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1. GSmart: An Efficient SPARQL Query Engine Using Sparse Matrix Algebra -- Full Version

5. Accurate and Scalable Graph Convolutional Networks for Recommendation Based on Subgraph Propagation

9. Tumor-intrinsic PRC2 inactivation drives a contextdependent immune-desert microenvironment and is sensitized by immunogenic viruses

12. Aberrant Activation of a Gastrointestinal Transcriptional Circuit in Prostate Cancer Mediates Castration Resistance

16. Data from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

17. supplementary table S1-S8 from FOXF1 Defines the Core-Regulatory Circuitry in Gastrointestinal Stromal Tumor

19. Figure S1-S8 from FOXF1 Defines the Core-Regulatory Circuitry in Gastrointestinal Stromal Tumor

20. Supplementary Table 1 from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

22. Supplementary Table 3 from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

23. Supplementary Figure Legends from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

24. Supplementary Table 2 from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

25. Data from FOXF1 Defines the Core-Regulatory Circuitry in Gastrointestinal Stromal Tumor

26. Supplementary Figures 1 - 6 from Combined Inhibition of MAP Kinase and KIT Signaling Synergistically Destabilizes ETV1 and Suppresses GIST Tumor Growth

28. Efficient Utilization of Multi-Threading Parallelism on Heterogeneous Systems for Sparse Tensor Contraction

29. Tumor-intrinsic PRC2 inactivation drives a context-dependent immune-desert microenvironment and is sensitized by immunogenic viruses

31. Abstract 6164: Tumor-intrinsic PRC2 inactivation drives a context-dependent immune-desert tumor microenvironment

32. Tumor-intrinsic PRC2 inactivation drives a context-dependent immune-desert tumor microenvironment and confers resistance to immunotherapy

35. Non-Myeloablative Chemotherapy as Consolidation Strategy After High-Dose Methotrexate-Based Chemoimmunotherapy in Patients With Primary CNS Lymphoma: A Retrospective Single Center Study in China

38. Fine‐tuning OsCPK18/OsCPK4 activity via genome editing of phosphorylation motif improves rice yield and immunity.

43. Expression of hypoxia‐inducible factor 1α, glucose transporter 1, and hexokinase 2 in primary central nervous system lymphoma and the correlation with the biological behaviors

44. Preliminary Exploration of Ibrutinib Combined with Methotrexate, Ifosfamide, Liposomal Doxorubicin and Methylprednisolone in the Treatment of Relapsed/Refractory Primary CNS Lymphoma

45. Management of secondary central nervous system involvement in systemic aggressive B cell lymphoma using R-MIADD chemotherapy: a single-center experience

47. Parenchymal central nervous system involvement in aggressive B-cell lymphoma: retrospective analysis of clinical and MRI features in a Chinese population

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