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A number of behavior therapists have proposed that depression results when a discriminative stimulus or reinforcer for behavior is removed. It is proposed here that the depressed person's general loss of interest in his environment suggests that there is a loss of reinforcer effectiveness. The manner in which environmental events, including the loss of a reinforcer, may result in this general loss of reinforcer effectiveness is discussed. The possible evolutionary significance of depression is also briefly discussed., (Copyright © 2016 Elsevier Ltd. All rights reserved.)

Two dimensional views of adult psychopathology are discussed: (a) the phenomenological view according to which symptoms of psychopathology can occur in normal people in less intense, persistent and debilitating, but not qualitatively different forms; and (b) the vulnerability view according to which the degree to which a person possesses the apparent normal counterparts of the symptoms of a psychopathological disorder is an index of that person's vulnerability for the disorder. Examples of research using questionnaires designed by two prominent advocates of these dimensional views, namely, H. J. Eysenck and L. J. Chapman, are reviewed. It is concluded that the research strategies to date do not provide adequate tests of the phenomenological view and provide only weak support for the vulnerability view. Alternative, more direct, research strategies are proposed.

Restless sleep was compared across 110 out-patients with rheumatoid arthritis (RA), 101 with end-stage renal disease (ESRD), 94 with multiple sclerosis (MS), and an unselected control group of 176 individuals attending their family practitioners (FP). It was also investigated in the three chronic illness groups as a contributor to illness intrusiveness--the extent to which one's illness and/or its treatment interfere with continued involvements in valued activities and interests--a determinant of depression and emotional distress in chronic conditions. Reported frequencies of restless sleep were highest in RA, lower in ESRD, and lowest in MS; FP patients reported frequencies that were similar, overall, to those observed in MS. These differences were evident among nondepressed, (chi 2 9, N = 309, p < 0.0001), but not depressed, individuals. The occurrence of restless sleep correlated significantly with increased illness intrusiveness, r (279) = 0.31, p < 0.001, in RA, ESRD, and MS. Results supported the hypothesis that the relation between restless sleep and emotional distress is mediated by illness intrusiveness. Treatment of restless sleep may offer the added benefit of diminishing illness intrusiveness and may, thereby, enhance quality of life in chronic physical illness.

Research on symptoms may often be more fruitful than research on syndromes because: (a) the validities of psychiatric diagnoses remain questionable; (b) interview assessments of a large number of symptoms are inadequate; (c) the diagnostic approach results in problems of misclassification and confounding; (d) the dimensional/categorical issue may be more readily resolved with research using a symptom approach; (e) useful animal models of symptoms are more likely to be developed than useful animal models of syndromes; and (f) symptoms may be better phenotypes than syndromes in genetic research.

Four problems in five recent tests of two cognitive theories of panic are discussed: (a) the ambiguity and indistinguishability of the 'body sensations' and 'cognitions' concepts; (b) the uncertain meaning of 'meaningful relationships'; (c) problems arising from differences in Clark's (Behaviour Research and Therapy, 24, 461-470, 1986) theory and Goldstein and Chambless' (Behavior Therapy, 9, 47-59, 1978) theory; (d) the ambiguity of correlational data. Some desiderata for future research on these theories are proposed.

The prediction was tested that during a simulated telephone conversation, the confidants of depressed women would provide less social support than the confidants of nondepressed psychiatric and nonpsychiatric control women. The coded speech of subjects and their confidants indicated that (a) confidants echoed the positive or negative valence of the statements made by subjects; (b) depressed women exhibited more depressotypic speech than women in both control groups; (c) confidants of depressed women also made more depressotypic statements, possibly due to the echoing phenomenon; (d) contrary to prediction, there were no significant group differences in the sequential dependencies of responses that confidants made to depressotypic or antidepressotypic subject statements; (e) contrary to prediction, the affect of depressed women's confidants was not more negative.

The literature on the distinction between primary and secondary depression is reviewed. The research data indicate that less severe, non life-threatening suicidal thoughts and behaviours occur more often in patients with secondary depression than in those with primary depression and that the prognosis for secondary depression after somatic forms of therapy is poorer than that for primary depression. The data also suggest that secondary depressives, unlike primary depressives, suffer from chronic dysphoria. It is concluded that, because the available evidence suggests that there are no qualitative differences between the episodes of primary and secondary depression, future research should concentrate on investigating the characteristics of different groups of patients with secondary depression rather than on comparisons between primary and secondary depression in general.

It has been hypothesised that community cases of depression compared with clinic cases: (a) have fewer symptoms; (b) have milder symptoms; (c) have different symptoms; (d) are of shorter duration; (e) are less incapacitating; (f) are more environmentally and less biologically caused; and (g) respond better to psychological than pharmacological intervention. A review of the literature indicates that some of these hypotheses have not been tested, others have not been adequately tested, and none has been unequivocally confirmed. Further investigations of these important issues are required.

A procedural replication of the Camberwell retrospective community study of depression in women (Brown et al. 1975; Brown & Harris, 1978a) was conducted in Calgary, Alberta. A random sample of 449 women between the ages of 18 and 65 were interviewed. The shorter form of the Present State Examination (PSE) and Brown's Interview Schedules for Life Events and Difficulties were used. By contrast to the findings of Brown, none of the following factors was associated with the onset of depression in the 12 months prior to interview: social class, employment status, number of children at home, loss of mother before age 11. In agreement with Brown, a lack of intimacy with spouse/cohabitant/boyfriend increased the risk of depression. Also in agreement with Brown, severe life events and difficulties were associated with depression. The association was particularly strong for 'possibly independent' events and difficulties, i.e. events (and difficulties) that may or may not have been caused in part by the woman herself. It was concluded that the role of social factors is community-specific and that the causal roles of events and difficulties in relation to depression remain uncertain. The implications of the findings in relation to the locus of vulnerability to depression are briefly discussed.

A frustration theory of pursuit rotor performance and reminiscence has previously been presented (Costello, 1967; Costello & Discipio, 1967). The theory was reformulated in terms of arousal theory and the following hypotheses were tested: (1) an inverted "U" relationship exists between level of arousal and performance level; (2) reminiscence results when S during the rest period moves closer to his optimum level of arousal; (3) massed practice produces a higher level of arousal than spaced practice; (4) experimental conditions producing a high drive state will also increase his arousal level. Findings in line with these hypotheses were found for EMG measures but not for GSR and pulse rate. In view of what appears to be the task-specific nature of the physiological results it was concluded that there was no particular advantage in reformulating the frustration theory in terms of arousal theory.