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1. Creeping Fat–Derived Free Fatty Acids Induce Hyperplasia of Intestinal Muscularis Propria Muscle Cells: A Novel Link Between Fat and Intestinal Stricture Formation in Crohn’s Disease

2. Human intestinal myofibroblasts deposited collagen VI enhances adhesiveness for T cells – A novel mechanism for maintenance of intestinal inflammation

4. Milk fat globule-epidermal growth factor 8 (MFGE8) prevents intestinal fibrosis

5. Milk fat globule-epidermal growth factor 8 (MFGE8) prevents intestinal fibrosis.

8. 1142 INTEGRIN INHIBITION REDUCES PROFIBROTIC RESPONSES IN CROHN'S DISEASE (CD) HUMAN INTESTINAL MYOFIBROBLASTS IN VITRO AND AMELIORATES EXPERIMENTAL INTESTINAL FIBROSIS IN VIVO - A POTENTIAL NOVEL APPROACH TO CD STRICTURE THERAPY

9. Su479 FIBROSTENOTIC CROHN'S DISEASE MESENCHYMAL CELLS ARE RESISTANT TO THE ANTI-FIBROTIC ACTIVITY OF MILK FAT GLOBULE-EGF FACTOR 8 (MFGE8)

10. Tu1246 CROHN'S DISEASE STRICTURE MATRISOME ANALYSIS REVEALS THE ANTI-FIBROTIC ACTIVITY OF MILK-FAT GLOBULE-EPIDERMAL GROWTH FACTOR 8 (MFGE8)

11. 951 CREEPING FAT-DERIVED LONG CHAIN FREE FATTY ACIDS DRIVE INTESTINAL MUSCULARIS PROPRIA MUSCLE CELL PROLIFERATION VIA LIPID METABOLISM AND CARNITINE PALMITOYLTRANSFERASE 1 (CPT-1) - A RELEVANT MECHANISM FOR STRICTURING CROHN'S DISEASE

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