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1. Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in pancreatic cancer

3. MYC Deregulation and PTEN Loss Model Tumor and Stromal Heterogeneity of Aggressive Triple-Negative Breast Cancer

4. Differentiation-state plasticity is a targetable resistance mechanism in basal-like breast cancer.

6. The Human Tumor Atlas Network: Charting Tumor Transitions across Space and Time at Single-Cell Resolution

7. cmIF: A Python Library for Scalable Multiplex Imaging Pipelines

9. Relevance of circulating hybrid cells as a non-invasive biomarker for myriad solid tumors

11. Multiomics analysis of serial PARP inhibitor treated metastatic TNBC inform on rational combination therapies

12. Abstract C087: Window-of-opportunity trial of metastatic pancreatic cancer reveals mechanisms of response to targeting RAS signaling

14. Micelle-Formulated Juglone Effectively Targets Pancreatic Cancer and Remodels the Tumor Microenvironment

15. Deregulating MYC in a model of [HER2.sup.+] breast cancer mimics human intertumoral heterogeneity

17. Chemotherapy response and recurrence-free survival in neoadjuvant breast cancer depends on biomarker profiles: results from the I-SPY 1 TRIAL (CALGB 150007/150012; ACRIN 6657)

19. Pathologic and biologic response to preoperative endocrine therapy in patients with ER-positive ductal carcinoma in situ

20. Longitudinal and multimodal auditing of tumor adaptation to CDK4/6 inhibitors in HR+ metastatic breast cancers

21. A Neural Survival Factor Is a Candidate Oncogene in Breast Cancer

23. Performance of Molecular Inversion Probes (MIP) in Allele Copy Number Determination

25. Analysis of molecular inversion probe performance for allele copy number determination

29. Breast tumor copy number aberration phenotypes and genomic instability

30. In situ quantification of genomic instability in breast cancer progression

32. cmIF: A Python Library for Scalable Multiplex Imaging Pipelines

34. Supplementary Tables 1-7 from Genomic Alterations during the In Situ to Invasive Ductal Breast Carcinoma Transition Shaped by the Immune System

35. Supplementary Methods and Figures 1-5 from Genomic Alterations during the In Situ to Invasive Ductal Breast Carcinoma Transition Shaped by the Immune System

36. Data from Genomic Alterations during the In Situ to Invasive Ductal Breast Carcinoma Transition Shaped by the Immune System

39. Data from A Human Breast Cell Model of Preinvasive to Invasive Transition

40. Data from An Integrated View of Copy Number and Allelic Alterations in the Cancer Genome Using Single Nucleotide Polymorphism Arrays

41. Supplementary Figure 5 from A Human Breast Cell Model of Preinvasive to Invasive Transition

42. Supplementary Figure 4 from A Human Breast Cell Model of Preinvasive to Invasive Transition

43. Supplementary Figure 2 from A Human Breast Cell Model of Preinvasive to Invasive Transition

44. Supplementary Methods and Materials from A Human Breast Cell Model of Preinvasive to Invasive Transition

45. Supplementary Figure 6 from A Human Breast Cell Model of Preinvasive to Invasive Transition

46. Supplementary Figure 7 from A Human Breast Cell Model of Preinvasive to Invasive Transition

47. Supplementary Data URL from An Integrated View of Copy Number and Allelic Alterations in the Cancer Genome Using Single Nucleotide Polymorphism Arrays

48. Supplementary Figure 1 from A Human Breast Cell Model of Preinvasive to Invasive Transition

49. Supplementary Figure 3 from A Human Breast Cell Model of Preinvasive to Invasive Transition

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