38 results on '"Castillo, Sandra D."'
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2. p53 rapidly restructures 3D chromatin organization to trigger a transcriptional response
3. New genetic drivers in hemorrhagic hereditary telangiectasia
4. Angiocrine polyamine production regulates adiposity
5. Behavioural immune landscapes of inflammation
6. PI3K-C2β limits mTORC1 signaling and angiogenic growth
7. p53 rapidly restructures 3D chromatin organization to trigger a transcriptional response
8. Blockade of VEGF-C signaling inhibits lymphatic malformations driven by oncogenic PIK3CA mutation
9. Data from Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
10. Supplementary Figures 1-3 from Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
11. Supplementary Table 2 from Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
12. Supplementary Table 1 from Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
13. Supplementary Figure Legends 1-8, Table Legends 1-3 from Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
14. Immune-interacting lymphatic endothelial subtype at capillary terminals drives lymphatic malformation
15. Behavioural immune landscapes of inflammation
16. PIK3CA mutations in vascular malformations
17. Immune-interacting lymphatic endothelial subtype at capillary terminals drives lymphatic malformation
18. Endothelial cell rearrangements during vascular patterning require PI3-kinase-mediated inhibition of actomyosin contractility
19. The onset of PI3K‐related vascular malformations occurs during angiogenesis and is prevented by the AKT inhibitor miransertib
20. Immunoregulatory subtype of dermal lymphatic endothelial cells at capillary terminals drives lymphatic malformations
21. The onset of PI3K-related vascular malformations occurs during angiogenesis and is prevented by the AKT inhibitor miransertib
22. Low dose AKT inhibitor miransertib cures PI3K-related vascular malformations in preclinical models of human disease
23. Determining the profiles and parameters for gene amplification testing of growth factor receptors in lung cancer
24. Gene amplification of the transcription factor DP1 and CTNND1 in human lung cancer
25. The SRY-HMG box gene, SOX4, is a target of gene amplification at chromosome 6p in lung cancer†
26. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling
27. Genome wide analysis of protein production load in Trichoderma reesei
28. Phosphoinositide 3-kinase: a new kid on the block in vascular anomalies
29. Somatic activating mutations in Pik3ca cause sporadic venous malformations in mice and humans
30. Abstract 3034: Determining the profiles and parameters for gene amplification testing of growth factor receptors in lung cancer.
31. The SOX family of genes in cancer development: biological relevance and opportunities for therapy
32. Novel Transcriptional Targets of the SRY-HMG Box Transcription Factor SOX4 Link Its Expression to the Development of Small Cell Lung Cancer
33. Abstract 4019: Identification of SOX4 transcriptional target genes in human lung cancer
34. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling
35. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling
36. BRCA2 Polymorphic stop codon K3326X and the risk of breast, prostate, and ovarian cancers
37. Somatic activating mutations in Pik3cacause sporadic venous malformations in mice and humans
38. Oncogenic Phosphoinositide 3-Kinase signalling in Venous Malformations
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