171 results on '"Darley, Richard L"'
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2. LEF-1 drives aberrant β-catenin nuclear localization in myeloid leukemia cells
3. Increased expression of RUNX3 inhibits normal human myeloid development
4. RUNX3 overexpression inhibits normal human erythroid development
5. Integrated nuclear proteomics and transcriptomics identifies S100A4 as a therapeutic target in acute myeloid leukemia
6. Using Flow Cytometry to Detect and Measure Intracellular Thiol Redox Status in Viable T Cells from Heterogeneous Populations
7. Supplemental Table S2 from Reactive Oxygen Species Drive Proliferation in Acute Myeloid Leukemia via the Glycolytic Regulator PFKFB3
8. Supplemental Methods, Fig S1, Fig S2, Fig S3, Fig S4, Fig S5, Fig S6, Fig S7, Fig S8, Fig S9, Fig S10, Fig S11, Fig S12. from Reactive Oxygen Species Drive Proliferation in Acute Myeloid Leukemia via the Glycolytic Regulator PFKFB3
9. Data from Reactive Oxygen Species Drive Proliferation in Acute Myeloid Leukemia via the Glycolytic Regulator PFKFB3
10. Nuclear factor I-C overexpression promotes monocytic development and cell survival in acute myeloid leukemia
11. Overproduction of NOX-derived ROS in AML promotes proliferation and is associated with defective oxidative stress signaling
12. Protein Kinase C Epsilon Overexpression Is Associated With Poor Patient Outcomes in AML and Promotes Daunorubicin Resistance Through p-Glycoprotein-Mediated Drug Efflux
13. Translocation or Just Location? Pseudopodia Affect Fluorescent Signals
14. Crosstalk between β-catenin and WT1 signaling activity in acute myeloid leukemia
15. Ras-induced reactive oxygen species promote growth factor–independent proliferation in human CD34+ hematopoietic progenitor cells
16. Reactive oxygen species in leukemias: maintaining cancer cell proliferation via redox signaling and changing metabolic homeostasis
17. The role of PKC and PDK1 in monocyte lineage specification by Ras
18. Reactive Oxygen Species Rewires Metabolic Activity in Acute Myeloid Leukemia
19. Use of an anti‐CD200‐blocking antibody improves immune responses to AML in vitro and in vivo
20. Reactive Oxygen Species Drive Proliferation in Acute Myeloid Leukemia via the Glycolytic Regulator PFKFB3
21. FUS expression alters the differentiation response to all-trans retinoic acid in NB4 and NB4R2 cells
22. Sequential changes in MHC antigen expression induced by the v-Ki-ras oncogene
23. Optimized Retroviral Transduction Protocol Which Preserves the Primitive Subpopulation of Human Hematopoietic Cells
24. The loss of class II MHC antigen expression byras-transformed murine fibroblasts passaged as tumours correlates with increased tumorigenicity but is not mediated by T cells
25. The AML1-ETO fusion gene promotes extensive self-renewal of human primary erythroid cells
26. Overexpression or Deficiency of RUNX3 Results in Defective Normal Human Hematopoietic Development Similar to RUNX1-ETO Expression in AML
27. PKC-Epsilon Overexpression Is Associated with Poor Outcomes in AML and Promotes Chemoresistance and Hematopoietic Stem Cell Quiescence
28. Integrated nuclear proteomics and transcriptomics identifies S100A4 as a therapeutic target in acute myeloid leukemia
29. Protein kinase C mediates mutant N-Ras–induced developmental abnormalities in normal human erythroid cells
30. Mutant N-RAS Induces Erythroid Lineage Dysplasia in Human CD34+ Cells
31. Use of an anti‐CD200‐blocking antibody improves immune responses to AML in vitro and in vivo.
32. Alternative effects of RAS and RAF oncogenes on the proliferation and apoptosis of factor-dependent FDC-P1 cells
33. Mutant RAS inhibits neutrophil but not macrophage differentiation and allows continued growth of neutrophil precursors
34. A sub-population of primary AML blasts shows resistance to oxidative stress and reduced p38MAPK activation
35. Cord blood-derived quiescent CD34+ cells are more transcriptionally matched to AML blasts than cytokine-induced normal human hematopoietic CD34+ cells
36. Targeting the Ataxia Telangiectasia Mutated-null Phenotype in Chronic Lymphocytic Leukemia with Pro-oxidants
37. Analysis of ROS-Responsive Genes in Mutant RAS Expressing Hematopoietic Progenitors Identifies the Glycolytic Pathway As a Major Target Promoting Both Proliferation and Survival
38. CD200 and PD1-L1 in AML Are Associated with Expanded PD-1+ Late Differentiated CD8+ T Cells and a Decreased CD4:CD8 Ratio: a New Link Between Distinct Immunosuppressive Pathways
39. Integrated Nuclear Proteomics and Transcriptomics Identifies New High Frequency Dysregulated Transcription Factors in AML
40. A Subpopulation of Blasts with Attenuated p38MAPK Response Is Seen in Virtually All AML Patients and AML Cell Lines and Is Defined By Cells with Augmented Lipid-Associated Anti-Oxidant Defense
41. Factors Affecting the Nuclear Localization of β‐Catenin in Normal and Malignant Tissue
42. Identification of the Wnt Signalling Protein, TCF7L2 As a Significantly Overexpressed Transcription Factor in AML
43. PDK1 Overexpression In Acute Myeloid Leukemia; Clinical Significance and Potential as a Therapeutic Target
44. Distinct Regulation of β- and γ-Catenin throughout Hematopoietic Development Contrasts with Their Cooperative Roles In Acute Myeloid Leukemia.
45. Over-Expression of CD200 In Acute Myeloid Leukemia Mediates the Expansion of Regulatory T-Lymphocytes and Directly Inhibits Natural Killer Cell Tumor Immunity
46. Large-Scale Integration of Gene Profiling Identifies TCF7L2/TCF4 as the Most Frequently Dysregulated Wnt Signaling Component In AML
47. Low Connexin Channel-Dependent Intercellular Communication in Human Adult Hematopoietic Progenitor/Stem Cells: Probing Mechanisms of Autologous Stem Cell Therapy
48. Ras Promotes Production of Reactive Oxygen Species in Normal Human CD34+ Cells: Role in Promoting Cell Survival and Protein Phosphorylation
49. Optimized Retroviral Transduction Protocol Which Preserves the Primitive Subpopulation of Human Hematopoietic Cells
50. Expression of RUNX1-RUNX1T1 Alone Has No Effect on the Intrinsic Susceptibility to Cytotoxic Chemicals.
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