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4. Angiocrine polyamine production regulates adiposity

5. Behavioural immune landscapes of inflammation

6. PI3K-C2β limits mTORC1 signaling and angiogenic growth

7. p53 rapidly restructures 3D chromatin organization to trigger a transcriptional response

14. Immune-interacting lymphatic endothelial subtype at capillary terminals drives lymphatic malformation

15. Behavioural immune landscapes of inflammation

17. Immune-interacting lymphatic endothelial subtype at capillary terminals drives lymphatic malformation

18. Endothelial cell rearrangements during vascular patterning require PI3-kinase-mediated inhibition of actomyosin contractility

19. The onset of PI3K‐related vascular malformations occurs during angiogenesis and is prevented by the AKT inhibitor miransertib

20. Immunoregulatory subtype of dermal lymphatic endothelial cells at capillary terminals drives lymphatic malformations

21. The onset of PI3K-related vascular malformations occurs during angiogenesis and is prevented by the AKT inhibitor miransertib

22. Low dose AKT inhibitor miransertib cures PI3K-related vascular malformations in preclinical models of human disease

26. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling

27. Genome wide analysis of protein production load in Trichoderma reesei

29. Somatic activating mutations in Pik3ca cause sporadic venous malformations in mice and humans

30. Abstract 3034: Determining the profiles and parameters for gene amplification testing of growth factor receptors in lung cancer.

34. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling

35. Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling

36. BRCA2 Polymorphic stop codon K3326X and the risk of breast, prostate, and ovarian cancers

37. Somatic activating mutations in Pik3cacause sporadic venous malformations in mice and humans

38. Oncogenic Phosphoinositide 3-Kinase signalling in Venous Malformations

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