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2. Supplementary Figures from Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer–Associated Fibroblasts

5. Data from A Cancer Cell–Intrinsic GOT2–PPARδ Axis Suppresses Antitumor Immunity

6. Supplementary Methods from Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer–Associated Fibroblasts

7. Supplementary Methods from A Stromal Lysolipid–Autotaxin Signaling Axis Promotes Pancreatic Tumor Progression

8. Supplementary Figures from A Stromal Lysolipid–Autotaxin Signaling Axis Promotes Pancreatic Tumor Progression

9. Supplementary Table 1 from A Stromal Lysolipid–Autotaxin Signaling Axis Promotes Pancreatic Tumor Progression

10. Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer–Associated Fibroblasts

11. A cancer cell-intrinsic GOT2-PPARδ axis suppresses antitumor immunity

13. A Stromal Lysolipid–Autotaxin Signaling Axis Promotes Pancreatic Tumor Progression

14. Mesenchymal Lineage Heterogeneity Underlies Non-Redundant Functions of Pancreatic Cancer-Associated Fibroblasts

15. A cancer cell-intrinsic GOT2-PPARδ axis suppresses antitumor immunity

16. Acidic fibroblast growth factor underlies microenvironmental regulation of MYC in pancreatic cancer

17. Abstract PO-095: A cancer cell-intrinsic GOT2-PPARδ axis suppresses antitumor immunity

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