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1. Common Cold Coronavirus 229E Induces Higher Interferon Stimulating Gene Responses in Human Nasal Epithelial Cells from Patients with Chronic Rhinosinusitis with Polyposis.

2. Human Coronavirus 229E Uses Clathrin-Mediated Endocytosis as a Route of Entry in Huh-7 Cells.

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3. Seasonal human coronaviruses OC43, 229E, and NL63 induce cell surface modulation of entry receptors and display host cell-specific viral replication kinetics.

4. Human Coronavirus 229E Infection Inactivates Pyroptosis Executioner Gasdermin D but Ultimately Leads to Lytic Cell Death Partly Mediated by Gasdermin E.

5. Human coronavirus 229E inactivation on porous and nonporous materials using dielectric barrier discharge (DBD) plasma.

6. Electromagnetic deactivation spectroscopy of human coronavirus 229E.

7. Structural basis of main proteases of HCoV-229E bound to inhibitor PF-07304814 and PF-07321332.

8. Continuously active disinfectant inactivates severe acute respiratory coronavirus virus 2 (SARS-CoV-2) and human coronavirus 229E two days after the disinfectant was applied and following wear exposures.

9. Flavonols and dihydroflavonols inhibit the main protease activity of SARS-CoV-2 and the replication of human coronavirus 229E.

10. Viral polymerase binding and broad-spectrum antiviral activity of molnupiravir against human seasonal coronaviruses.

11. Comparative genomic analysis reveals varying levels of mammalian adaptation to coronavirus infections.

12. Impacts of p97 on Proteome Changes in Human Cells during Coronaviral Replication.

13. Improved estimates of 222 nm far-UVC susceptibility for aerosolized human coronavirus via a validated high-fidelity coupled radiation-CFD code.

14. Multi-level inhibition of coronavirus replication by chemical ER stress.

15. Pulsed blue light inactivates two strains of human coronavirus.

16. Antiviral Activity of Umifenovir In Vitro against a Broad Spectrum of Coronaviruses, Including the Novel SARS-CoV-2 Virus.

17. Coronavirus Pseudotypes for All Circulating Human Coronaviruses for Quantification of Cross-Neutralizing Antibody Responses.

18. SPR-Based Kinetic Analysis of the Early Stages of Infection in Cells Infected with Human Coronavirus and Treated with Hydroxychloroquine.

19. Reactive T Cells in Convalescent COVID-19 Patients With Negative SARS-CoV-2 Antibody Serology.

20. TMEM41B is a host factor required for the replication of diverse coronaviruses including SARS-CoV-2.

21. EGCG, a green tea polyphenol, inhibits human coronavirus replication in vitro.

22. Transcriptomic profiling and genomic mutational analysis of Human coronavirus (HCoV)-229E -infected human cells.

23. Resveratrol Inhibits HCoV-229E and SARS-CoV-2 Coronavirus Replication In Vitro.

24. Coronavirus replication-transcription complex: Vital and selective NMPylation of a conserved site in nsp9 by the NiRAN-RdRp subunit.

25. Natural and Nature-Derived Products Targeting Human Coronaviruses.

26. Cryo-EM analysis of the HCoV-229E spike glycoprotein reveals dynamic prefusion conformational changes.

27. Genome-Scale Identification of SARS-CoV-2 and Pan-coronavirus Host Factor Networks.

28. Protease Inhibitors: Candidate Drugs to Inhibit Severe Acute Respiratory Syndrome Coronavirus 2 Replication.

29. Influences of cyclosporin A and non-immunosuppressive derivatives on cellular cyclophilins and viral nucleocapsid protein during human coronavirus 229E replication.

30. Functional Carbon Quantum Dots as Medical Countermeasures to Human Coronavirus.

31. Establishment of Primary Transgenic Human Airway Epithelial Cell Cultures to Study Respiratory Virus-Host Interactions.

32. Identification and Characterization of a Human Coronavirus 229E Nonstructural Protein 8-Associated RNA 3'-Terminal Adenylyltransferase Activity.

33. Characterization of the Lipidomic Profile of Human Coronavirus-Infected Cells: Implications for Lipid Metabolism Remodeling upon Coronavirus Replication.

34. Crystal structure of the post-fusion core of the Human coronavirus 229E spike protein at 1.86 Å resolution.

35. Receptor-binding loops in alphacoronavirus adaptation and evolution.

36. Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

37. Genetic deficiency and polymorphisms of cyclophilin A reveal its essential role for Human Coronavirus 229E replication.

38. Screening of an FDA-approved compound library identifies four small-molecule inhibitors of Middle East respiratory syndrome coronavirus replication in cell culture.

39. The ORF4a protein of human coronavirus 229E functions as a viroporin that regulates viral production.

40. BST2/CD317 counteracts human coronavirus 229E productive infection by tethering virions at the cell surface.

41. TMPRSS2 activates the human coronavirus 229E for cathepsin-independent host cell entry and is expressed in viral target cells in the respiratory epithelium.

42. Tropism of and innate immune responses to the novel human betacoronavirus lineage C virus in human ex vivo respiratory organ cultures.

43. SARS-CoV regulates immune function-related gene expression in human monocytic cells.

44. A human coronavirus responsible for the common cold massively kills dendritic cells but not monocytes.

45. Replication of human coronaviruses SARS-CoV, HCoV-NL63 and HCoV-229E is inhibited by the drug FK506.

46. Blocking eIF4E-eIF4G interaction as a strategy to impair coronavirus replication.

47. Human coronaviruses 229E and NL63: close yet still so far.

48. Protease-mediated entry via the endosome of human coronavirus 229E.

49. Human coronavirus 229E papain-like proteases have overlapping specificities but distinct functions in viral replication.

50. Human coronavirus 229E encodes a single ORF4 protein between the spike and the envelope genes.