173 results on '"Chuprun, J. Kurt"'
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2. GRK5 is a regulator of fibroblast activation and cardiac fibrosis
3. A peptide of the amino-terminus of GRK2 induces hypertrophy and yet elicits cardioprotection after pressure overload
4. Nuclear translocation of cardiac G protein-Coupled Receptor kinase 5 downstream of select Gq-activating hypertrophic ligands is a calmodulin-dependent process.
5. Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity
6. GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels
7. SPG7 Is an Essential and Conserved Component of the Mitochondrial Permeability Transition Pore
8. p38α regulates SERCA2a function
9. Abstract 24071: A Peptide Of The Amino-terminus Of Grk2 Induces Hypertrophy And Yet Elicits Cardioprotection After Pressure Overload.
10. Uncovering G Protein-Coupled Receptor Kinase-5 as a Histone Deacetylase Kinase in the Nucleus of Cardiomyocytes
11. Differential Role of G Protein–Coupled Receptor Kinase 5 in Physiological Versus Pathological Cardiac Hypertrophy
12. Darbepoetin alfa, a long-acting erythropoietin analog, offers novel and delayed cardioprotection for the ischemic heart
13. In Vivo Stimulation of α- and β-Adrenoceptors in Mice Differentially Alters Small RNA Content of Circulating Extracellular Vesicles
14. Characterization of βARKct engineered cellular extracellular vesicles and model specific cardioprotection
15. A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure
16. Abstract 475: Beta-adrenoceptor Stimulation Uniquely Regulates Exosome Micro-rna Content Secreted From Cardiomyocytes in vitro and Those Found in vivo Circulating in Blood
17. A Peptide of the Amino-Terminus of GRK2 Induces Hypertrophy and Yet Elicits Cardioprotection after Pressure Overload
18. S100A1 Gene Therapy Preserves in Vivo Cardiac Function after Myocardial Infarction
19. Alteration of myocardial GRK2 produces a global metabolic phenotype
20. Characterization of βARKct engineered cellular extracellular vesicles and model specific cardioprotection.
21. Restricting mitochondrial GRK2 post-ischemia confers cardioprotection by reducing myocyte death and maintaining glucose oxidation
22. Domain‐Specific Roles for GRK2 in Cardiac Hypertrophy and Heart Failure
23. Abstract 278: Domain-specific Roles for GRK2 in Cardiac Hypertrophy and Heart Failure
24. Pro-Death Signaling of GRK2 in Cardiac Myocytes after Ischemic Stress Occurs via ERK-Dependent, Hsp90-Mediated Mitochondrial Targeting
25. Abstract 15544: GRK2 RGS Domain-Specific Interaction With Gαq Inhibits Hypertrophy and Cardiac Dysfunction During Pressure Overload
26. Paroxetine-Mediated GRK2 Inhibition Reverses Cardiac Dysfunction and Remodeling After Myocardial Infarction
27. Abstract 365: Domain-specific Roles for GRK2 in Cardiac Hypertrophy and Heart Failure
28. The Evolving Impact of G Protein-Coupled Receptor Kinases in Cardiac Health and Disease
29. Paroxetine‐mediated GRK2 Inhibition Reverses Cardiac Dysfunction and Remodeling Post‐Myocardial Infarction
30. Mitochondrial Fusion Dynamics in the Heart
31. β-Adrenoceptors in cardiovascular and respiratory diseases
32. GRK5-Mediated Exacerbation of Pathological Cardiac Hypertrophy Involves Facilitation of Nuclear NFAT Activity
33. GRK2 in the Heart: A GPCR Kinase and Beyond
34. Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model.
35. Convergence of G Protein–Coupled Receptor and S-Nitrosylation Signaling Determines the Outcome to Cardiac Ischemic Injury
36. Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model
37. Prodeath Signaling of G Protein–Coupled Receptor Kinase 2 in Cardiac Myocytes After Ischemic Stress Occurs Via Extracellular Signal–Regulated Kinase-Dependent Heat Shock Protein 90–Mediated Mitochondrial Targeting
38. Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity.
39. A peptide of the RGS domain of GRK2 binds and inhibits Gαq to suppress pathological cardiac hypertrophy and dysfunction.
40. Abstract 18646: The Dynamic Relationship Between GRK2 and NOS/NO Critically Determines Cardiac Ischemia/Reperfusion Injury
41. Paroxetine Is a Direct Inhibitor of G Protein-Coupled Receptor Kinase 2 and Increases Myocardial Contractility
42. G i -Biased β 2 AR Signaling Links GRK2 Upregulation to Heart Failure
43. Abstract P339: The Mechanistic Relationship Between GRK2 and eNOS During Cardiac Ischemia/Reperfusion Injury
44. Abstract P195: Nuclear Translocation of GRK5 Following Hypertrophic Stimuli Is a Calmodulin-Dependent Process
45. G Protein–Coupled Receptor Kinase 2 Activity Impairs Cardiac Glucose Uptake and Promotes Insulin Resistance After Myocardial Ischemia
46. A Novel and Efficient Model of Coronary Artery Ligation and Myocardial Infarction in the Mouse
47. Level of G protein–Coupled Receptor Kinase-2 Determines Myocardial Ischemia/Reperfusion Injury via Pro- and Anti-Apoptotic Mechanisms
48. Nuclear Translocation of GRK5 Following Hypertrophic Stimuli Is a Calmodulin-Dependent Process
49. Dynamic Changes in Lymphocyte GRK2 Levels in Cardiac Transplant Patients: A Biomarker for Left Ventricular Function
50. Endothelial S100A1 Modulates Vascular Function via Nitric Oxide
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