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2. Innate Immune Activation and Mitochondrial ROS Invoke Persistent Cardiac Conduction System Dysfunction after COVID-19

4. Tendon Tissue-Engineering Scaffolds

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7. Uncompensated mitochondrial oxidative stress underlies heart failure in an iPSC-derived model of congenital heart disease

9. iPSC modeling shows uncompensated mitochondrial mediated oxidative stress underlies early heart failure in hypoplastic left heart syndrome

17. Innate Immune Activation and Mitochondrial ROS Invoke Persistent Cardiac Conduction System Dysfunction after COVID-19.

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