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1. Towards Precision Cardiovascular Analysis in Zebrafish: The ZACAF Paradigm

4. Atrial Standstill in the Pediatric Population: A Multi-Institution Collaboration

5. Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

6. The Role of MAPRE2 and Microtubules in Maintaining Normal Ventricular Conduction

7. Author Correction: Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

9. Loss of SPEG Inhibitory Phosphorylation of Ryanodine Receptor Type-2 Promotes Atrial Fibrillation

12. Loss of Protein Phosphatase 1 Regulatory Subunit PPP1R3A Promotes Atrial Fibrillation

15. Competitive Sales Notices.

17. BS-513-02 GENOME-WIDE ASSOCIATION ANALYSES IDENTIFY NOVEL BRUGADA SYNDROME RISK LOCI AND HIGHLIGHT A NEW MECHANISM OF SODIUM CHANNEL REGULATION IN DISEASE SUSCEPTIBILITY

22. Abstract 13171: The Role of Novel Brugada Syndrome Gene MAPRE2 in Maintaining Normal Cardiac Electrophysiology

23. Phosphorylation-Dependent Interactome of Ryanodine Receptor Type 2 in the Heart

25. Targeting the Microtubule EB1-CLASP2 Complex Modulates Na V 1.5 at Intercalated Discs

26. Phosphorylation-Dependent Interactome of Ryanodine Receptor Type 2 in the Heart

29. Loss of SPEG Inhibitory Phosphorylation of RyR2 Promotes Atrial Fibrillation

31. Loss of SPEG Inhibitory Phosphorylation of RyR2 Promotes Atrial Fibrillation

33. Impact of functional studies on exome sequence variant interpretation in early-onset cardiac conduction system diseases

34. Loss of Protein Phosphatase 1 Regulatory Subunit PPP1R3A Promotes Atrial Fibrillation

36. Targeting the Microtubule EB1-CLASP2 Complex Modulates NaV1.5 at Intercalated Discs.

37. Contributors

38. Oxidized CaMKII is Essential for Ventricular Arrhythmia in a Mouse Model of Duchenne Muscular Dystrophy

39. Oxidized CaMKII (Ca 2+ /Calmodulin-Dependent Protein Kinase II) Is Essential for Ventricular Arrhythmia in a Mouse Model of Duchenne Muscular Dystrophy

40. Regulating the regulator: Insights into the cardiac protein phosphatase 1 interactome

41. Chronic exercise: a contributing factor to atrial fibrillation?

42. Oxidized CaMKII (Ca2+/Calmodulin-Dependent Protein Kinase II) Is Essential for Ventricular Arrhythmia in a Mouse Model of Duchenne Muscular Dystrophy.

43. Genetic Deletion of Rnd3/RhoE Results in Mouse Heart Calcium Leakage Through Upregulation of Protein Kinase A Signaling

44. Loss of MicroRNA-106b-25 Cluster Promotes Atrial Fibrillation by Enhancing Ryanodine Receptor Type-2 Expression and Calcium Release

46. Ryanodine Receptor–Mediated Calcium Leak Drives Progressive Development of an Atrial Fibrillation Substrate in a Transgenic Mouse Model

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