779 results on '"Canalis, Ernesto"'
Search Results
2. A NOTCH2 pathogenic variant and HES1 regulate osteoclastogenesis in induced pluripotent stem cells
3. TNFα has differential effects on the transcriptome profile of selected populations in murine cartilage
4. NOTCH2 promotes osteoclast maturation and metabolism and modulates the transcriptome profile during osteoclastogenesis
5. Antisense oligonucleotides targeting a NOTCH3 mutation in male mice ameliorate the cortical osteopenia of lateral meningocele syndrome
6. NOTCH2 sensitizes the chondrocyte to the inflammatory response of tumor necrosis factor α
7. Induced pluripotent stem cell technology in bone biology
8. A NOTCH3 pathogenic variant influences osteogenesis and can be targeted by antisense oligonucleotides in induced pluripotent stem cells.
9. Skeletal disorders associated with the growth hormone–insulin-like growth factor 1 axis
10. Hairy and enhancer of split 1 is a primary effector of NOTCH2 signaling and induces osteoclast differentiation and function
11. Activation of Notch3 in osteoblasts/osteocytes causes compartment-specific changes in bone remodeling
12. Notch and the regulation of osteoclast differentiation and function
13. Antisense oligonucleotides targeting Notch2 ameliorate the osteopenic phenotype in a mouse model of Hajdu-Cheney syndrome
14. Glucocorticoid-Induced Osteoporosis
15. The Hajdu Cheney mutation sensitizes mice to the osteolytic actions of tumor necrosis factor α
16. Skeletal growth factors
17. List of contributors
18. The lateral meningocele syndrome mutation causes marked osteopenia in mice
19. Induction of the Hajdu-Cheney Syndrome Mutation in CD19 B Cells in Mice Alters B-Cell Allocation but Not Skeletal Homeostasis
20. Clinical and experimental aspects of notch receptor signaling: Hajdu-Cheney syndrome and related disorders
21. The Hajdu Cheney Mutation Is a Determinant of B-Cell Allocation of the Splenic Marginal Zone
22. Sustained Notch2 signaling in osteoblasts, but not in osteoclasts, is linked to osteopenia in a mouse model of Hajdu-Cheney syndrome
23. List of Contributors
24. Notch Signaling in Skeletal Diseases
25. Notch and its ligands
26. Hajdu Cheney Mouse Mutants Exhibit Osteopenia, Increased Osteoclastogenesis, and Bone Resorption
27. Connective Tissue Growth Factor is a Target of Notch Signaling in Cells of the Osteoblastic Lineage
28. Notch1 and Notch2 expression in osteoblast precursors regulates femoral microarchitecture
29. Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway
30. Hajdu-Cheney Syndrome, a Disease Associated with NOTCH2 Mutations
31. Notch Signaling in Osteocytes Differentially Regulates Cancellous and Cortical Bone Remodeling
32. Hairy and Enhancer of Split-related with YRPW Motif (HEY)2 Regulates Bone Remodeling in Mice
33. Nuclear Factor of Activated T-cells (NFAT)c2 Inhibits Notch Receptor Signaling in Osteoblasts
34. A soluble bone morphogenetic protein type IA receptor increases bone mass and bone strength
35. A Soluble Activin Type IIA Receptor Induces Bone Formation and Improves Skeletal Integrity
36. The Heparin-binding Domain of IGFBP-2 Has Insulin-like Growth Factor Binding-independent Biologic Activity in the Growing Skeleton
37. Reciprocal Regulation of Notch and Nuclear Factor of Activated T-cells (NFAT) c1 Transactivation in Osteoblasts
38. HES1 (Hairy and Enhancer of Split 1) Is a Determinant of Bone Mass
39. Use of antisense oligonucleotides to target Notch3 in skeletal cells
40. Notch Signaling and the Skeleton
41. New Treatment Modalities in Osteoporosis
42. Col3.6-HSD2 transgenic mice: A glucocorticoid loss-of-function model spanning early and late osteoblast differentiation
43. Glucocorticoid-induced osteoporosis in rheumatic diseases
44. Drug-induced Osteoporosis: Mechanisms and Clinical Implications
45. Murine Bone Exhibits Temporal and Dosage Response to Metabolic Acidosis
46. Igfbp2 Deletion in Ovariectomized Mice Enhances Energy Expenditure but Accelerates Bone Loss
47. Transforming and Nontransforming Growth Factors are Present in Medium Conditioned by Fetal Rat Calvariae
48. Parathyroid Hormone Modulates Transforming Growth Factor β Activity and Binding in Osteoblast-Enriched Cell Cultures from Fetal Rat Parietal Bone
49. Chapter 11 - Skeletal growth factors
50. Notch Regulation of Bone Development and Remodeling and Related Skeletal Disorders
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.