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1. T.12.2: THE LEVELS OF AFP, PIVKA-II AND GPC-3 SERUM BIOMARKERS AT DIAGNOSIS CORRELATE WITH HCC CLINICAL-PATHOLOGICAL FEATURES AND WITH OVERALL SURVIVAL

5. Cancer cells and tumor vasculature dynamics analyzed by bio-mathematical modeling in HCC patients with different response to TKIs, TACE and TARE

6. Mathematical modeling of cancer cells and vasculature dynamics with serological and imaging biomarkers suggests synergistic effects of TACE and TKIs in HCC patients

8. OC.04.9 A SINGLE CENTER, LARGE LONG TERM COHORT STUDY OF THE RELATIONS BETWEEN MUTATIONS IN BASIC-CORE-PROMOTER AND PRE-C REGIONS OF HEPATITIS B VIRUS DNA AND CLINICPATHOLOGIC OUTCOME OF HBEAG-NEGATIVE/ANTI-HBEPOSITIVE PHASE

10. AF.84 BIOIMAGING, BIOCHEMICAL AND GENETIC MARKERS TO STRATIFY PATIENTS WITH METABOLIC ASSOCIATED FATTY LIVER DISEASE (MAFLD) IN CLINICAL PRACTICE: A SINGLE CENTER COHORT STUDY

11. AF.96 MODELING HEPATOCELLULAR CARCINOMA CELLS DYNAMICS BY SEROLOGICAL AND IMAGING BIOMARKERS TO EXPLAIN COMPLETE RESPONSE TO SORAFENIB

12. Incremental value of HBcrAg to classify 1582 HBeAg-negative individuals in chronic infection without liver disease or hepatitis

13. Modeling hepatocellular carcinoma cells dynamics by serolo-gical and imaging biomarkers in patients with response to sorafenib and/or regorafenib

15. A single center, large long-term cohort study of the relations between mutations in Basic-Core-Promoter and pre-C regions of Hepatitis B virus DNA and clinic-pathologic outcome of HBeAg-negative/anti-HBe-positive phase

17. T06.01.10 DATA DRIVEN MODEL OF NEOPLASTIC CELLS DYNAMICS BY CIRCULATING AND IMAGING BIOMARKERS OF HEPATOCELLULAR CARCINOMA: A PROMISING TOOL FOR INDIVIDUALIZED THERAPY

20. OC.05.3: MODELING CANCER CELLS AND TUMOR VASCULATURE DYNAMICS BY SERUM BIOMARKERS IN HCC PATIENTS WITH DIFFERENT RESPONSE TO TKIS, TACE AND TARE SUGGESTS A SYNERGISTIC EFFECT OF SYSTEMIC AND ENDOVASCULAR TREATMENTS.

21. P.04.6 MODELING THE TIME-RELATED FLUCTUATIONS OF AFP AND PIVKA-II SERUM LEVELS IN PATIENTS WITH CIRRHOSIS UNDERGOING SURVEILLANCE FOR HEPATOCELLULAR CARCINOMA

23. Cover Image

24. Quantification of serum markers of hepatitis B (HBV) and Delta virus (HDV) infections in patients with chronic HDV infection

25. National quality control and validation of hepatitis C NS3, NS5A and NS5B genotypic resistance testing

26. Clinical significance of time related fluctuations of AFP and PIVKA-II serum levels in single patients with cirrhosis undergoing surveillance for hepatocellular carcinoma

27. Clinical significance of time related fluctuations of AFP and PIVKA-II serum levels in patients with cirrhosis undergoing surveillance for hepatocellular carcinoma

28. In HCV infected patients with cirrhosis Squamous Cell Carcinoma Antigen (SCCA)-IgM levels may contribute to identify the individual risk of HCC development after antiviral therapy

29. Low viremic HBeAg negative HBsAg carriers: Clinical outcome and non invasive diagnostic accuracy for chronic hepatitis B or inactive infection by combinations of HBV markers

30. In hepatitis C infected patients with cirrhosis squamous cell carcinoma antigen (SCCA)-IgM levels may contribute to identify the individual risk of hepatocellular carcinoma development after antiviral therapy

31. Combined GS-4774 and tenofovir therapy can improve HBV-specific T cell responses in patients with chronic active hepatitis B

33. Etiology of chronic liver disease and HCC biomarkers

36. THU-359 - National quality control and validation of hepatitis C NS3, NS5A and NS5B genotypic resistance testing

37. National quality control and validation of hepatitis C NS3, NS5A and NS5B genotypic resistance testing

41. P1202 EARLY PREDICTION OF SUSTAINED VIROLOGIC RESPONSE BY HCV CORE ANTIGEN KINETICS IN CHRONIC HEPATITIS C PATIENTS TREATED WITH PEGINTERFERON PLUS RIBAVIRIN

42. P688 LONG-TERM OUTCOME OF INACTIVE AND LOW VIREMIC HBeAg NEGATIVE ACTIVE CARRIERS: BENIGN ONE DIRECTION TRANSITION TOWARDS SPONTANEOUS HBsAg CLEARANCE

43. Long-term outcome of inactive and low viremic HBeAg negative active carriers: Benign one direction transition towards spontaneous HBsAg clearance

44. Early prediction of sustained virologic response by HCV Core Ag kinetics in chronic hepatitis C patients treated with Peginterferon plus Ribavirin

46. 251 THERAPY WITH T CELL RECEPTOR GENE MODIFIED T CELLS TARGETING HBsAg-PRODUCING HCC METASTASES

48. T-06 Early HCV-Core-Ag kinetics predict sustained virologic response in chronic hepatitis C patients treated with standard of care

50. P.04.1 IL28B POLYMORPHISM IN CHRONIC HBV INFECTION: THE CC ALLELE DOES NOT CORRELATE WITH THE PHASE OF THE INFECTION NOR WITH HBSAG CLEARANCE IN IFN TREATED PATIENTS

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