230 results on '"Cannito, Stefania"'
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2. SAT-219 Discovering 1-piperidine propionic acid inhibiting effect on protease activated receptor 2 and serpinb3 as a new therapeutic strategy for metabolic dysfunction-associated steatohepatitis
3. FRI-479-YI The role of OSM/OSMRβ axis in shaping the immunemicroenvironment favouring MASLD-related HCC immune evasion
4. Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell Proliferation
5. Histidine-rich glycoprotein in metabolic dysfunction-associated steatohepatitis-related disease progression and liver carcinogenesis.
6. The polymorphic variant of SerpinB3 (SerpinB3‐PD) is associated with faster cirrhosis decompensation
7. Effects of the rare elements lanthanum and cerium on the growth of colorectal and hepatic cancer cell lines
8. Hepatocellular Carcinoma: Latest Research in Pathogenesis, Detection and Treatment
9. Chronic Liver Disease: Latest Research in Pathogenesis, Detection and Treatment
10. Inhibition of CCAT/enhancer-binding protein beta-serpinB3 axis by 1-piperidin propionic acid: a new targeted therapy for non-alcoholic steatohepatitis
11. Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC
12. The polymorphic variant of SerpinB3 (SerpinB3‐PD) is associated with faster cirrhosis decompensation.
13. SerpinB3 Upregulates Low-Density Lipoprotein Receptor-Related Protein (LRP) Family Members, Leading to Wnt Signaling Activation and Increased Cell Survival and Invasiveness
14. The CCAAT/Enhancer-Binding Protein Beta - SerpinB3 Axis Inhibition as a Novel Strategy for Non-Alcoholic Steatohepatitis Treatment
15. Inflammatory processes involved in NASH-related hepatocellular carcinoma
16. Serpinb3 is overexpressed in the liver in presence of iron overload
17. Oncostatin M: From Intracellular Signaling to Therapeutic Targets in Liver Cancer
18. SerpinB3 as a Pro-Inflammatory Mediator in the Progression of Experimental Non-Alcoholic Fatty Liver Disease
19. G protein–coupled receptor 21 in macrophages: An in vitro study
20. Hepatic Myofibroblasts: A Heterogeneous and Redox-Modulated Cell Population in Liver Fibrogenesis
21. Therapeutic pro-fibrogenic signaling pathways in fibroblasts
22. Metabolic Reprogramming of HCC: A New Microenvironment for Immune Responses.
23. Oncostatin M is overexpressed in NASH‐related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis
24. Oncostatin M is overexpressed in NASH-related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis
25. Hyaluronated and PEGylated Liposomes as a Potential Drug-Delivery Strategy to Specifically Target Liver Cancer and Inflammatory Cells
26. GPR21 Inhibition Increases Glucose-Uptake in HepG2 Cells
27. The mitogen-activated protein kinase ERK5 regulates the development and growth of hepatocellular carcinoma
28. Hypoxia, Hypoxia-Inducible Factors and Liver Fibrosis
29. WED-449 - Inhibition of CCAT/enhancer-binding protein beta-serpinB3 axis by 1-piperidin propionic acid: a new targeted therapy for non-alcoholic steatohepatitis
30. SAT-211 - Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC
31. Hepatic Myofibroblasts in Liver Fibrogenesis
32. Celecoxib inactivates epithelial–mesenchymal transition stimulated by hypoxia and/or epidermal growth factor in colon cancer cells
33. The biphasic nature of hypoxia-induced directional migration of activated human hepatic stellate cells
34. SERPINB3 inhibition as a novel target therapy for non-alcoholic steatohepatitis
35. Liver fibrogenesis: un update on established and emerging basic concepts
36. Oncostatin M, A Profibrogenic Mediator Overexpressed in Non-Alcoholic Fatty Liver Disease, Stimulates Migration of Hepatic Myofibroblasts
37. SerpinB3 Differently Up-Regulates Hypoxia Inducible Factors -1α and -2α in Hepatocellular Carcinoma: Mechanisms Revealing Novel Potential Therapeutic Targets
38. SERPINB3 induces epithelial–mesenchymal transition
39. Redox mechanisms switch on hypoxia-dependent epithelial–mesenchymal transition in cancer cells
40. NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2 alpha
41. ERK Pathway in Activated, Myofibroblast-Like, Hepatic Stellate Cells: A Critical Signaling Crossroad Sustaining Liver Fibrosis
42. THU-464-NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2 alpha
43. Fibroinflammatory Liver Injuries as Preneoplastic Condition in Cholangiopathies
44. Oncostatin M, A Profibrogenic Mediator Overexpressed in Non-Alcoholic Fatty Liver Disease, Stimulates Migration of Hepatic Myofibroblasts.
45. Hypoxia‐inducible factor 2α drives nonalcoholic fatty liver progression by triggering hepatocyte release of histidine‐rich glycoprotein
46. Hyaluronated mesoporous silica nanoparticles for active targeting: influence of conjugation method and hyaluronic acid molecular weight on the nanovector properties
47. Serpinb3 is overexpressed in the liver in presence of iron overload
48. SerpinB3 Promotes Pro-fibrogenic Responses in Activated Hepatic Stellate Cells
49. Microvesicles released from fat-laden cells promote activation of hepatocellular NLRP3 inflammasome: A pro-inflammatory link between lipotoxicity and non-alcoholic steatohepatitis
50. SerpinB3 and Yap Interplay Increases Myc Oncogenic Activity
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