35 results on '"Cilleros-Holgado, Paula"'
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2. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases
3. Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels
4. A Multi-Target Pharmacological Correction of a Lipoyltransferase LIPT1 Gene Mutation in Patient-Derived Cellular Models.
5. Polydatin and Nicotinamide Rescue the Cellular Phenotype of Mitochondrial Diseases by Mitochondrial Unfolded Protein Response (mtUPR) Activation.
6. Mitochondrial Unfolded Protein Response (Mtupr) Activation by Polydatin and Nicotinamide Corrects Pathological Alterations in Cellular Models of Gfm1 Mutations
7. Mitochondrial Quality Control via Mitochondrial Unfolded Protein Response (mtUPR) in Ageing and Neurodegenerative Diseases
8. Actin Polymerization Defects Induce Mitochondrial Dysfunction in Cellular Models of Nemaline Myopathies
9. Antioxidants Prevent Iron Accumulation and Lipid Peroxidation, but Do Not Correct Autophagy Dysfunction or Mitochondrial Bioenergetics in Cellular Models of BPAN
10. Patient-Derived Cellular Models for Polytarget Precision Medicine in Pantothenate Kinase-Associated Neurodegeneration
11. Patient-Derived Cellular Models for Polytarget Precision Medicine in Pantothenate Kinase-Associated Neurodegeneration
12. Neurodegeneration, Mitochondria, and Antibiotics
13. Actin Polymerization Defects Induce Mitochondrial Dysfunction in Cellular Models of Nemaline Myopathies
14. mtUPR Modulation as a Therapeutic Target for Primary and Secondary Mitochondrial Diseases
15. Mitochondrial Quality Control via Mitochondrial Unfolded Protein Response (mtUPR) in Ageing and Neurodegenerative Diseases
16. Vicious cycle of lipid peroxidation and iron accumulation in neurodegeneration
17. Actin Polymerization Defects Induce Mitochondrial Dysfunction in Cellular Models of Nemaline Myopathies
18. Neurodegeneration, Mitochondria, and Antibiotics
19. mtUPR Modulation as a Therapeutic Target for Primary and Secondary Mitochondrial Diseases
20. Vicious cycle of lipid peroxidation and iron accumulation in neurodegeneration
21. Pantothenate and L-Carnitine Supplementation Improves Pathological Alterations in Cellular Models of KAT6A Syndrome
22. Alpha-Lipoic acid supplementation corrects pathological alterations in cellular models of pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels
23. Pantothenate and L-carnitine Supplementation Corrects Pathological Alterations in Cellular Models of KAT6A Syndrome
24. Additional file 1 of Alpha-lipoic acid supplementation corrects pathological alterations in cellular models of pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels
25. Vicious cycle of lipid peroxidation and iron accumulation in neurodegeneration.
26. Activation of the Mitochondrial Unfolded Protein Response: A New Therapeutic Target?
27. Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels.
28. Activation of the mitochondrial unfolded protein response: A new therapeutic target?
29. Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels
30. Pantothenate and L-Carnitine supplementation improves pathological alterations in cellular models of KAT6A syndrome
31. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases
32. Additional file 1 of UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases
33. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases
34. Additional file 1 of Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels
35. UPRmt activation improves pathological alterations in cellular models of mitochondrial diseases.
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