132 results on '"Chin, Y. Rebecca"'
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2. TNA‐Mediated Antisense Strategy to Knockdown Akt Genes for Triple‐Negative Breast Cancer Therapy
3. Cellular uptake, tissue penetration, biodistribution, and biosafety of threose nucleic acids: Assessing in vitro and in vivo delivery
4. Light-Activated Nanodiamond-Based Drug Delivery Systems for Spatiotemporal Release of Antisense Oligonucleotides.
5. Distinguishing High-Metastasis-Potential Circulating Tumor Cells through Fluidic Shear Stress in a Bloodstream-Like Microfluidic Circulatory System
6. An integrated and multi-functional droplet-based microfluidic platform for digital DNA amplification
7. Defining super-enhancer landscape in triple-negative breast cancer by multiomic profiling
8. 3D Biomimetic Models to Reconstitute Tumor Microenvironment In Vitro: Spheroids, Organoids, and Tumor‐on‐a‐Chip (Adv. Healthcare Mater. 18/2023)
9. Suppl. Figure 2 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
10. Data from PtdIns(3,4,5)P3-Dependent Activation of the mTORC2 Kinase Complex
11. Data from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
12. Suppl. Figure 1 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
13. Supplementary Methods, Figures S1 - S7 from PtdIns(3,4,5)P3-Dependent Activation of the mTORC2 Kinase Complex
14. Data from PTEN-Deficient Tumors Depend on AKT2 for Maintenance and Survival
15. Suppl. Figure 3 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
16. Supplementary Data from PTEN-Deficient Tumors Depend on AKT2 for Maintenance and Survival
17. Suppl. Table 1 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
18. High Throughput Confined Migration Microfluidic Device for Drug Screening (Small 16/2023)
19. Data from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
20. Data from 3-Phosphoinositide–Dependent Kinase 1 Potentiates Upstream Lesions on the Phosphatidylinositol 3-Kinase Pathway in Breast Carcinoma
21. Data from Targeting Akt3 Signaling in Triple-Negative Breast Cancer
22. Supplementary Figure 1 from Targeting Akt3 Signaling in Triple-Negative Breast Cancer
23. Supplementary Figure 6 from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
24. Supplementary Information, Methods, Table 1, Figures 1-7 from 3-Phosphoinositide–Dependent Kinase 1 Potentiates Upstream Lesions on the Phosphatidylinositol 3-Kinase Pathway in Breast Carcinoma
25. Supplementary Figure 5 from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
26. Supplementary Figure 4 from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
27. Supplementary Figure 1 from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
28. Supplementary Methods and Materials from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
29. Supplementary Figure 3 from RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
30. Akt3 promotes cancer stemness in triple-negative breast cancer through YB1-Snail/Slug signaling axis
31. Distinguishing high-metastasis-potential circulating tumor cells through fluidic shear stress in a bloodstream-like microfluidic circulatory system
32. Upregulation of Receptor Tyrosine Kinase Activity and Stemness as Resistance Mechanisms to Akt Inhibitors in Breast Cancer
33. An Integrated and Multi-Functional Droplet-Based Microfluidic Platform for Digital DNA Amplification
34. Cancer stem cells: advances in biology and clinical translation—a Keystone Symposia report
35. ANLN Enhances Triple-Negative Breast Cancer Stemness Through TWIST1 and BMP2 and Promotes its Spheroid Growth
36. Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer
37. Inhibition of Rb Phosphorylation Leads to mTORC2-Mediated Activation of Akt
38. PtdIns(3,4,5)P3-Dependent Activation of the mTORC2 Kinase Complex
39. Abstract A42: Increased Akt3 expression as a resistance mechanism to targeted therapy
40. Akt2 regulates expression of the actin-bundling protein palladin
41. PTEN-Deficient Tumors Depend on AKT2 for Maintenance and Survival
42. Akt-ing Up on SRPK1: Oncogene or Tumor Suppressor?
43. Targeting Akt3 Signaling in Triple-Negative Breast Cancer
44. RhoB Differentially Controls Akt Function in Tumor Cells and Stromal Endothelial Cells during Breast Tumorigenesis
45. Acetylation-Dependent Regulation of Skp2 Function
46. NFAT promotes carcinoma invasive migration through glypican-6
47. mTOR Drives Its Own Activation via SCFβTrCP-Dependent Degradation of the mTOR Inhibitor DEPTOR
48. Akt isoform-specific signaling in breast cancer
49. The Actin-Bundling Protein Palladin Is an Akt1-Specific Substrate that Regulates Breast Cancer Cell Migration
50. 3-Phosphoinositide–Dependent Kinase 1 Potentiates Upstream Lesions on the Phosphatidylinositol 3-Kinase Pathway in Breast Carcinoma
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