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1. Oncogenic role and target properties of the lysine-specific demethylase KDM1A in chronic lymphocytic leukemia

Author

Jiang, Qu, Stachelscheid, Johanna, Bloehdorn, Johannes, Pacholewska, Alicja, Aszyk, Christoph, Grotenhuijs, Francien, Müller, Tony, Onder, Ozlem, Wagle, Prerana, Herling, Carmen D., Kleppe, Maria, Wang, Zhefang, Coombes, Kevin R., Robrecht, Sandra, Dalvi, Priya S., Plosnita, Bianca, Mayer, Petra, Abruzzo, Lynne V., Altmüller, Janine, Gathof, Birgit, Persigehl, Thorsten, Fischer, Kirsten, Jebaraj, Billy, Rienhoff, Hugh Y., Ecker, Rupert, Zhao, Yue, Bruns, Christiane J., Stilgenbauer, Stephan, Elenitoba-Johnson, Kojo, Hallek, Michael, Schweiger, Michal R., Odenthal, Margarete, Vasyutina, Elena, and Herling, Marco

Published
2023
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3. Single Cell Sequencing: A New Dimension in Cancer Diagnosis and Treatment

Author

Wu, Fengying, Fan, Jue, Fang, Jingwen, Dalvi, Priya S., Odenthal, Margarete, Fang, Nan, Crusio, Wim E., Series Editor, Dong, Haidong, Series Editor, Radeke, Heinfried H., Series Editor, Rezaei, Nima, Series Editor, Xiao, Junjie, Series Editor, Yu, Buwei, editor, Zhang, Jiaqiang, editor, Zeng, Yiming, editor, Li, Li, editor, and Wang, Xiangdong, editor

Published
2020
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6. Genome-wide Generation and Systematic Phenotyping of Knockout Mice Reveals New Roles for Many Genes

Author

Baker, Lauren, Barnes, Caroline, Beveridge, Ryan, Cambridge, Emma, Carragher, Damian, Chana, Prabhjoat, Clarke, Kay, Hooks, Yvette, Igosheva, Natalia, Ismail, Ozama, Jackson, Hannah, Kane, Leanne, Lacey, Rosalind, Lafont, David Tino, Lucas, Mark, Maguire, Simon, McGill, Katherine, McIntyre, Rebecca E., Messager, Sophie, Mottram, Lynda, Mulderrig, Lee, Pearson, Selina, Protheroe, Hayley J., Roberson, Laura-Anne, Salsbury, Grace, Sanderson, Mark, Sanger, Daniel, Shannon, Carl, Thompson, Paul C., Tuck, Elizabeth, Vancollie, Valerie E., Brackenbury, Lisa, Bushell, Wendy, Cook, Ross, Dalvi, Priya, Gleeson, Diane, Habib, Bishoy, Hardy, Matt, Liakath-Ali, Kifayathullah, Miklejewska, Evelina, Price, Stacey, Sethi, Debarati, Trenchard, Elizabeth, von Schiller, Dominique, Vyas, Sapna, West, Anthony P., Woodward, John, Wynn, Elizabeth, Evans, Arthur, Gannon, David, Griffiths, Mark, Holroyd, Simon, Iyer, Vivek, Kipp, Christian, Lewis, Morag, Li, Wei, Oakley, Darren, Richardson, David, Smedley, Damian, Agu, Chukwuma, Bryant, Jackie, Delaney, Liz, Gueorguieva, Nadia I., Tharagonnet, Helen, Townsend, Anne J., Biggs, Daniel, Brown, Ellen, Collinson, Adam, Dumeau, Charles-Etienne, Grau, Evelyn, Harrison, Sarah, Harrison, James, Ingle, Catherine E., Kundi, Helen, Madich, Alla, Mayhew, Danielle, Metcalf, Tom, Newman, Stuart, Pass, Johanna, Pearson, Laila, Reynolds, Helen, Sinclair, Caroline, Wardle-Jones, Hannah, Woods, Michael, Alexander, Liam, Brown, Terry, Flack, Francesca, Frost, Carole, Griggs, Nicola, Hrnciarova, Silvia, Kirton, Andrea, McDermott, Jordan, Rogerson, Claire, White, Gemma, Zielezinski, Pawel, DiTommaso, Tia, Edwards, Andrew, Heath, Emma, Mahajan, Mary Ann, Yalcin, Binnaz, White, Jacqueline K., Gerdin, Anna-Karin, Karp, Natasha A., Ryder, Ed, Buljan, Marija, Bussell, James N., Salisbury, Jennifer, Clare, Simon, Ingham, Neil J., Podrini, Christine, Houghton, Richard, Estabel, Jeanne, Bottomley, Joanna R., Melvin, David G., Sunter, David, Adams, Niels C., Tannahill, David, Logan, Darren W., MacArthur, Daniel G., Flint, Jonathan, Mahajan, Vinit B., Tsang, Stephen H., Smyth, Ian, Watt, Fiona M., Skarnes, William C., Dougan, Gordon, Adams, David J., Ramirez-Solis, Ramiro, Bradley, Allan, and Steel, Karen P.

Published
2013
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8. Rapid conversion of EUCOMM/KOMP-CSD alleles in mouse embryos using a cell-permeable Cre recombinase

Author

Ryder, Edward, Doe, Brendan, Gleeson, Diane, Houghton, Richard, Dalvi, Priya, Grau, Evelyn, Habib, Bishoy, Miklejewska, Evelina, Newman, Stuart, Sethi, Debarati, Sinclair, Caroline, Vyas, Sapna, Wardle-Jones, Hannah, Bottomley, Joanna, Bussell, James, Galli, Antonella, Salisbury, Jennifer, Ramirez-Solis, Ramiro, and Sanger Mouse Genetics Project

Published
2014
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9. A resource of targeted mutant mouse lines for 5,061 genes

Author

Birling, Marie-Christine, Yoshiki, Atsushi, Chiani, Francesco, Kaloff, Cornelia, Hörlein, Andreas, Teichmann, Sandy, Tasdemir, Adriane, Krause, Heidi, German, Dorota, Könitzer, Anne, Weber, Sarah, Beig, Joachim, McKay, Matthew, Chin, Hsian-Jean Genie, Bedigian, Richard, Dion, Stephanie, Kutny, Peter, Kelmenson, Jennifer, Perry, Emily, Nguyen-Bresinsky, Dong, Seluke, Audrie, Leach, Timothy, Perkins, Sara, Slater, Amanda, Christou, Skevoulla, Petit, Michaela, Urban, Rachel, Kales, Susan, DaCosta, Michael, McFarland, Michael, Palazola, Rick, Peterson, Kevin A, Svenson, Karen, Braun, Robert E, Taft, Robert, Codner, Gemma F, Rhue, Mark, Garay, Jose, Clary, Dave, Araiza, Renee, Grimsrud, Kristin, Bower, Lynette, Anchell, Nicole L, Jager, Kayla M, Young, Diana L, Dao, Phuong T, DeMayo, Francesco J, Gardiner, Wendy, Bell, Toni, Kenyon, Janet, Stewart, Michelle E, Lynch, Denise, Loeffler, Jorik, Caulder, Adam, Hillier, Rosie, Quwailid, Mohamed M, Zaman, Rumana, Dickinson, Mary E, Santos, Luis, Obata, Yuichi, Iwama, Mizuho, Nakata, Hatsumi, Hashimoto, Tomomi, Kadota, Masayo, Masuya, Hiroshi, Tanaka, Nobuhiko, Miura, Ikuo, Yamada, Ikuko, Doe, Brendan, Furuse, Tamio, Selloum, Mohammed, Jacquot, Sylvie, Ayadi, Abdel, Ali-Hadji, Dalila, Charles, Philippe, Le Marchand, Elise, El Amri, Amal, Kujath, Christelle, Fougerolle, Jean-Victor, Donahue, Leah Rae, Mellul, Peggy, Legeay, Sandrine, Vasseur, Laurent, Moro, Anne-Isabelle, Lorentz, Romain, Schaeffer, Laurence, Dreyer, Dominique, Erbs, Valérie, Eisenmann, Benjamin, Rossi, Giovanni, Fray, Martin D, Luppi, Laurence, Mertz, Annelyse, Jeanblanc, Amélie, Grau, Evelyn, Sinclair, Caroline, Brown, Ellen, Kundi, Helen, Madich, Alla, Woods, Mike, Pearson, Laila, Gambadoro, Alessia, Mayhew, Danielle, Griggs, Nicola, Houghton, Richard, Bussell, James, Ingle, Catherine, Valentini, Sara, Gleeson, Diane, Sethi, Debarati, Bayzetinova, Tanya, Burvill, Jonathan, Adams, David J, Gao, Xiang, Habib, Bishoy, Weavers, Lauren, Maswood, Ryea, Miklejewska, Evelina, Cook, Ross, Platte, Radka, Price, Stacey, Vyas, Sapna, Collinson, Adam, Hardy, Matt, Gertsenstein, Marina, Dalvi, Priya, Iyer, Vivek, West, Tony, Thomas, Mark, Mujica, Alejandro, Sins, Elodie, Barrett, Daniel, Dobbie, Michael, Grobler, Anne, Loots, Glaudina, Gomez-Segura, Alba, Hayeshi, Rose, Scholtz, Liezl-Marie, Bester, Cor, Pheiffer, Wihan, Venter, Kobus, Bosch, Fatima, Goodwin, Leslie O, Heaney, Jason D, Hérault, Yann, de Angelis, Martin Hrabě, Jiang, Si-Tse, Justice, Monica J, Kasparek, Petr, Ayabe, Shinya, King, Ruairidh E, Kühn, Ralf, Lee, Ho, Lee, Youngik, Liu, Zhiwei, Lloyd, K C Kent, Lorenzo, Isabel, Mallon, Ann-Marie, McKerlie, Colin, Meehan, Terrence F, Beaudet, Arthur L, Fuentes, Violeta Munoz, Newman, Stuart, Nutter, Lauryl M J, Oh, Goo Taeg, Pavlovic, Guillaume, Ramirez-Solis, Ramiro, Rosen, Barry, Ryder, Edward J, Santos, Luis A, Schick, Joel, Bottomley, Joanna, Seavitt, John R, Sedlacek, Radislav, Seisenberger, Claudia, Seong, Je Kyung, Skarnes, William C, Sorg, Tania, Steel, Karen P, Tamura, Masaru, Tocchini-Valentini, Glauco P, Wang, Chi-Kuang Leo, Bradley, Allan, Wardle-Jones, Hannah, Wattenhofer-Donzé, Marie, Wells, Sara, Wiles, Michael V, Willis, Brandon J, Wood, Joshua A, Wurst, Wolfgang, Xu, Ying, Consortium, International Mouse Phenotyping, Teboul, Lydia, Brown, Steve D M, Murray, Stephen A, Gallegos, Juan J, Green, Jennie R, Bohat, Ritu, Zimmel, Katie, Pereira, Monica, MacMaster, Suzanne, Tondat, Sandra, Wei, Linda, Carroll, Tracy, Bürger, Antje, Cabezas, Jorge, Fan-Lan, Qing, Jacob, Elsa, Creighton, Amie, Castellanos-Penton, Patricia, Danisment, Ozge, Clarke, Shannon, Joeng, Joanna, Kelly, Deborah, To, Christine, Bushell, Wendy, van Bruggen, Rebekah, Gailus-Durner, Valerie, Fuchs, Helmut, Marschall, Susan, Dunst, Stefanie, Romberger, Markus, Rey, Bernhard, Fessele, Sabine, Gormanns, Philipp, Friedel, Roland, Institut de Génétique et de Biologie Moléculaire et Cellulaire (IGBMC), and Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

Subjects
Cancer Research, Genotype, Knockout, International Cooperation, [SDV]Life Sciences [q-bio], Mutant, Mutagenesis (molecular biology technique), cytology [Mouse Embryonic Stem Cells], Mouse Mutant Strains, Biology, Genome, Medical and Health Sciences, Article, 03 medical and health sciences, Mice, 0302 clinical medicine, Research community, ddc:570, Genetics, Animals, Allele, Gene, Genetic Association Studies, 030304 developmental biology, Mice, Knockout, 0303 health sciences, Internet, metabolism [Mouse Embryonic Stem Cells], Information Dissemination, mutant mouse IMPC, Mouse Embryonic Stem Cells, Biological Sciences, Null allele, Embryonic stem cell, Phenotype, Mutagenesis, Montertondo Mouse Production, Technology Platforms, International Mouse Phenotyping Consortium, Function (biology), 030217 neurology & neurosurgery, Gene Deletion, Developmental Biology
Abstract

The International Mouse Phenotyping Consortium reports the generation of new mouse mutant strains for over 5,000 genes from targeted embryonic stem cells on the C57BL/6N genetic background. This includes 2,850 null alleles for which no equivalent mutant mouse line exists, 2,987 novel conditional-ready alleles, and 4,433 novel reporter alleles. This nearly triples the number of genes with reporter alleles and almost doubles the number of conditional alleles available to the scientific community. When combined with more than 30 years of community effort, the total mutant allele mouse resource covers more than half of the genome. The extensively validated collection is archived and distributed through public repositories, facilitating availability to the worldwide biomedical research community, and expanding our understanding of gene function and human disease.

Published
2021
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11. Expression profiling on subclasses of primary parotid gland carcinomas

Author

Meinrath, Jeannine, primary, Haak, Anja, additional, Igci, Nesrin, additional, Dalvi, Priya, additional, Arolt, Christoph, additional, Meemboor, Sonja, additional, Siebolts, Udo, additional, Eischeidt-Scholz, Hannah, additional, Wickenhauser, Claudia, additional, Grünewald, Inga, additional, Drebber, Uta, additional, Büttner, Reinhard, additional, Quaas, Alexander, additional, Klußmann, Jens-Peter, additional, Odenthal, Margarete, additional, Beutner, Dirk, additional, and Meyer, Moritz, additional

Published
2020
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12. LSD1 Inhibition Attenuates Tumor Growth by Disrupting PLK1 Mitotic Pathway

Author

Dalvi, Priya S., Kacheleidt, Iris F., Lim, So-Young, Meemboor, Sonja, Mueller, Marion, Eischeid-Scholz, Hannah, Schaefer, Stephan C., Buettner, Reinhard, Klein, Sebastian, Odenthal, Margarete, Dalvi, Priya S., Kacheleidt, Iris F., Lim, So-Young, Meemboor, Sonja, Mueller, Marion, Eischeid-Scholz, Hannah, Schaefer, Stephan C., Buettner, Reinhard, Klein, Sebastian, and Odenthal, Margarete

Abstract

Lysine-specific demethylase 1 (LSD1) is a histone modifier that is highly overexpressed in lung adenocarcinoma, which results in aggressive tumor biology. Tumor cell proliferation and migration analysis after LSD1 inhibition in the lung adenocarcinoma cell line PC9, using the LSD1 inhibitor HCI-2509 and siRNA, demonstrated that LSD1 activity was essential for proliferation and migration capacities of tumor cells. Moreover, reduced proliferation rates after LSD1 inhibition were shown to be associated with a cell-cycle arrest of the tumor cells in the G2-M-phase. Expression profiling followed by functional classification and pathway analysis indicated prominent repression of the polo-like kinase 1 (PLK1) pathway upon LSD1 inhibition. In contrast, transient overexpression of exogenous PLK1 plasmid rescued the LSD1 inhibition-mediated downregulation of PLK1 pathway genes. Mechanistically, LSD1 directly regulates expression of PLK1 by binding to its promoter region that subsequently affects expression of its downstream target genes. Notably, using lung adenocarcinoma TCGA datasets a significant correlation between LSD1 and PLK1 along with its downstream targets was observed. Furthermore, the LSD1/ PLK1 linkage was confirmed by IHC analysis in a clinical lung adenocarcinoma cohort (n 1/4 43). Conclusively, this is the first study showing a direct transcriptional link between LSD1 and PLK1. Implications: These findings point to a role of LSD1 in regulating PLK1 and thus efficient G2-M-transitionmediating proliferation of tumor cells and suggest targeting the LSD1/ PLK1 axis as a novel therapeutic approach for lung adenocarcinoma treatment.

Published
2019

14. Yoga: Can it be integrated with treatment of neuropathic pain?

Author

Telles, Shirley, primary, Sayal, Natasha, additional, Nacht, Carrie, additional, Chopra, Abhishek, additional, Patel, Kush, additional, Wnuk, Alice, additional, Dalvi, Priya, additional, Bhatia, Kulsajan, additional, Miranpuri, Gurwattan, additional, and Anand, Akshay, additional

Published
2019
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15. MP73-13 COMPREHENSIVE ANALYSIS OF METASTATIC SEMINOMA GERM CELL TUMORS SHOWS DIVERGENT EXPRESSION OF IMMUNE-RELATED PATHWAYS

Author

Nestler*, Tim, primary, Haidl, Friederike, additional, Wittersheim, Maike, additional, Dalvi, Priya, additional, Paffenholz, Pia, additional, Wagener-Ryczek, Svenja, additional, Pfister, David, additional, Hellmich, Martin, additional, Büttner, Reinhard, additional, Odenthal, Margarete, additional, and Heidenreich, Axel, additional

Published
2019
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16. Seminomatous germ cell tumors are heterogenic and metastasis can be predicted based on a gene signature of the tumor invasive front.

Author

Nestler, Tim, primary, Haidl, Friederike, additional, Wittersheim, Maike, additional, Dalvi, Priya, additional, Paffenholz, Pia, additional, Wagener-Ryczek, Svenja, additional, Pfister, David J. K. P., additional, Hellmich, Martin, additional, Büttner, Reinhard, additional, Odenthal, Margarete, additional, and Heidenreich, Axel, additional

Published
2019
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17. Comprehensive analysis of metastatic seminoma germ cell tumors shows divergent expression of immune-related pathways.

Author

Nestler, Tim, primary, Haidl, Friederike, additional, Wittersheim, Maike, additional, Dalvi, Priya, additional, Paffenholz, Pia, additional, Wagener-Ryczek, Svenja, additional, Pfister, David J. K. P., additional, Hellmich, Martin, additional, Büttner, Reinhard, additional, Odenthal, Margarete, additional, and Heidenreich, Axel, additional

Published
2019
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18. LSD1 modulates the non-canonical integrin beta 3 signaling pathway in non-small cell lung carcinoma cells (vol 7, 10292, 2017)

Author

Lim, So-Young, Macheleidt, Iris, Dalvi, Priya, Schaefer, Stephan C., Kerick, Martin, Ozretic, Luka, Ortiz-Cuaran, Sandra, George, Julie, Merkelbach-Bruse, Sabine, Wolf, Juergen, Timmermann, Bernd, Thomas, Roman K., Schweiger, Michal R., Buettner, Reinhard, Odenthal, Margarete, Lim, So-Young, Macheleidt, Iris, Dalvi, Priya, Schaefer, Stephan C., Kerick, Martin, Ozretic, Luka, Ortiz-Cuaran, Sandra, George, Julie, Merkelbach-Bruse, Sabine, Wolf, Juergen, Timmermann, Bernd, Thomas, Roman K., Schweiger, Michal R., Buettner, Reinhard, and Odenthal, Margarete

Published
2018

19. Preclinical studies reveal that LSD1 inhibition results in tumor growth arrest in lung adenocarcinoma independently of driver mutations

Author

Macheleidt, Iris F., Dalvi, Priya S., Lim, So-Young, Meemboor, Sonja, Meder, Lydia, Kaesgen, Olivia, Mueller, Marion, Kleemann, Karolin, Wang, Lingyu, Nuernberg, Peter, Ruesseler, Vanessa, Schaefer, Stephan C., Mahabir, Esther, Buettner, Reinhard, Odenthal, Margarete, Macheleidt, Iris F., Dalvi, Priya S., Lim, So-Young, Meemboor, Sonja, Meder, Lydia, Kaesgen, Olivia, Mueller, Marion, Kleemann, Karolin, Wang, Lingyu, Nuernberg, Peter, Ruesseler, Vanessa, Schaefer, Stephan C., Mahabir, Esther, Buettner, Reinhard, and Odenthal, Margarete

Abstract

Lung adenocarcinoma (LUAD) is the most prevalent subtype of non-small cell lung cancer. Despite the development of novel targeted and immune therapies, the 5-year survival rate is still only 21%, indicating the need for more efficient treatment regimens. Lysine-specific demethylase 1 (LSD1) is an epigenetic eraser that modifies histone 3 methylation status, and is highly overexpressed in LUAD. Using representative human cell culture systems and two autochthonous transgenic mouse models, we investigated inhibition of LSD1 as a novel therapeutic option for treating LUAD. The reversible LSD1 inhibitor HCI-2509 significantly reduced cell growth with an IC50 of 0.3-5 mu min vitro, which was linked to an enhancement of histone 3 lysine methylation. Most importantly, growth arrest, as well as inhibition of the invasion capacities, was independent of the underlying driver mutations. Subsequent expression profiling revealed that the cell cycle and replication machinery were prominently affected after LSD1 inhibition. In addition, our data provide evidence that LSD1 blockade significantly interferes with EGFR downstream signaling. Finally, our in vitro results were confirmed by preclinical therapeutic approaches, including the use of two autochthonous transgenic LUAD mouse models driven by either EGFR or KRAS mutations. Importantly, LSD1 inhibition resulted in significantly lower tumor formation and a strong reduction in tumor progression, which were independent of the underlying mutational background of the mouse models. Hence, our findings provide substantial evidence indicating that tumor growth of LUAD can be markedly decreased by HCI-2509 treatment, suggesting its use as a single agent maintenance therapy or combined therapeutical application in novel concerted drug approaches.

Published
2018

21. Preclinical studies reveal that LSD1 inhibition results in tumor growth arrest in lung adenocarcinoma independently of driver mutations

Author

Macheleidt, Iris F., primary, Dalvi, Priya S., additional, Lim, So‐Young, additional, Meemboor, Sonja, additional, Meder, Lydia, additional, Käsgen, Olivia, additional, Müller, Marion, additional, Kleemann, Karolin, additional, Wang, Lingyu, additional, Nürnberg, Peter, additional, Rüsseler, Vanessa, additional, Schäfer, Stephan C., additional, Mahabir, Esther, additional, Büttner, Reinhard, additional, and Odenthal, Margarete, additional

Published
2018
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22. LSD1 modulates the non-canonical integrin beta 3 signaling pathway in non-small cell lung carcinoma cells

Author

Lim, So-Young, Macheleidt, Iris, Dalvi, Priya, Schaefer, Stephan C., Kerick, Martin, Ozretic, Luka, Ortiz-Cuaran, Sandra, George, Julie, Merkelbach-Bruse, Sabine, Wolf, Juergen, Timmermann, Bernd, Thomas, Roman K., Schweiger, Michal R., Buettner, Reinhard, Odenthal, Margarete, Lim, So-Young, Macheleidt, Iris, Dalvi, Priya, Schaefer, Stephan C., Kerick, Martin, Ozretic, Luka, Ortiz-Cuaran, Sandra, George, Julie, Merkelbach-Bruse, Sabine, Wolf, Juergen, Timmermann, Bernd, Thomas, Roman K., Schweiger, Michal R., Buettner, Reinhard, and Odenthal, Margarete

Abstract

The epigenetic writer lysine-specific demethylase 1 (LSD1) is aberrantly upregulated in many cancer types and its overexpression correlates with poor survival and tumor progression. In this study, we analysed LSD1 function in non-small cell lung cancer adenocarcinomas. Expression profiling of 182 cases of lung adenocarcinoma proved a significant correlation of LSD1 overexpression with lung adenocarcinoma progression and metastasis. KRAS-mutated lung cancer cell clones were stably silenced for LSD1 expression. RNA-seq and comprehensive pathway analysis revealed, that genes related to a recently described non-canonical integrin beta 3 pathway, were significantly downregulated by LSD1 silencing. Hence, invasion and self-renewal capabilities were strongly decreased. Notably, this novel defined LSD1/integrin beta 3 axis, was also detected in human lung adenocarcinoma specimens. Furthermore, the linkage of LSD1 to an altered expression pattern of lung-lineage specific transcription factors and genes, which are involved in alveolar epithelial differentiation, was demonstrated. Thus, our findings point to a LSD1-integrin beta 3 axis, conferring attributes of invasiveness and tumor progression to lung adenocarcinoma.

Published
2017

23. Yoga: Can It Be Integrated with Treatment of Neuropathic Pain

Author

Telles, Shirley, primary, Sayal, Natasha, additional, Nacht, Carrie, additional, Chopra, Abhishek, additional, Patel, Kush, additional, Wnuk, Alice, additional, Dalvi, Priya, additional, Bhatia, Kulsajan, additional, Miranpuri, Gurwattan, additional, and Anand, Akshay, additional

Published
2017
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24. Genome-wide Generation and Systematic Phenotyping of Knockout Mice Reveals New Roles for Many Genes

Author

White, Jacqueline K., primary, Gerdin, Anna-Karin, additional, Karp, Natasha A., additional, Ryder, Ed, additional, Buljan, Marija, additional, Bussell, James N., additional, Salisbury, Jennifer, additional, Clare, Simon, additional, Ingham, Neil J., additional, Podrini, Christine, additional, Houghton, Richard, additional, Estabel, Jeanne, additional, Bottomley, Joanna R., additional, Melvin, David G., additional, Sunter, David, additional, Adams, Niels C., additional, Tannahill, David, additional, Logan, Darren W., additional, MacArthur, Daniel G., additional, Flint, Jonathan, additional, Mahajan, Vinit B., additional, Tsang, Stephen H., additional, Smyth, Ian, additional, Watt, Fiona M., additional, Skarnes, William C., additional, Dougan, Gordon, additional, Adams, David J., additional, Ramirez-Solis, Ramiro, additional, Bradley, Allan, additional, Steel, Karen P., additional, Baker, Lauren, additional, Barnes, Caroline, additional, Beveridge, Ryan, additional, Cambridge, Emma, additional, Carragher, Damian, additional, Chana, Prabhjoat, additional, Clarke, Kay, additional, Hooks, Yvette, additional, Igosheva, Natalia, additional, Ismail, Ozama, additional, Jackson, Hannah, additional, Kane, Leanne, additional, Lacey, Rosalind, additional, Lafont, David Tino, additional, Lucas, Mark, additional, Maguire, Simon, additional, McGill, Katherine, additional, McIntyre, Rebecca E., additional, Messager, Sophie, additional, Mottram, Lynda, additional, Mulderrig, Lee, additional, Pearson, Selina, additional, Protheroe, Hayley J., additional, Roberson, Laura-Anne, additional, Salsbury, Grace, additional, Sanderson, Mark, additional, Sanger, Daniel, additional, Shannon, Carl, additional, Thompson, Paul C., additional, Tuck, Elizabeth, additional, Vancollie, Valerie E., additional, Brackenbury, Lisa, additional, Bushell, Wendy, additional, Cook, Ross, additional, Dalvi, Priya, additional, Gleeson, Diane, additional, Habib, Bishoy, additional, Hardy, Matt, additional, Liakath-Ali, Kifayathullah, additional, Miklejewska, Evelina, additional, Price, Stacey, additional, Sethi, Debarati, additional, Trenchard, Elizabeth, additional, von Schiller, Dominique, additional, Vyas, Sapna, additional, West, Anthony P., additional, Woodward, John, additional, Wynn, Elizabeth, additional, Evans, Arthur, additional, Gannon, David, additional, Griffiths, Mark, additional, Holroyd, Simon, additional, Iyer, Vivek, additional, Kipp, Christian, additional, Lewis, Morag, additional, Li, Wei, additional, Oakley, Darren, additional, Richardson, David, additional, Smedley, Damian, additional, Agu, Chukwuma, additional, Bryant, Jackie, additional, Delaney, Liz, additional, Gueorguieva, Nadia I., additional, Tharagonnet, Helen, additional, Townsend, Anne J., additional, Biggs, Daniel, additional, Brown, Ellen, additional, Collinson, Adam, additional, Dumeau, Charles-Etienne, additional, Grau, Evelyn, additional, Harrison, Sarah, additional, Harrison, James, additional, Ingle, Catherine E., additional, Kundi, Helen, additional, Madich, Alla, additional, Mayhew, Danielle, additional, Metcalf, Tom, additional, Newman, Stuart, additional, Pass, Johanna, additional, Pearson, Laila, additional, Reynolds, Helen, additional, Sinclair, Caroline, additional, Wardle-Jones, Hannah, additional, Woods, Michael, additional, Alexander, Liam, additional, Brown, Terry, additional, Flack, Francesca, additional, Frost, Carole, additional, Griggs, Nicola, additional, Hrnciarova, Silvia, additional, Kirton, Andrea, additional, McDermott, Jordan, additional, Rogerson, Claire, additional, White, Gemma, additional, Zielezinski, Pawel, additional, DiTommaso, Tia, additional, Edwards, Andrew, additional, Heath, Emma, additional, Mahajan, Mary Ann, additional, and Yalcin, Binnaz, additional

Published
2013
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