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1. Nlrp2 deletion ameliorates kidney damage in a mouse model of cystinosis

4. Mechanisms of mesothelial cell response to viral infections: HDAC1-3 inhibition blocks poly(I:C)-induced type I interferon response and modulates the mesenchymal/inflammatory phenotype

5. Inflammasome Activation by Cystine Crystals: Implications for the Pathogenesis of Cystinosis

6. Persistently active interferon‐γ pathway and expansion of T‐bet + B cells in a subset of patients with childhood‐onset systemic lupus erythematosus

7. Microbiota transplant to control inflammation in a patient with NLRC4 gain-of-function–induced disease

10. Persistently active interferon‐γ pathway and expansion of T‐bet+ B cells in a subset of patients with childhood‐onset systemic lupus erythematosus.

12. Pro Nerve Growth Factor and Its Receptor p75NTR Activate Inflammatory Responses in Synovial Fibroblasts: A Novel Targetable Mechanism in Arthritis

13. Identification of a Novel Mutation in TNFAIP3 in a Family With Poly-Autoimmunity

15. Early Treatment and IL1RN Single‐Nucleotide Polymorphisms Affect Response to Anakinra in Systemic Juvenile Idiopathic Arthritis

16. Autoantibody-mediated impairment of DNASE1L3 activity in sporadic systemic lupus erythematosus

17. Monocytes From Patients With Macrophage Activation Syndrome and Secondary Hemophagocytic Lymphohistiocytosis Are Hyperresponsive to Interferon Gamma

19. IFNAR2 Deficiency Causing Dysregulation of NK Cell Functions and Presenting With Hemophagocytic Lymphohistiocytosis

20. The interferon-gamma pathway is selectively up-regulated in the liver of patients with secondary hemophagocytic lymphohistiocytosis

23. Type I Interferon Score and Interferon Induced Mediators CXCL10 and Neopterin Are Correlated with Disease Activity in Juvenile Dermatomyositis

24. THU0513 WHOLE BLOOD CELLS FROM PATIENTS WITH SYSTEMIC JUVENILE IDIOPATHIC ARTHRITIS (SJIA) IN CLINICAL INACTIVE DISEASE DISPLAY A DYSREGULATED RESPONSE TO TLR-4 STIMULATION

25. FRI0539 WNT6 MUTATION CAUSES AN EARLY ONSET GRANULOMATOSUS INTESTINAL DISEASE WITH RECURRENT HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS (HLH)

26. AB0185 INTERFERON-Γ AMPLIFIES IMMUNE RESPONSE MEDIATED BY TYPE I INTERFERONS IN PAEDIATRIC SYSTEMIC LUPUS ERYTHEMATOSUS AND CORRELATES WITH DISEASE ACTIVITY

27. OP0255 MICROBIOTA TRANSPLANT TO CONTROL INFLAMMATION IN A NLRC4-RELATED DISEASE PATIENT WITH RECURRENT HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS (HLH)

31. Expansion of CD4dimCD8+T cells characterizes macrophage activation syndrome and other secondary HLH

32. Neutralization of IFN-γ reverts clinical and laboratory features in a mouse model of macrophage activation syndrome

33. Switched Memory B Cells Are Increased in Oligoarticular and Polyarticular Juvenile Idiopathic Arthritis and Their Change Over Time Is Related to Response to Tumor Necrosis Factor Inhibitors

35. Elevated circulating levels of interferon-γ and interferon-γ-induced chemokines characterise patients with macrophage activation syndrome complicating systemic juvenile idiopathic arthritis

36. A patient with stimulator of interferon genes–associated vasculopathy with onset in infancy without skin vasculopathy.

40. Interferon-gamma (IFNy) in macrophage activation syndrome (MAS) associated with systemic juvenile idiopathic arthritis (sJIA). High levels in patients and a role in a murine mas model

43. Proceedings of the 23rd Paediatric Rheumatology European Society Congress: part two: Genoa, Italy. 28 September – 01 October 2016

44. Autologous CD19‐Targeting CAR T Cells in a Patient With Refractory Juvenile Dermatomyositis.

48. The Anti-Inflammatory Action of Nerve Growth Factor Is Mediated by Trka Signalling: Modification in Trka Expression in Juvenile Idiopathic Arthritis Mononuclear Cells Create An Unbalance Between Inflammatory and Anti-Inflammatory Pathways?

49. Correlation of Type I Interferon Score and CXCL10 (C-X-C Motif Chemokine Ligand 10) with Cutaneous and Muscular Disease Activity in Juvenile Dermatomyositis Patients

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