu, H., Luengo, A., Parra, J., Colas, J., Fernandez, M. J., Manzanares, R., Kornhuber, M. E., Malashkhia, V., Orkodashili, G., Martinez, M., Bonaventura, I., Porta, G., Martinez, I., Fernandez, A., Aguilar, M., Masnou, P., Drouet, A., Dreyfus, M., Cartron, J., Morel-Kopp, M. C., Tchernia, G., Kaplan, C., Lammers, M. W., Hekster, Y. A., Keyser, A., Meinardi, H., Renier, W. O., Boon, P. A. J. M., Have, M. D., Kint, B., Cruz, P., Cadilha, A., Almeida, R., Goncalves, M., Pimenta, M., Ramos, L. M. P., Polder, T. W., Broere, C. A., Polman, L., Rother, I., Rother, M., Schlaug, G., Arnold, S., Holthausen, H., Wunderlich, G., Ebner, A., Luders, H., Witte, O. W., Seitz, R. J., Serra, L. L., Gallicchio, B., Rotondi, F., Wieshmann, U., Meierkord, H., Sabev, K., Di Carlo, V., Gueguen, B., Derouesné, Ch., Ancri, D., Bourdel, M. C., Guillou, S., Aliaga, R., Chornet, M. A., Rodrigo, A., Pascual, A. Pascual -Leone, Catala, M. D., Pascual-Leone, A., Benbadis, S. R., Dinner, D. S., Chelune, G. J., Lüders, H. O., Piedmonte, M. R., Blanco, T., Lopez, M. P., Romero, B., Deltoro, A., Pascual, A., Pascual, Leone, Bolgert, F., Josse, M. O., Tassan, P., Touze, E., Laplane, D., Godenberg, F., Brizioli, E., Del Gobbo, M., Pelliccioni, G., Scarpino, O., Durak, H., Damlacik, G., Tunca, Z., Fidaner, H., Yurekli, Y., Yemez, B., Kaygisiz, A., Anllo, E. A., Esperet, E., Giovagnoli, A. R., Casazza, M., Spreafico, R., Avanzini, G., Mascheroni, S., Vecchio, I., Tornali, C., Antonuzzo, A., Grasso, A. A., Bella, R., Pennisi, G., Raffaele, R., Broeckx, J., Schildermans, F., Hospers, W., Deberdt, W., Carney, J. M., Aksenova, M., Chen, M. S., Juncadella, M., Busquets, N., De la Fuente, I., Rodriguez, A., Rubio, F., Soler, R., Khati, C., Pillon, B., Deweer, B., Malapani, C., Malichard, N., Dubois, B., Rancurel, G., Lopez, D. L., Jungreia, G., DeKosky, S. T., Boiler, F., Weiller, C., Rijntjes, M., Mueller, S. P., Maguire, E. A., Burke, E. T., Staunton, H., Phillips, J., Rousseaux, M., Pena, J., Bertran, I., Santacruz, P., Lopez, R., Catafau, A., Lomena, F., Blesa, R., Rampello, L., Nicoletti, A., Cabaret, M., Lesoin, F., Steinling, M., Tournev, I., Maier-Hauff, K., Schroeder, M., Wolf, A., Cochin, J. P., Noel, I., Augustin, P., Auzou, P., Hannequin, D., Maria, V., Lopez-Bresnahan, Danielle, D. M., Antin-Ozerkis B. A., Bartels, E., Rodiek, S. O., Flugel, K. A., Campos, D. M., Salas-Puig, J., Del Rio, J. Sanhez, Vidal, J. A., Lahoz, C. H., Eraksoy, M., Barlas, O., Barlas, M., Bayindir, C., Ozcan, H., Birbamer, G., Gerstenbrand, F., Felber, S., Luz, G., Aichner, F., Seidel, G., Kaps, M., Hutzelmann, A., Gerriets, T., Kruggel, F., Martin, P. J., Gaunt, M. E., Abbot, R. J., Naylor, A. R., Meary, E., Dilouya, A., Meder, J. F., De Recondo, J., Lebtahi, R., Neff, K. W., Meairs, S., Viola, S., Matta, E., Aquilone, L., Rise, I. R., Authier, F. J., Kondo, H., Ghnassia, R. T., Degos, J. D., Gherardi, R. K., Bardoni A., Ciafaloni E., Comi G. P., Bresolin N., Robotti M., Moggio M., Rigoletto C., Roses A., Scarlato G., Castelli, E., Turconi, A., Bresolin, N., Perani, D., Felisari, G., Chariot, P., de Pinieux, G., Astier, A., Jacotot, B., Gherardi, R., Fischer-Gagnepain, V., Louboutin, J. P., Crespo, F., Florea-Strat, A., Fromont, G., Sabourin, J. -C., Gonano, E. -F., Moroni, I., Prelle, A., Iannaccone, S., Quattrini, A., deRino, F., Sessa, M., Golzi, V., Smirne, S., Nemni, R., Turpin, J. C., Lucotte, G., Jacobs, S. C. J. M., Willems, P. W. A., Bootsma, A. L., Lasa, A., Calaf, M., Baiget, M., Gallano, B., Fichter-Gagnepain, V., Mazzucchelli, F., D'Angelo, M. G., Velicogna, M., Bet, L., Comi, G. P., Bordoni, A., Gonano, E. F., Bazzi, P., Rapuzzi, S., Moggio, M., Fagiolari, G., Ciscato, P., Messina, A., Battistel, A., Ryniewicz, B., Sangla, I., Desnuelle, C., Paquis, V., Cozzone, P. J., Bendahan, D., Sturenburg, H. 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4. A comparative technetium 99m hexamethylpropylene amine oxime SPET study in different types of dementia
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Habert, M. O., Spampinato, U., Mast, J. L., Piketty, M. L., Bourdel, M. C., de Recondo, J., Rondot, P., and Askienazy, S.
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- 1991
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5. EEG Mapping in Pathological Aging and Dementia: Utility for Diagnosis and Therapeutic Evaluation
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Gueguen, B., Etevenon, P., Plancon, D., Gaches, J., De Recondo, J., Rondot, P., and Maurer, Konrad, editor
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- 1989
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6. Dystonia-parkinsonism syndrome resulting from a bullet injury in the midbrain.
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Rondot, P, primary, Bathien, N, additional, de Recondo, J, additional, Gueguen, B, additional, Fredy, D, additional, de Recondo, A, additional, and Samson, Y, additional
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- 1994
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7. Clinical Efficacy of a Liquid Formulation of Levodopa (Madopar Dispersible) in Reversing Afternoon “Off” Periods in Parkinsonʼs Disease
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Ziégler, M., primary, anoux, D. R, additional, and de Recondo, J., additional
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- 1994
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8. Polyradiculonévrite chronique associée à une hépatite B et delta
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Dhôte, R, primary, Zuber, M, additional, Madeira, I, additional, Bachmeyer, C, additional, Le Dinh, T, additional, Mas, JL, additional, De Recondo, J, additional, and Christoforov, B, additional
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- 1994
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9. Atrial septal aneurysm and patent foramen ovale as risk factors for cryptogenic stroke in patients less than 55 years of age. A study using transesophageal echocardiography.
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Cabanes, L, primary, Mas, J L, additional, Cohen, A, additional, Amarenco, P, additional, Cabanes, P A, additional, Oubary, P, additional, Chedru, F, additional, Guérin, F, additional, Bousser, M G, additional, and de Recondo, J, additional
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- 1993
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10. Botulinum antibodies in dystonic patients treated with type A botulinum toxin: Frequency and significance
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Zuber, M., primary, Sebald, M., additional, Bathien, N., additional, de Recondo, J., additional, and Rondot, P., additional
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- 1993
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11. Hereditary cerebral hemorrhage with amyloidosis-Dutch type
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Mas, J.-L., primary and de Recondo, J., additional
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- 1993
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12. (99mTc)-HM-PAO SPECT and cognitive impairment in Parkinson's disease: a comparison with dementia of the Alzheimer type.
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Spampinato, U, primary, Habert, M O, additional, Mas, J L, additional, Bourdel, M C, additional, Ziegler, M, additional, de Recondo, J, additional, Askienazy, S, additional, and Rondot, P, additional
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- 1991
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13. Postradiation lower motor neuron syndrome presenting as monomelic amyotrophy.
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Lamy, C, primary, Mas, J L, additional, Varet, B, additional, Ziegler, M, additional, and de Recondo, J, additional
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- 1991
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14. THA effects on EEG activity in a double-blind cross-over study in alzheimer's disease
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Gasnault, J., primary, Gueguen, B., additional, Derouesne, C., additional, Bourdel, M.-C., additional, Richet, S., additional, Cosson, J.-P., additional, Tarade, T., additional, Mas, J.-L., additional, De Recondo, J., additional, and Rondot, P., additional
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15. Ageing and Parkinson's Disease.
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Rondot, P., Bianco, C., and de Recondo, J.
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16. VISUOMOTOR ATAXIA.
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RONDOT, P., DE RECONDO, J., and DUMAS, J. L. RIBADEAU
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17. A familial disorder with subcortical ischemic strokes, dementia, and leukoencephalopathy.
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Mas, Jean Louis, Dilouya, Annie, Recondo, Jean de, Mas, J L, Dilouya, A, and de Recondo, J
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- 1992
- Full Text
- View/download PDF
18. [Positron-emission tomographic study of the dopaminergic system in a case of secondary unilateral tremor after mesencephalic hematoma]
- Author
-
De Recondo A, Rondot P, Loc'h C, Crouzel C, Mh, Dao-Castellana, Ng, Bathien, Rascol O, Clanet M, De Recondo J, and yves samson
- Subjects
Adult ,Mesencephalon ,Brain Injuries ,Dopamine ,Tremor ,Humans ,Female ,Magnetic Resonance Imaging ,Corpus Striatum ,Cerebral Hemorrhage ,Tomography, Emission-Computed - Abstract
A 30 year-old woman developed a postural and rest tremor of the left hand following a right peduncular post-traumatic hematoma. Two years later, positron emission tomography showed a marked decrease in [18F] fluorodopa uptake contrasting with a normal [76Br] bromolisuride uptake in the right striatum. This suggests that: 1) chronic unilateral dopaminergic striatal denervation may occur without persistent D2 dopaminergic receptor upregulation in humans; and 2) symptomatic mesencephalic tremor may be, at least in part, related to dopaminergic striatal denervation.
19. [Meningoradiculitis after injection of an antirabies vaccine. A vaccine from human diploid cell culture]
- Author
-
Antoine MOULIGNIER, Richer A, Fritzell C, Foulon D, Khoubesserian P, and de Recondo J
- Subjects
Male ,Rabies Vaccines ,Rabies ,Humans ,Middle Aged ,Radiculopathy - Abstract
We report the case of a 45-year old farmer who developed meningoradiculitis after preventive anti-rabies vaccination with a vaccine obtained from human diploid cell culture. Two weeks after the second injection of vaccine, the patient complained of sensory symptoms in the right half of his body. These symptoms spontaneously regressed. The literature is reviewed and the physiopathological hypotheses are discussed.
20. Ataxie optique: trouble de la coordination visuo-motrice
- Author
-
Rondot, P., primary and De Recondo, J., additional
- Published
- 1974
- Full Text
- View/download PDF
21. 19. EEG mapping in dementia: utility for diagnosis and therapeutic evaluation
- Author
-
Gueguen, B., primary, Etevenon, P., additional, Plancon, D., additional, Gaches, J., additional, de Recondo, J., additional, and Rondot, P., additional
- Published
- 1987
- Full Text
- View/download PDF
22. Menzel's hereditary ataxia with slow eye movements and myoclonus
- Author
-
Rondot, P., primary, De Recondo, J., additional, Davous, P., additional, and Vedrenne, C., additional
- Published
- 1983
- Full Text
- View/download PDF
23. Conduction of the sciatic nerve in its proximal and distal segment in patients with ALS (amyotrophic lateral sclerosis)
- Author
-
Koutlidis, R.M., primary, de Recondo, J., additional, and Bathien, N., additional
- Published
- 1984
- Full Text
- View/download PDF
24. Autosomal dominant familial spastic paraplegia is genetically heterogeneous and one locus maps to chromosome 14q.
- Author
-
Hazan J, Lamy C, Melki J, Munnich A, de Recondo J, and Weissenbach J
- Subjects
- Adult, Base Sequence, Child, Chromosome Mapping, DNA Primers, Female, Humans, Male, Molecular Sequence Data, Pedigree, Chromosomes, Human, Pair 14, Genes, Dominant, Spastic Paraplegia, Hereditary genetics
- Abstract
Autosomal dominant familial spastic paraplegia (FSP) is a degenerative disorder of unknown aetiology characterized by a progressive spasticity of the legs. Three families with autosomal dominant FSP of early onset were analysed in linkage studies using highly polymorphic microsatellite markers. Close linkage to a group of markers on chromosome 14q (maximum multipoint lodscore z = 10) was observed in one family. This chromosome 14q candidate region was entirely excluded in the two other families, providing evidence of genetic heterogeneity within a homogeneous clinical form of FSP.
- Published
- 1993
- Full Text
- View/download PDF
25. [Brachial plexus involvement disclosing systemic lupus erythematosus].
- Author
-
Assal F, Blanché P, Lamy C, Sicard D, and De Recondo J
- Subjects
- Female, Humans, Middle Aged, Brachial Plexus, Lupus Erythematosus, Systemic complications
- Published
- 1993
26. [Positron-emission tomographic study of the dopaminergic system in a case of secondary unilateral tremor after mesencephalic hematoma].
- Author
-
De Recondo A, Rondot P, Loc'h C, Crouzel C, Dao-Castellana MH, Bathien NG, Rascol O, Clanet M, De Recondo J, and Samson Y
- Subjects
- Adult, Brain Injuries diagnosis, Cerebral Hemorrhage diagnosis, Corpus Striatum diagnostic imaging, Female, Humans, Magnetic Resonance Imaging, Mesencephalon, Tremor diagnostic imaging, Brain Injuries complications, Cerebral Hemorrhage complications, Corpus Striatum physiopathology, Dopamine physiology, Tomography, Emission-Computed, Tremor etiology
- Abstract
A 30 year-old woman developed a postural and rest tremor of the left hand following a right peduncular post-traumatic hematoma. Two years later, positron emission tomography showed a marked decrease in [18F] fluorodopa uptake contrasting with a normal [76Br] bromolisuride uptake in the right striatum. This suggests that: 1) chronic unilateral dopaminergic striatal denervation may occur without persistent D2 dopaminergic receptor upregulation in humans; and 2) symptomatic mesencephalic tremor may be, at least in part, related to dopaminergic striatal denervation.
- Published
- 1993
27. Clinical and genetic heterogeneity of Charcot-Marie-Tooth disease.
- Author
-
Hentati A, Lamy C, Melki J, Zuber M, Munnich A, and de Recondo J
- Subjects
- Chromosome Mapping, Chromosomes, Human, Pair 17, Female, Genetic Linkage, Humans, Lod Score, Male, Pedigree, Charcot-Marie-Tooth Disease genetics
- Abstract
The autosomal dominant forms of hereditary motor and sensory neuropathies include the hypertrophic form (CMT1) and the neuronal form of Charcot-Marie-Tooth disease (CMT2). While at least two distinct loci have been shown to be linked to the CMT1 phenotype (CMT1A and CMT1B, on chromosomes 17 and 1, respectively), whether the CMT2 phenotype results from mutations allelic to either of the CMT1 genes remains unknown. Studying one CMT1 and two CMT2 pedigrees, we were able to exclude the CMT2 disease locus from the region of chromosome 17 (Z = -2.80 at theta = 0.05 for D17S58) where the CMT1A gene maps (Z = +3.67 at theta = 0.00). Similarly, negative lod score values were obtained in CMT2 for the region of chromosome 1 where the CMT1B gene has been located (Z = -3.09 at theta = 0.05 for D1S61). The present study therefore provides evidence for genetic heterogeneity between the hypertrophic and the neuronal forms of Charcot-Marie-Tooth disease and demonstrates that the CMT2 gene is not allelic to either of the CMT1 genes mapped to date.
- Published
- 1992
- Full Text
- View/download PDF
28. Intravascular lymphomatosis (neoplastic angioendotheliosis) of the central nervous system: case report and literature review.
- Author
-
Smadja D, Mas JL, Fallet-Bianco C, Meyniard O, Sicard D, de Recondo J, and Rondot P
- Subjects
- Blood Vessels pathology, Brain blood supply, Brain pathology, Female, Humans, L-Lactate Dehydrogenase metabolism, Middle Aged, Brain Neoplasms pathology, Lymphoma, B-Cell pathology
- Abstract
A 63-year-old woman developed gradual slowness, recurrent multifocal deficits, severe constitutional symptoms and hypopituitarism which progressed to death over 2.5 months. Elevation in lactico dehydrogenases was the main biological abnormality. Necropsy showed an intravascular malignant proliferation which proved to be a lymphoma of B-lineage. In order to better define diagnostic criteria, we reviewed previously reported cases of intravascular lymphomatosis of the central nervous system. A strategy for establishing the diagnosis is proposed.
- Published
- 1991
- Full Text
- View/download PDF
29. [Meningoradiculitis after injection of an antirabies vaccine. A vaccine from human diploid cell culture].
- Author
-
Moulignier A, Richer A, Fritzell C, Foulon D, Khoubesserian P, and de Recondo J
- Subjects
- Humans, Male, Middle Aged, Rabies Vaccines therapeutic use, Rabies prevention & control, Rabies Vaccines adverse effects, Radiculopathy chemically induced
- Abstract
We report the case of a 45-year old farmer who developed meningoradiculitis after preventive anti-rabies vaccination with a vaccine obtained from human diploid cell culture. Two weeks after the second injection of vaccine, the patient complained of sensory symptoms in the right half of his body. These symptoms spontaneously regressed. The literature is reviewed and the physiopathological hypotheses are discussed.
- Published
- 1991
30. [Late postpartum eclampsia, myth or reality?].
- Author
-
Richer A, Richer E, Mouligner A, Dilouya A, Vige P, and de Recondo J
- Subjects
- Adult, Brain Ischemia etiology, Cerebral Angiography standards, Cerebrovascular Circulation, Eclampsia complications, Eclampsia pathology, Female, Humans, Hypertension etiology, Magnetic Resonance Imaging standards, Pregnancy, Puerperal Disorders complications, Puerperal Disorders pathology, Ultrasonography standards, Brain Ischemia diagnosis, Eclampsia diagnosis, Hypertension diagnosis, Puerperal Disorders diagnosis
- Abstract
It has never been formally established whether eclampsia can come on more than 48 hours after delivery. We report a case of a patient who had convulsions together with transitory raised blood pressure coming on 14 days after her delivery. MRI was carried out 24 hours after the attack and showed pathological sub-cortical images and there was a hyper signal at T2. This is now a well known feature and has been described in the course of typical eclampsia fits. On the other hand MRI is able to eliminate a certain number of differential diagnoses such as cerebral thrombophlebitis, cerebral vascular accidents and tumours. A cerebral blood flow study and a trans-cranial Doppler flow study showed (as far as we know for the first time) a lessening in perfusion and in speed of flow giving rise to lowered blood circulation in that part of the brain and suggestive of vascular spasm. This observation makes it very likely that eclamptic crises can occur two weeks after delivery and favours a vascular spasm being the origin of these crises.
- Published
- 1991
31. [Neurogenic, medullary, cerebellar, myogenic syndrome. Diagnostic orientation and management].
- Author
-
Lamy C and de Recondo J
- Subjects
- Cerebellar Diseases etiology, Humans, Muscular Diseases etiology, Neuromuscular Diseases etiology, Spinal Cord Injuries etiology, Cerebellar Diseases diagnosis, Motor Neurons, Muscular Diseases diagnosis, Neuromuscular Diseases diagnosis, Spinal Cord Injuries diagnosis
- Published
- 1990
32. [Cerebellar atrophies].
- Author
-
Zuber M, Lamy C, and de Recondo J
- Subjects
- Atrophy congenital, Atrophy diagnosis, Central Nervous System Diseases diagnosis, Central Nervous System Diseases physiopathology, Humans, Nerve Degeneration, Olivopontocerebellar Atrophies diagnosis, Cerebellum pathology
- Abstract
Cerebellar atrophy is often discovered at computerized tomography or MRI of the brain, but its exact cause is frequently difficult to determine. Alcoholism is the predominant factor, but numerous other diseases may also be considered. The diseases fall into two main categories: primary cerebellar degeneration with, in particular, olivo-ponto-cerebellar atrophy, and acquired atrophy which may be of paraneoplastic or endocrine origin or caused by toxic agents other than alcohol. In this review paper the stress is placed on the clinical, paraclinical and histological distinctive features of the different types of cerebellar atrophy. The modalities of onset and the course of these lesions are of particular importance. The authors propose a practical aid to the diagnosis in the form of a deliberately simplified diagram.
- Published
- 1990
33. [Agenesis of the corpus callosum in a man with complete mosaic trisomy 8].
- Author
-
Baverel F, de Recondo J, Rouffet A, Fredy D, Salesses A, and Rondot P
- Subjects
- Adult, Corpus Callosum diagnostic imaging, Humans, Karyotyping, Male, Radiography, Agenesis of Corpus Callosum, Chromosomes, Human, 6-12 and X ultrastructure, Epilepsy etiology, Trisomy
- Abstract
The coexistence in an epileptic patient of a radiologically confirmed agenesis of the corpus callosum and other somatic abnormalities, notably skeletal, prompted us to perform a karyotype which showed an extra chromosome 8. The trisomy 8-callosal agenesis association is not exceptional, but it may easily be missed owing to the absence or scarcity of clinical signs of the cerebral malformation. We would suggest that patients with confirmed agenesis of the corpus callosum should be investigated for trisomy 8 and conversely, that patients with trisomy 8 detected during examination for a characteristic malformative syndrome should be systematically investigated for abnormality of the neocortical commissure.
- Published
- 1985
34. [Oculomotor paralysis and peripheral neuropathy caused by IgM kappa monoclonal immunoglobulin. Transient effect of plasmapheresis].
- Author
-
Ruel M, Baudrimont M, Dreyfus F, Cavicchi L, De Recondo J, and Varet B
- Subjects
- Humans, Immunoglobulin kappa-Chains, Male, Middle Aged, Ophthalmoplegia therapy, Peripheral Nervous System Diseases therapy, Plasmapheresis, Hypergammaglobulinemia complications, Immunoglobulin M, Ophthalmoplegia etiology, Peripheral Nervous System Diseases etiology
- Published
- 1989
35. [Functional occular paralysis].
- Author
-
de Recondo J
- Subjects
- Cerebellar Diseases complications, Facial Paralysis complications, Fixation, Ocular, Functional Laterality, Humans, Ophthalmoplegia classification, Reflex, Pupillary, Strabismus complications, Syndrome, Ophthalmoplegia physiopathology
- Published
- 1974
36. [X-ray computed tomography of lumbosacral roots and primary hypertrophic neuritis (Dejerine-Sottas disease)].
- Author
-
Mas JL, Buthiau D, Fallet-Bianco C, Cheron F, Raulo P, de Recondo J, and Rondot P
- Subjects
- Adult, Aged, Electrophysiology, Female, Hereditary Sensory and Motor Neuropathy classification, Hereditary Sensory and Motor Neuropathy etiology, Humans, Male, Middle Aged, Hereditary Sensory and Motor Neuropathy diagnostic imaging, Spinal Nerve Roots diagnostic imaging, Tomography, X-Ray Computed
- Abstract
CT without contrast of lumbosacral nerve roots was performed in 13 patients with peroneal atrophy and 28 control subjects. Two series of 5 mm serial sections parallel to the plane of the disk were examined at the L4-L5 and L5-S1 levels, and the transverse diameter of the S1 nerve roots measured at the lower part of the lateral recess. Results showed frank bilateral, grossly symmetrical hypertrophy of lumbosacral roots in 8 of the 13 patients. This hypertrophy involved all roots examined (L4, L5, S1), except in one case where only S1 roots were involved. Hypertrophy was often more marked on the distal part of the roots and on spinal nerves, contrasting with the sometimes normal or only slightly altered appearance of the nerve roots emerging from the dural sac. In these 8 cases, the diameter of the S1 nerve roots was 8 to 18 mm, in contrast to a mean of 3.5 +/- 1 mm in the 28 controls. CT scan images were normal in the remaining 5 patients. The presence of a CT image of nerve hypertrophy was in all cases associated with a marked fall in nerve conduction rate (median nerve motor conduction rate less than 25 msec-1), and a decrease in number of myelinated fibers with numerous onion bulbs. In contrast, the absence of CT nerve hypertrophy could not predict the results of electrophysiological and histological examinations.
- Published
- 1989
37. [Difficulty in manual prehension under visual control (author's transl)].
- Author
-
Rondot P and de Recondo J
- Subjects
- Adult, Ataxia etiology, Brain Diseases complications, Brain Diseases surgery, Female, Functional Laterality, Humans, Male, Middle Aged, Postoperative Complications, Visual Fields, Ataxia physiopathology, Hand physiopathology, Vision Disorders physiopathology
- Abstract
Visuomotor ataxia causes difficulty in the prehension of objects under visual control which can occur without paralysis of ocular fixation and spatial agnosia. This can involve the total visual field or the right or left half-fields, and can affect both hands or one only. 1) Unilateral visuomotor ataxia is localized to the two right or left homonymous half-fields and can affect both hands or one only. It is direct when the ataxic hand is on the same side as the affected visual half-field, and crossed when the ataxic hand is contralateral to the affected visual half-field. 2) Bilateral visuomotor ataxia involves the total visual field. Each hand may be ataxic only for the contralateral visual field with a crossed bilateral visuomotor ataxia, or in the homolateral field with a direct bilateral visuomotor ataxia. The anatomical and clinical observations reported imply the existence of visuomotor connections, both direct and crossed, the latter crossing the median line through the corpus callosum.
- Published
- 1978
38. [Diffuse corticoid-sensitive cerebral arteritis (author's transl)].
- Author
-
Rondot P, de Recondo J, Benhamou CL, Sicard D, and Daumas-Duport C
- Subjects
- Adult, Arteritis diagnosis, Arteritis etiology, Cerebral Angiography, Cerebral Arterial Diseases diagnosis, Cerebral Arterial Diseases etiology, Diagnosis, Differential, Female, Giant Cell Arteritis diagnosis, Humans, Male, Middle Aged, Adrenal Cortex Hormones therapeutic use, Arteritis drug therapy, Cerebral Arterial Diseases drug therapy
- Abstract
Three cases of cerebral vascularitis are reported. This affection does not occur frequently and is often unrecognized. It can be isolated or associated with disease such as sarcoidosis, Behcet's disease, or collagen disorders (lupus erythematosus, mixed connective tissue diseases). Diagnosis is made from the evolution with transient remission, the inflammatory nature of the CSF, and the presence of segmentary narrowing on arteriography. In case of doubt an exploratory operation with biopsy is necessary. The most likely etiology is an auto-immune disturbance, and cortico-therapy has greatly improved the prognosis.
- Published
- 1980
39. [Adrenomyeloneuropathy: clinical, genetic and morphologic features. Apropos of 5 cases].
- Author
-
Rondot P, de Recondo J, Davous P, Dubas F, and Khoubesserian P
- Subjects
- Adrenoleukodystrophy genetics, Adrenoleukodystrophy pathology, Biopsy, Fatty Acids blood, Female, Genes, Recessive, Humans, Male, Microscopy, Electron, Middle Aged, Peripheral Nerves pathology, X Chromosome, Adrenoleukodystrophy diagnosis, Diffuse Cerebral Sclerosis of Schilder diagnosis
- Abstract
The authors describe five cases of adrenomyeloneuropathy, an adult form of adrenoleukodystrophy. Three of the cases were related. The clinical picture comprises progressive spasmodic paraplegia, sphincter disorders, peripheral neuropathy, sometimes hollow foot and posterior columns disorders. In one case psychic disorders were observed. Biochemical analyses confirmed or revealed adrenal insufficiency in four cases and gonadal insufficiency in three cases. A detailed genealogical study was made in two patients belonging to the same family. Histocompatibility tests revealed the presence of A2 and B15 antigens in three cases. Diagnosis was confirmed in three cases by plasma determination of long-chain fatty acids, with an increase in the level of C26 acids and the C26/C22 ratio; biopsy of the peripheral nerve in one patient revealed inclusions characteristic of Schwann's cells. Symptomatological links between ALD and familial spasmodic paraplegia, the mode of genetic transmission of the disease, the contribution of neuromuscular biopsy to the diagnosis and the therapeutic prospects of a diet low in long-chain fatty acids are discussed.
- Published
- 1983
40. [Aphasia caused by left paramedian thalamic infarction. Anatomo-clinical case].
- Author
-
Davous P, Bianco C, Duval-Lota AM, de Recondo J, Vedrenne C, and Rondot P
- Subjects
- Aged, Aphasia pathology, Cerebral Infarction pathology, Humans, Male, Thalamic Diseases pathology, Aphasia etiology, Brain pathology, Cerebral Infarction complications, Thalamic Diseases complications
- Abstract
A 67 year-old right-handed man presented with an aphasia of acute onset associated to paralysis of vertical eye movements, mild cerebellar right dysmetria and right hemiparesis without hemianopia. CT scan showed two low density areas in the left thalamus and left occipital lobe. Neuropsychological examination revealed a non fluent aphasia with normal repetition, semantic paraphasias, perseverations and good comprehension of verbal and written commands. There was no alexia. Writing was impaired only by motor disturbances. Three weeks after the onset, tetraparesis and impairment of consciousness followed by a locked in syndrome supervened. Death occurred four months after onset. Neuropathological examination showed a left paramedian thalamic infarct involving the ventrolateral, dorso-medial and intralaminar nuclei, sparing the pulvinar. There was an occipital infarct sparing the calcarine scissura and multiple infarctions in the pons and the cerebellum. Broca's and Wernicke's areas were spared. We suggest that the involvement of medial nuclei could be partly responsible of language disturbances in thalamic aphasia.
- Published
- 1984
41. Mental disorders in Parkinson's disease after treatment with L-DOPA.
- Author
-
Rondot P, de Recondo J, Coignet A, and Ziegler M
- Subjects
- Aged, Anxiety Disorders etiology, Confusion etiology, Delirium etiology, Dementia etiology, Depressive Disorder etiology, Female, Hallucinations etiology, Humans, Male, Mental Disorders chemically induced, Middle Aged, Parkinson Disease complications, Levodopa adverse effects, Mental Disorders etiology, Parkinson Disease drug therapy
- Published
- 1984
42. Visuomotor ataxia.
- Author
-
Rondot P, de Recondo J, and Dumas JL
- Subjects
- Adult, Aged, Ataxia pathology, Ataxia physiopathology, Brain pathology, Female, Fixation, Ocular, Humans, Middle Aged, Neural Pathways, Perceptual Disorders pathology, Perceptual Disorders physiopathology, Visual Fields, Ataxia diagnosis, Perceptual Disorders diagnosis, Visual Perception
- Abstract
Visuomotor ataxia is a disorder of movement performed under visual control. It can occur in the absence of disturbance of ocular fixation and in the absence of spatial agnosia. This disorder may extend over the whole visual field or it may be localized to one visual half-field, right or left. It may involve both hands or one hand only, so that visuomotor ataxia may be divided into: (1) Unilateral visuomotor ataxia, localized to a single field. In this case it may affect both hands or a single hand. It is direct when the hand is ataxic in the ipsilateral visual field and it is crossed when the hand is ataxic in the contralateral visual field; (2) Bilateral visuomotor ataxia, involving the whole visual field. Each hand may be ataxic only in the contralateral visual field, that is, bilateral crossed visuomotor ataxia; or in the ipsilateral field when it is called bilateral direct visuomotor ataxia. The observed clinical variations which are described here imply the existence of both direct and crossed visuomotor connections, the latter probably crossing the corpus callosum in the splenium.
- Published
- 1977
- Full Text
- View/download PDF
43. [Familial dementia of the Neumann type (subcortical gliosis)].
- Author
-
Khoubesserian P, Davous P, Bianco C, Puymirat J, Fontaine C, de Recondo J, and Rondot P
- Subjects
- Aged, Brain pathology, Brain ultrastructure, Brain Chemistry, Dementia diagnosis, Diagnosis, Differential, Humans, Male, Neurotransmitter Agents analysis, Dementia genetics, Gliosis genetics
- Abstract
The case of a 70 year-old patient with familial dementia is reported. A cerebral biopsy and neurotransmitters dosages in CSF and brain tissue ruled out a primary dementia as Pick's or Alzheimer's disease. Our case may be related to Neumann's cases of subcortical gliosis.
- Published
- 1985
44. [Extrapyramidal rigidity with dystonia, optic atrophy and bilateral putaminal lesions in an adolescent. Juvenile form of Leigh's disease (author's transl)].
- Author
-
Rondot P, de Recondo J, Davous P, Fredy D, and Roux FX
- Subjects
- Adult, Humans, Intellectual Disability etiology, Male, Muscle Rigidity etiology, Optic Atrophy etiology, Psychomotor Disorders etiology, Putamen diagnostic imaging, Tomography, X-Ray Computed, Basal Ganglia Diseases diagnostic imaging, Brain Stem diagnostic imaging, Encephalomalacia diagnostic imaging
- Abstract
A 19-year-old man presented with an apparently non-familial neurological disorder that had progressed from the age of 6 years. Dystonia of the trunk and limbs with extrapyramidal rigidity, dysarthria, a pyramidal syndrome with spasticity of the lower limbs, bilateral optic atrophy, and nystagmiform ocular movements were present. CT scan demonstrated symmetrical putaminal lesions. The different aetiologies of bilateral striatal lesions are considered, the final diagnosis being a juvenile form of Leigh's disease.
- Published
- 1982
45. [Bilateral thalamic infarcts with abnormal movements and permanent amnesia].
- Author
-
Rondot P, de Recondo J, Davous P, Bathien N, and Coignet A
- Subjects
- Cerebral Infarction diagnosis, Electroencephalography, Electromyography, Female, Heart Valve Diseases complications, Humans, Middle Aged, Ophthalmoplegia etiology, Rheumatic Heart Disease complications, Amnesia etiology, Cerebral Infarction complications, Movement Disorders etiology, Thalamus blood supply
- Abstract
A 45 year-old woman with mitral valve disease developed bilateral thalamic infarcts presenting as vigilance disorders, with mutism and downwards gaze paralysis during several days, followed by involuntary rhythmic movements over 13 days. These movements affected the four limbs with a rhythm variable with time as alternate or synchronized agonist-antagonist periods. Subsequent examinations demonstrated global, lasting amnesia and a subclinical disorder of vertical ocular movements. Repeat CT scan confirmed the presence of bilateral anterior thalamic infarcts affecting polar and/or paramedian territories. Emphasis is placed on the similarity of the abnormal movements in this case with L-dopa induced involuntary movements.
- Published
- 1986
46. [Abnormal movements].
- Author
-
de Recondo J
- Subjects
- Athetosis diagnosis, Chorea diagnosis, Humans, Hyperkinesis diagnosis, Myoclonus diagnosis, Parkinson Disease diagnosis, Tremor diagnosis, Movement Disorders diagnosis
- Published
- 1980
47. [Carotid bruit due to increased blood flow without stenosis: report on two cases (author's transl)].
- Author
-
Rondot P, de Recondo J, and Gaillard-Sizaret C
- Subjects
- Adolescent, Adult, Arteriosclerosis physiopathology, Auscultation, Carotid Artery, External diagnostic imaging, Cerebrovascular Circulation, Hemangioma diagnostic imaging, Humans, Male, Radiography, Brain Neoplasms diagnosis, Carotid Artery, External physiopathology, Hemangioma diagnosis
- Abstract
Initial reaction to the discovery of a carotid bruit in an adult is that a local atheromatous lesion exists. This sign may arise, however, when more distal lesions are present, such as an angioma provoking an increased rate of blood flow. All patients presenting with neurological signs of a lesion in one hemisphere should, therefore, in the absence of contra-indications, be investigated by arteriography.
- Published
- 1982
48. [Adrenomyeloneuropathy. 4 cases developing with the features of familial spasmodic paraplegia].
- Author
-
Rondot P, De Recondo J, Davous P, Dubas F, Khoubesserian P, and Coignet A
- Subjects
- Adrenoleukodystrophy complications, Adrenoleukodystrophy diagnosis, Adult, Humans, Male, Middle Aged, Muscle Spasticity etiology, Muscle Spasticity genetics, Paraplegia etiology, Adrenoleukodystrophy genetics, Diffuse Cerebral Sclerosis of Schilder genetics, Paraplegia genetics
- Abstract
Four cases of adrenomyelopathy, the adult form of adrenoleukodystrophy, three of which were familial, were reported. The neurological disease consisted of progressive spasmodic paraplegia, sphincter disorders, peripheral neuropathy, occasional deep sensibility impairment and psychiatric disturbances in one case. Adrenal insufficiency was confirmed after the appearance of the neurological signs in two cases; in another case it had been diagnosed in childhood. Gonadal insufficiency was present in all cases and clinically apparent in two patients. Detailed genetic studies were carried out in two patients of the same family. Histocompatibility tests showed A2 and B15 antigens in three out of four cases. The diagnosis was confirmed in three cases by measurement of serum long chain fatty acid with increased C26 levels and C26/C22 ratios. Peripheral nerve biopsy in 1 patient showed characteristic inclusion bodies in the Schwann cells. The authors discuss the relationship between ALD and familial spasmodic paraplegia, the mode of genetic transmission of the disease and the diagnostic value of neuromuscular biopsy and measurement of serum long chain fatty acid levels.
- Published
- 1984
49. [Progressive external ophthalmoplegia with brain stem nuclei spongiosis].
- Author
-
Castaigne P, Laplane D, Escourolle R, Augustin P, de Recondo J, Martinez Lage GJ, and Villanueva Eusa JA
- Subjects
- Adult, Brain Diseases physiopathology, Electroencephalography, Female, Heart Block complications, Humans, Intellectual Disability complications, Ophthalmoplegia physiopathology, Pharyngeal Diseases etiology, Retinitis Pigmentosa complications, Vestibule, Labyrinth physiopathology, Brain Diseases complications, Brain Stem, Ophthalmoplegia etiology
- Published
- 1971
50. [Electron microscopic study of a peripheral nerve biopsy specimen in four cases of Dejerine-Sottas hypertrophic neuritis].
- Author
-
Garcin R, Lapresle J, Fardeau M, and De Recondo J
- Subjects
- Adolescent, Adult, Female, Humans, Male, Microscopy, Electron, Middle Aged, Myelin Sheath pathology, Schwann Cells, Hypertrophy pathology, Musculocutaneous Nerve pathology, Neuritis pathology
- Published
- 1966
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