164 results on '"Caprini, Elisabetta"'
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2. Mollusk allergy in shrimp-allergic patients: Still a complex diagnosis. An Italian real-life cross-sectional multicenter study
3. Challenging Cutaneous T-Cell Lymphoma: What Animal Models Tell us So Far
4. Preclinical Evidence for Targeting PI3K/mTOR Signaling with Dual-Inhibitors as a Therapeutic Strategy against Cutaneous T-Cell Lymphoma
5. A stereotyped light chain may shape virus-specific B-cell receptors in HCV-dependent lymphoproliferative disorders
6. Blood and skin-derived Sezary cells: differences in proliferation-index, activation of PI3K/AKT/mTORC1 pathway and its prognostic relevance
7. Data from CXCL13 Is Highly Produced by Sézary Cells and Enhances Their Migratory Ability via a Synergistic Mechanism Involving CCL19 and CCL21 Chemokines
8. Supplementary Table 2 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
9. Supplementary Table 1 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
10. Supplementary Figure 1 and Table 1 from CXCL13 Is Highly Produced by Sézary Cells and Enhances Their Migratory Ability via a Synergistic Mechanism Involving CCL19 and CCL21 Chemokines
11. Data from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
12. Supplementary Figure 1 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
13. Supplementary Table 3 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
14. Supplementary Table 4 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
15. Supplementary Figure 2 from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
16. Genome Data Link from Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
17. Human papilomaviru ‐related oropharyngeal squamous cell carcinoma and radiomics: A new era?
18. A spindle cell neoplasm withMYH9 :: EGFRfusion and co‐expression of S100 and CD34 , further expanding the family of kinase fusion positive spindle cell neoplasms
19. Multiplex Proteomic Evaluation in Inborn Errors with Deregulated IgE Response
20. A spindle cell neoplasm with MYH9::EGFR fusion and co‐expression of S100 and CD34, further expanding the family of kinase fusion positive spindle cell neoplasms.
21. Loss of the candidate tumor suppressor ZEB1 (TCF8, ZFHX1A) in Sézary syndrome
22. Comprehensive analysis of PTEN status in Sézary syndrome
23. IgE Sensitization Profile in Patients with Netherton Syndrome
24. A qualitative and quantitative comparison of IgE antibody profiles with two multiplex platforms for component‐resolved diagnostics in allergic patients
25. Loss of RALT/MIG-6 expression in ERBB2-amplified breast carcinomas enhances ErbB-2 oncogenic potency and favors resistance to Herceptin
26. Persistently biased T-cell receptor repertoires in HIV-1-infected combination antiretroviral therapy-treated patients despite sustained suppression of viral replication
27. Skin homing of Sézary cells involves SDF-1-CXCR4 signaling and down-regulation of CD26/dipeptidylpeptidase IV
28. Low frequency of alterations of the α (PPP2R1A) and β (PPP2R1B) isoforms of the subunit A of the serine-threonine phosphatase 2A in human neoplasms
29. Impaired T- and B-cell development in Tcl1-deficient mice
30. Abstract 3912: The PI3K/mTOR dual inhibitor PF-04691502 shows antitumor activity in Sezary cells and in a xenograft mouse model
31. Loss of β-arrestin-2 gene and possible functional consequences on Sezary Syndrome
32. Skewed T-cell receptor repertoire, decreased thymic output, and predominance of terminally differentiated T cells in ataxia telangiectasia
33. Blood and skin-derived Sezary cells: differences in proliferation-index, activation of PI3K/AKT/mTORC1 pathway and its prognostic relevance
34. T Cell Leukemia/Lymphoma 1A is essential for mouse epidermal keratinocytes proliferation promoted by insulin-like growth factor 1
35. Abstract 761: The role of PI3 kinase pathway in the the skin of Sezary syndrome
36. Abstract 936: Skin microenvironment enhances proliferation index and activates mTORC 1 signaling in sezary syndrome
37. Targeting the PI3K/AKT/mTOR pathway overcomes the stimulating effect of dabrafenib on the invasive behavior of melanoma cells with acquired resistance to the BRAF inhibitor
38. Abstract 3974: A preclinical study for miR181b as therapeutic in Eu-TCL1FL-tg mouse model for CLL
39. miR-181b as a therapeutic agent for chronic lymphocytic leukemia in the Eμ-TCL1 mouse model
40. Ralt as a potential tumor suppressor in cancer cells
41. BCR/ABL1-positive acute lymphoblastic leukemia relapsing asBCR/ABL1-negative acute lymphoblastic leukemia
42. Anayisis of Chromosomal Alterations by Array-Based Comparative Genomic Hybridization in 25 Patients with Sézary Syndrome.
43. Gistic Evaluation in Sezary Syndrome
44. Abstract 4178: Tcl1 enhances keratinocytes’ survival/proliferation by promoting erk and jnk/sap phosphorylation at the expense of p38 and by controlling c-fos expression through miR-29b and miR-181a-1
45. Blastic Plasmacytoid Dendritic Cell Neoplasm: Clinical, Immunohistochemical and Molecular Evaluation of 23 Cases with Primary Cutaneous Involvement,
46. Abstract 150: Regulation of TGFB receptor by miR21 in Sezary syndrome
47. Abstract 1736: Skewed usage of TCRVbeta repertoire and predictive role of CD60 and CD49d expression in the survival rate of patients with Sézàry Syndrome
48. Combined High Resolution Genomic and Expression Profiles Microarray Analysis in Sezary Syndrome.
49. Identification of Key Regions and Genes Important in the Pathogenesis of Sézary Syndrome by Combining Genomic and Expression Microarrays
50. CXCL13 Is Highly Produced by Sézary Cells and Enhances Their Migratory Ability via a Synergistic Mechanism Involving CCL19 and CCL21 Chemokines
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