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3. Targeted immunotherapy against distinct cancer-associated fibroblasts overcomes treatment resistance in refractory HER2+ breast tumors

4. Interobserver variability in the assessment of stromal tumor-infiltrating lymphocytes (sTILs) in triple-negative invasive breast carcinoma influences the association with pathological complete response: the IVITA study

6. MiRNA-449 family is epigenetically repressed and sensitizes to doxorubicin through ACSL4 downregulation in triple-negative breast cancer

8. The GATA3 X308_Splice breast cancer mutation is a hormone context-dependent oncogenic driver

9. Clinical Impact of New Treatment Strategies for HER2-Positive Metastatic Breast Cancer Patients with Resistance to Classical Anti-HER Therapies

11. miR-146a-5p Promotes Angiogenesis and Confers Trastuzumab Resistance in HER2+ Breast Cancer

14. High VEGFR3 Expression Reduces Doxorubicin Efficacy in Triple-Negative Breast Cancer

16. Role of SALL4 in HER2+ Breast Cancer Progression: Regulating PI3K/AKT Pathway

19. Targeting HER2-AXL heterodimerization to overcome resistance to HER2 blockade in breast cancer

22. First Nationwide Molecular Screening Program in Spain for Patients With Advanced Breast Cancer: Results From the AGATA SOLTI-1301 Study

25. Abstract 1075: AXL is a potential druggable target in trastuzumab resistance in HER2+ breast cancer patients

26. The GATA3 X308_Splice breast cancer mutation is a hormone context-dependent oncogenic driver

28. Abstract PS7-71: Breast cancer fast-track programme to shorten time between initial symptoms, diagnosis and initiation of treatment. 10 years update

29. Abstract PS19-20: Identification of a novel two-microRNA signature for recurrence prediction in HER2 positive breast cancer

30. Abstract PS18-20: Frequency and spectrum of doublePIK3CAsomatic mutations in metastatic breast cancer patients

32. MicroRNA-33b Suppresses Epithelial–Mesenchymal Transition Repressing the MYC–EZH2 Pathway in HER2+ Breast Carcinoma

33. FGFR4 regulates tumor subtype differentiation in luminal breast cancer and metastatic disease

34. Abstract P6-03-05: AXL-HER2 dimer as mechanism of anti-HER2 acquired resistance in HER2 amplified brest cancer models: A new step towards precision medicine

35. HDAC5 Inhibitors as a Potential Treatment in Breast Cancer Affecting Very Young Women

36. Additional file 5: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

37. Additional file 3: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

38. Additional file 1: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

39. Additional file 8: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

40. Additional file 6: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

41. Additional file 2: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

42. Additional file 7: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

43. Additional file 4: of A two-gene epigenetic signature for the prediction of response to neoadjuvant chemotherapy in triple-negative breast cancer patients

44. The GATA3 X308_Splice breast cancer mutation is a hormone context-dependent oncogenic driver

47. Patterns of HER2 Gene Amplification and Response to Anti-HER2 Therapies

48. Trastuzumab or lapatinib with standard chemotherapy for HER2-positive breast cancer: results from the GEICAM/2006-14 trial

49. Non-canonical NF-κB pathway activation predicts outcome in borderline oestrogen receptor positive breast carcinoma

50. Differential expression of receptor activator of nuclear factor kappa B in healthy endometrium, ovarian endometrioma, and endometrioid ovarian cancer.

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