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1. Human IL-6 fosters long-term engraftment of patient derived disease-driving myeloma cells in immunodeficient mice

11. NOD-scid IL2rγnull mice lacking TLR4 support human immune system development and the study of human-specific innate immunity.

14. The mouse mutation “thrombocytopenia and cardiomyopathy” (trac) disrupts Abcg5: a spontaneous single gene model for human hereditary phytosterolemia/sitosterolemia

16. Enhanced development of functional human NK cells in NOD‐scid‐IL2rgnull mice expressing human IL15.

20. Development of Novel Major Histocompatibility Complex Class I and Class II-Deficient NOD-SCID IL2R Gamma Chain Knockout Mice for Modeling Human Xenogeneic Graft-Versus-Host Disease

28. Lack of acute xenogeneic graft‐versus‐host disease, but retention of T‐cell function following engraftment of human peripheral blood mononuclear cells in NSG mice deficient in MHC class I and II expression

31. Abstract 5665: NOD-scid IL2rgnull(NSG) mice deficient in murine MHC Class I and Class II expression support engraftment of functional human T cells in the absence of acute xenogeneic GVHD following injection of PBMC

36. SVEP1 plays a crucial role in epidermal differentiation

38. Chronic Proliferative Dermatitis in Sharpin Null Mice: Development of an Autoinflammatory Disease in the Absence of B and T Lymphocytes and IL4/IL13 Signaling.

40. Retrotransposon Insertion in the T-cell Acute Lymphocytic Leukemia 1 (Tal1) Gene Is Associated with Severe Renal Disease and Patchy Alopecia in Hairpatches (Hpt) Mice

45. The Tetratricopeptide Repeat Domain 7 Gene is Mutated in Flaky Skin Mice: A Model for Psoriasis, Autoimmunity, and Anemia

46. Human Lymphoid and Myeloid Cell Development in NOD/LtSz-scid IL2Rγnull Mice Engrafted with Mobilized Human Hemopoietic Stem Cells

48. NOD/LtSz-Rag1 null Pfp null mice: a new model system with increased levels of human peripheral leukocyte and hematopoietic stem-cell engraftment

50. Chronic Proliferative Dermatitis in Sharpin Null Mice: Development of an Autoinflammatory Disease in the Absence of B and T Lymphocytes and IL4/IL13 Signaling.

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