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3. Index

Author

Kealey, Gregory S., Clement, Dominique, and Campbell, Lara A.

Published
2012

4. Contributors

Author

Kealey, Gregory S., Clement, Dominique, and Campbell, Lara A.

Published
2012

5. Notes

Author

Kealey, Gregory S., Clement, Dominique, and Campbell, Lara A.

Published
2012

24. Preface

Author

Kealey, Gregory S., Clement, Dominique, and Campbell, Lara A.

Published
2012

27. References

Author

Campbell, Lara A.

Published
2009

29. Contents

Author

Campbell, Lara A.

Published
2009

31. Index

Author

Campbell, Lara A.

Published
2009

32. Notes

Author

Campbell, Lara A.

Published
2009

42. Donor-Derived Malignancy and Transplantation Morbidity: Risks of Patient and Donor Genetics in Allogeneic Hematopoietic Stem Cell Transplantation

Author

Williams, Lacey S., Williams, Kirsten M., Gillis, Nancy, Bolton, Kelly, Damm, Frederik, Deuitch, Natalie T., Farhadfar, Nosha, Gergis, Usama, Keel, Siobán B., Michelis, Fotios V., Panch, Sandhya R., Porter, Christopher C., Sucheston-Campbell, Lara, Tamari, Roni, Stefanski, Heather E., Godley, Lucy A., and Lai, Catherine

Published
2024
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43. Cross-ancestry GWAS meta-analysis identifies six breast cancer loci in African and European ancestry women.

Author

Adedokun, Babatunde, Du, Zhaohui, Gao, Guimin, Ahearn, Thomas U, Lunetta, Kathryn L, Zirpoli, Gary, Figueroa, Jonine, John, Esther M, Bernstein, Leslie, Zheng, Wei, Hu, Jennifer J, Ziegler, Regina G, Nyante, Sarah, Bandera, Elisa V, Ingles, Sue A, Press, Michael F, Deming-Halverson, Sandra L, Rodriguez-Gil, Jorge L, Yao, Song, Ogundiran, Temidayo O, Ojengbede, Oladosu, Blot, William, Troester, Melissa A, Nathanson, Katherine L, Hennis, Anselm, Nemesure, Barbara, Ambs, Stefan, Fiorica, Peter N, Sucheston-Campbell, Lara E, Bensen, Jeannette T, Kushi, Lawrence H, Torres-Mejia, Gabriela, Hu, Donglei, Fejerman, Laura, Bolla, Manjeet K, Dennis, Joe, Dunning, Alison M, Easton, Douglas F, Michailidou, Kyriaki, Pharoah, Paul DP, Wang, Qin, Sandler, Dale P, Taylor, Jack A, O'Brien, Katie M, Kitahara, Cari M, Falusi, Adeyinka G, Babalola, Chinedum, Yarney, Joel, Awuah, Baffour, Addai-Wiafe, Beatrice, GBHS Study Team, Chanock, Stephen J, Olshan, Andrew F, Ambrosone, Christine B, Conti, David V, Ziv, Elad, Olopade, Olufunmilayo I, Garcia-Closas, Montserrat, Palmer, Julie R, Haiman, Christopher A, and Huo, Dezheng

Subjects
GBHS Study Team, Humans, Breast Neoplasms, Genetic Predisposition to Disease, Polymorphism, Single Nucleotide, Quantitative Trait Loci, Introns, African Continental Ancestry Group, European Continental Ancestry Group, Female, Genome-Wide Association Study
Abstract

Our study describes breast cancer risk loci using a cross-ancestry GWAS approach. We first identify variants that are associated with breast cancer at P

Published
2021

44. Shared graft-versus-leukemia minor histocompatibility antigens in DISCOVeRY-BMT

Author

Olsen, Kelly S., Jadi, Othmane, Dexheimer, Sarah, Bortone, Dante S., Vensko, Steven P., II, Bennett, Sarah, Tang, Hancong, Diiorio, Marisa, Saran, Tanvi, Dingfelder, David, Zhu, Qianqian, Wang, Yiwen, Haiman, Christopher A., Pooler, Loreall, Sheng, Xin, Webb, Amy, Pasquini, Marcelo C., McCarthy, Philip L., Spellman, Stephen R., Weimer, Eric, Hahn, Theresa, Sucheston-Campbell, Lara, Armistead, Paul M., and Vincent, Benjamin G.

Published
2023
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46. Author Correction: Genome-wide association study identifies susceptibility loci for acute myeloid leukemia

Author

Lin, Wei-Yu, Fordham, Sarah E., Hungate, Eric, Sunter, Nicola J., Elstob, Claire, Xu, Yaobo, Park, Catherine, Quante, Anne, Strauch, Konstantin, Gieger, Christian, Skol, Andrew, Rahman, Thahira, Sucheston-Campbell, Lara, Wang, Junke, Hahn, Theresa, Clay-Gilmour, Alyssa I., Jones, Gail L., Marr, Helen J., Jackson, Graham H., Menne, Tobias, Collin, Mathew, Ivey, Adam, Hills, Robert K., Burnett, Alan K., Russell, Nigel H., Fitzgibbon, Jude, Larson, Richard A., Le Beau, Michelle M., Stock, Wendy, Heidenreich, Olaf, Alharbi, Abrar, Allsup, David J., Houlston, Richard S., Norden, Jean, Dickinson, Anne M., Douglas, Elisabeth, Lendrem, Clare, Daly, Ann K., Palm, Louise, Piechocki, Kim, Jeffries, Sally, Bornhäuser, Martin, Röllig, Christoph, Altmann, Heidi, Ruhnke, Leo, Kunadt, Desiree, Wagenführ, Lisa, Cordell, Heather J., Darlay, Rebecca, Andersen, Mette K., Fontana, Maria C., Martinelli, Giovanni, Marconi, Giovanni, Sanz, Miguel A., Cervera, José, Gómez-Seguí, Inés, Cluzeau, Thomas, Moreilhon, Chimène, Raynaud, Sophie, Sill, Heinz, Voso, Maria Teresa, Lo-Coco, Francesco, Dombret, Hervé, Cheok, Meyling, Preudhomme, Claude, Gale, Rosemary E., Linch, David, Gaal-Wesinger, Julia, Masszi, Andras, Nowak, Daniel, Hofmann, Wolf-Karsten, Gilkes, Amanda, Porkka, Kimmo, Milosevic Feenstra, Jelena D., Kralovics, Robert, Grimwade, David, Meggendorfer, Manja, Haferlach, Torsten, Krizsán, Szilvia, Bödör, Csaba, Stölzel, Friedrich, Onel, Kenan, and Allan, James M.

Published
2022
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47. Physical activity modifies genetic susceptibility to obesity in postmenopausal women

Author

Ochs-Balcom, Heather M, Preus, Leah, Nie, Jing, Wactawski-Wende, Jean, Agyemang, Linda, Neuhouser, Marian L, Tinker, Lesley, Zheng, Cheng, Kazlauskaite, Rasa, Qi, Lihong, and Sucheston-Campbell, Lara E

Subjects
Epidemiology, Biomedical and Clinical Sciences, Public Health, Health Sciences, Genetics, Prevention, Human Genome, Obesity, Nutrition, Aetiology, 2.1 Biological and endogenous factors, Metabolic and endocrine, Cancer, Oral and gastrointestinal, Age Factors, Aged, Aged, 80 and over, Analysis of Variance, Body Mass Index, Exercise, Female, Follow-Up Studies, Genetic Predisposition to Disease, Genetic Testing, Genome-Wide Association Study, Genotype, Healthy Aging, Humans, Linear Models, Logistic Models, Middle Aged, Motor Activity, Polymorphism, Single Nucleotide, Postmenopause, Self Report, White People, Body mass index, Genetic susceptibility, Physical activity, Medical and Health Sciences, Obstetrics & Reproductive Medicine, Biomedical and clinical sciences, Health sciences, Psychology
Abstract

ObjectiveWe conducted a gene-environment interaction study to evaluate whether the association of body mass index (BMI) associated meta genome-wide association study single-nucleotide polymorphisms (SNPs) (as a genetic risk score) and BMI is modified by physical activity and age.MethodsIn 8,206 women of European ancestry from the Women's Health Initiative (WHI), we used linear regression to examine main effects of the 95 SNP BMI genetic risk score (GRS) and physical activity on BMI, and evaluated whether genetic associations are modified by physical activity (two-way interaction) and age (three-way interaction).ResultsWe found evidence for modification of the BMI GRS-BMI association according to both physical activity and age. We observed a significant two-way interaction of BMI GRS × physical activity in the crude model (P interaction = 0.05), where a smaller effect of the BMI GRS on BMI with increasing physical activity. The beta coefficient was 0.05 (standard error [SE] = 0.02, P = 0.01) for the high-activity group compared with beta = 0.13 (SE = 0.02, P = 4.8 × 10) for the sedentary group. The three-way interaction was statistically significant (adjusted P interaction = 0.01). Notably, in the 70+ age group, the BMI GRS-BMI association was attenuated and no longer significant in the high-activity group; the beta coefficient for the 70+ high-activity group was relatively small and nonsignificant (beta = 0.02, SE = 0.03, P = 0.58) compared with 70+ sedentary group (beta = 0.17, SE = 0.03, P = 2.5 × 10).ConclusionOur study suggests that physical activity attenuates the influence of genetic predisposition to obesity, and this effect is more profound in the oldest age group.

Published
2018

48. Genome-wide meta-analyses identifies novel taxane-induced peripheral neuropathy-associated loci

Author

Sucheston-Campbell, Lara E, Clay-Gilmour, Alyssa I, Barlow, William E, Budd, G Thomas, Stram, Daniel O, Haiman, Christopher A, Sheng, Xin, Yan, Li, Zirpoli, Gary, Yao, Song, Jiang, Chen, Owzar, Kouros, Hershman, Dawn, Albain, Kathy S, Hayes, Daniel F, Moore, Halle C, Hobday, Timothy J, Stewart, James A, Rizvi, Abbas, Isaacs, Claudine, Salim, Muhammad, Gralow, Jule R, Hortobagyi, Gabriel N, Livingston, Robert B, Kroetz, Deanna L, and Ambrosone, Christine B

Subjects
Biological Sciences, Biomedical and Clinical Sciences, Genetics, Oncology and Carcinogenesis, Peripheral Neuropathy, Cancer, Neurodegenerative, Human Genome, Neurosciences, Clinical Research, Breast Cancer, 2.1 Biological and endogenous factors, Aetiology, Black or African American, Breast Neoplasms, Bridged-Ring Compounds, Female, Genetic Predisposition to Disease, Genome-Wide Association Study, Genomics, Genotype, Humans, Peripheral Nervous System Diseases, Polymorphism, Single Nucleotide, Taxoids, White People, breast cancer, genome-wide association study, neuropathy, taxane, Pharmacology and Pharmaceutical Sciences, Pharmacology & Pharmacy, Pharmacology and pharmaceutical sciences
Abstract

ObjectiveTaxane containing chemotherapy extends survival for breast cancer patients. However, taxane-induced peripheral neuropathy (TIPN) cannot be predicted, prevented or effectively treated. Using genome-wide analyses, we sought to identify common risk variants for TIPN.Patients and methodsWomen with high-risk breast cancer enrolled in SWOG 0221 were genotyped using the Illumina 1M chip. Genome-wide analyses were performed in relation to ≥grade 3 Common Terminology Criteria for Adverse Events (CTCAE) neuropathy in European and African Americans. Data were meta-analyzed with GW associations of CTCAE ≥grade 3 versus ResultsThe percentage of ≥grade 3 TIPN in 1269 European Americans and 139 African Americans in S0221, was 11.6 and 22.3%, respectively. CALGB 40101 ≥grade 3 TOPN was 7.2%. The most significant association with ≥grade 3 TIPN was the G allele of rs1858826 in GNGT1 (Pmeta=1.1×10), which showed a decrease in risk of ≥grade 3 TIPN (odds ratio=0.29, 95% confidence interval: 0.18-0.46).ConclusionThe genetic variants associated with ≥grade 3 TIPN are hypothesized to have biochemical functions and reside in and near genes involved in diabetes and diabetic neuropathy. This finding is consistent with results from CALGB 40101 pathway analyses. Larger homogeneous trials with similar dosing and criteria for defining neuropathy are needed to properly assess the relationship of genomics with the neuropathy spectrum.

Published
2018

50. Novel genetic variants associated with mortality after unrelated donor allogeneic hematopoietic cell transplantation

Author

Hahn, Theresa, Wang, Junke, Preus, Leah M., Karaesmen, Ezgi, Rizvi, Abbas, Clay-Gilmour, Alyssa I., Zhu, Qianqian, Wang, Yiwen, Yan, Li, Liu, Song, Stram, Daniel O., Pooler, Loreall, Sheng, Xin, Haiman, Christopher A., Berg, David Van Den, Webb, Amy, Brock, Guy, Spellman, Stephen R., Onel, Kenan, McCarthy, Philip L., Pasquini, Marcelo C., and Sucheston-Campbell, Lara E.

Published
2021
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