40 results on '"Cohen, Shenhav"'
Search Results
2. The Trans-Golgi Network-Associated Human Ubiquitin-Protein Ligase POSH Is Essential for HIV Type 1 Production
3. Myofibril breakdown during atrophy is a delayed response requiring the transcription factor PAX4 and desmin depolymerization
4. A signaling hub of insulin receptor, dystrophin glycoprotein complex and plakoglobin regulates muscle size
5. Profiling of the muscle-specific dystroglycan interactome reveals the role of Hippo signaling in muscular dystrophy and age-dependent muscle atrophy
6. Proteasome gene expression is controlled by the coordinated functions of multiple transcription factors
7. The extracellular matrix protein agrin promotes heart regeneration in mice
8. The AAA‐ATPase ATAD1 and its partners promote degradation of desmin intermediate filaments in muscle
9. Insulin receptor turnover in fasting is dependent on NAGLU-mediated β-dystroglycan deglycosylation
10. Author Reply to Peer Reviews of The AAA-ATPase ATAD1 and its partners promote degradation of desmin intermediate filaments in muscle
11. Reply to Kissane and Eggington
12. A semiautomated measurement of muscle fiber size using the Imaris software
13. Novel Signaling Hub of Insulin Receptor Dystrophin Glycoprotein Complex and Plakoglobin Regulates Muscle Size
14. Supplementary macro excel files for calculation of cell size
15. Supplementary material for Gilda et al. 2021, AJP-Cell Physiol
16. During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation
17. New roles for desmin in the maintenance of muscle homeostasis
18. New roles for desmin in the maintenance of muscle homeostasis.
19. Breakdown of Filamentous Myofibrils by the UPS–Step by Step
20. Role of calpains in promoting desmin filaments depolymerization and muscle atrophy
21. USP 1 deubiquitinates Akt to inhibit PI 3K‐Akt‐FoxO signaling in muscle during prolonged starvation
22. JAK–STAT signaling involved in phorbol 12-myristate 13-acetate- and dimethyl sulfoxide-induced 2′-5′ oligoadenylate synthetase expression in human HL-60 leukemia cells
23. USP1 deubiquitinates protein kinase Akt to inhibit PI3K-Akt-FoxO signaling
24. Novel signaling hub of insulin receptor, dystrophin glycoprotein complex and plakoglobin regulates muscle size
25. Profiling of the Muscle-Specific Dystroglycan Complexome Identifies Novel Muscular Dystrophy Factors
26. USP1 deubiquitinates Akt to inhibit PI3K‐Akt‐FoxO signaling in muscle during prolonged starvation.
27. GSK3-β promotes calpain-1–mediated desmin filament depolymerization and myofibril loss in atrophy
28. Non-canonical activation of DAPK2 by AMPK constitutes a new pathway linking metabolic stress to autophagy
29. Muscle wasting in disease: molecular mechanisms and promising therapies
30. VWA domain of S5a restricts the ability to bind ubiquitin and Ubl to the 26S proteasome
31. Trim32 reduces PI3K–Akt–FoxO signaling in muscle atrophy by promoting plakoglobin–PI3K dissociation
32. Ubiquitylation by Trim32 causes coupled loss of desmin, Z-bands, and thin filaments in muscle atrophy
33. During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation
34. Muscle wasting in disease: molecular mechanisms and promising therapies.
35. Expression of a PKR Dominant-Negative Mutant in Myogenic Cells Interferes with the Myogenic Process
36. During muscle atrophy, thick, but not thin, filament components are degraded by MuRF 1-dependent ubiquitylation.
37. During Muscle Atrophy, Thick, but not Thin, Filament Components are Degraded by MuRF1-Dependent Ubiquitylation
38. Proteasome gene expression is controlled by the coordinated functions of multiple transcription factors.
39. Reply to Kissane and Eggington.
40. A semiautomated measurement of muscle fiber size using the Imaris software.
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