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1. Diagnostic and prognostic performance of urine ubiquitin carboxy-terminal hydrolase L1 across multiple acute brain injury types – A longitudinal prospective cohort study

2. Common lipidomic signatures across distinct acute brain injuries in patient outcome prediction

4. Fluoxetine and Ketamine Enhance Extinction Memory and Brain Plasticity by Triggering the p75 Neurotrophin Receptor Proteolytic Pathway

5. A small TAT-TrkB peptide prevents BDNF receptor cleavage and restores synaptic physiology in Alzheimer's disease

7. Brain plasticity and neuroinflammatory protein biomarkers with circulating MicroRNAs as predictors of acute brain injury outcome – A prospective cohort study

14. Antidepressant drugs act by directly binding to TRKB neurotrophin receptors

15. Abstract TMP25: Common Molecular Biomarkers in Different Types of Acute Brain Injuries Predicting Outcome

25. Psychedelics promote plasticity by directly binding to BDNF receptor TrkB

26. Abstract TP234: Different Type Of Acute Brain Injuries Share Similar Pattern Of Lipidomic Profile Changes In Human Serum

27. Abstract TP236: Temporal Metabolomic Profiling Exposed Affiliated Metabolites Altered And Associations To Outcome After Different Acute Brain Injuries

28. Abstract WP222: Temporally Differentially Expressed MikroRNAs In Serum Predict Recovery In Different Acute Brain Injuries

30. Slitrk5 Mediates BDNF-Dependent TrkB Receptor Trafficking and Signaling

31. A small TAT-TrkB peptide prevents BDNF receptor cleavage and restores synaptic physiology in Alzheimer’s disease

34. BDNF receptor TrkB as the mediator of the antidepressant drug action

38. Activation of TrkB in Parvalbumin interneurons is required for the promotion of reversal learning in spatial and fear memory by antidepressants

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