399 results on '"Cross, Justin R."'
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2. A genetically encoded tool to increase cellular NADH/NAD+ ratio in living cells
3. Altered microbial bile acid metabolism exacerbates T cell-driven inflammation during graft-versus-host disease
4. Autotaxin–lysolipid signaling suppresses a CCL11–eosinophil axis to promote pancreatic cancer progression
5. Distinct metabolic requirements regulate B cell activation and germinal center responses
6. Connecting copper and cancer: from transition metal signalling to metalloplasia
7. Reporting NADPH fluxes
8. Protein folding stress potentiates NLRP1 and CARD8 inflammasome activation
9. M24B aminopeptidase inhibitors selectively activate the CARD8 inflammasome
10. Alloreactive T cells deficient of the short-chain fatty acid receptor GPR109A induce less graft-versus-host disease
11. Characterization, structure and inhibition of the human succinyl-CoA:glutarate-CoA transferase, a genetic modifier of glutaric aciduria type 1
12. Characterization, Structure, and Inhibition of the Human Succinyl-CoA:glutarate-CoA Transferase, a Putative Genetic Modifier of Glutaric Aciduria Type 1.
13. A High-Fiber Dietary Intervention (NUTRIVENTION) in Precursor Plasma Cell Disorders Improves Disease Biomarkers and Delays Progression to Myeloma
14. Copper depletion modulates mitochondrial oxidative phosphorylation to impair triple negative breast cancer metastasis
15. Transsulfuration Activity Can Support Cell Growth upon Extracellular Cysteine Limitation
16. Metabolic adaptation of acute lymphoblastic leukemia to the central nervous system microenvironment depends on stearoyl-CoA desaturase
17. Impaired mitochondrial oxidative phosphorylation limits the self-renewal of T cells exposed to persistent antigen
18. Microbiota modulate sympathetic neurons via a gut–brain circuit
19. Bacterial metabolism of bile acids promotes generation of peripheral regulatory T cells
20. A genetically encoded tool to increase cellular NADH/NAD+ ratio in living cells
21. Impact of gut colonization with butyrate-producing microbiota on respiratory viral infection following allo-HCT
22. Microbiota-derived lantibiotic restores resistance against vancomycin-resistant Enterococcus
23. In Vivo Imaging of Glutamine Metabolism to the Oncometabolite 2-Hydroxyglutarate in IDH1/2 Mutant Tumors
24. Capacitation induces changes in metabolic pathways supporting motility of epididymal and ejaculated sperm
25. Stable Isotope Tracers for Metabolic Pathway Analysis
26. Comparison of Topical and Intravenous Tranexamic Acid for Total Knee Replacement: A Randomized Double-Blinded Controlled Study of Effects on Tranexamic Acid Levels and Thrombogenic and Inflammatory Marker Levels
27. Acquired resistance to IDH inhibition through trans or cis dimer-interface mutations
28. Data from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
29. Supplementary Table 1 from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
30. Data from Clinical and Biological Correlates of Neurotoxicity Associated with CAR T-cell Therapy in Patients with B-cell Acute Lymphoblastic Leukemia
31. Figure S1, Figure S2, Figure S3, Figure S4 from Clinical and Biological Correlates of Neurotoxicity Associated with CAR T-cell Therapy in Patients with B-cell Acute Lymphoblastic Leukemia
32. Supplementary Figure 5 from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
33. Supplementary Figure 2 from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
34. Supplementary Figure 4 from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
35. Supplementary Figure 1B from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
36. Table S1, Table S2, Table S3, Table S4 from Clinical and Biological Correlates of Neurotoxicity Associated with CAR T-cell Therapy in Patients with B-cell Acute Lymphoblastic Leukemia
37. Supplementary Figure 3 from Serine Catabolism Regulates Mitochondrial Redox Control during Hypoxia
38. Supplemental Figure 5 from Mitochondrial Inhibition Augments the Efficacy of Imatinib by Resetting the Metabolic Phenotype of Gastrointestinal Stromal Tumor
39. Supplemental Figure 1 from Mitochondrial Inhibition Augments the Efficacy of Imatinib by Resetting the Metabolic Phenotype of Gastrointestinal Stromal Tumor
40. Supplemental Fig.1-4;Supplemental Tables 1-3; Suipplemental Methods from Molecular and Clinical Effects of Notch Inhibition in Glioma Patients: A Phase 0/I Trial
41. Supplemental Figure 3 from Mitochondrial Inhibition Augments the Efficacy of Imatinib by Resetting the Metabolic Phenotype of Gastrointestinal Stromal Tumor
42. Supplemental Figure 4 from Mitochondrial Inhibition Augments the Efficacy of Imatinib by Resetting the Metabolic Phenotype of Gastrointestinal Stromal Tumor
43. Supplemental Figure 2 from Mitochondrial Inhibition Augments the Efficacy of Imatinib by Resetting the Metabolic Phenotype of Gastrointestinal Stromal Tumor
44. Microbial Bile Acid Metabolism Shapes Effector T Cell Responses during Graft-Versus-Host Disease in Mouse and Human
45. Acyl‐CoA dehydrogenase substrate promiscuity: Challenges and opportunities for development of substrate reduction therapy in disorders of valine and isoleucine metabolism.
46. Intestinal Blautia Is Associated with Reduced Death from Graft-versus-Host Disease
47. A Pilot Plant Based Dietary Intervention in MGUS and SMM Patients with Elevated BMI Is Feasible and Associated with Improvements in Metabolic and Microbiome Biomarkers of Progression
48. A Randomized Placebo Controlled Study of a Plant-Based Dietary Versus Supplement Versus Placebo Intervention in Patients with Monoclonal Gammopathy of Undetermined Significance (MGUS) and Smoldering Multiple Myeloma (SMM) - the Nutrition Prevention (NUTRIVENTION-3) Study
49. Fructose Treatment Targets Myeloid Leukemogenesis in IDH2 Mutants By Exhausting Alpha-Ketoglutarate Pools
50. Ogdh Is a Genetic Vulnerability and Therapeutic Target in Acute Myeloid Leukemia
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