715 results on '"Burgess, Antony W."'
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2. S100 family proteins are linked to organoid morphology and EMT in pancreatic cancer
3. PI3Kα Translocation Mediates Nuclear PtdIns(3,4,5)P3 Effector Signaling in Colorectal Cancer
4. Correction to: S100 family proteins are linked to organoid morphology and EMT in pancreatic cancer
5. Phenotypic screen for oxygen consumption rate identifies an anti-cancer naphthoquinone that induces mitochondrial oxidative stress
6. Low-viscosity matrix suspension culture enables scalable analysis of patient-derived organoids and tumoroids from the large intestine
7. Transplantable programmed death ligand 1 expressing gastroids from gastric cancer prone Nfkb1−/− mice
8. Epidermal growth factor potentiates EGFR(Y992/1173)-mediated therapeutic response of triple negative breast cancer cells to cold atmospheric plasma-activated medium
9. Epidermal growth factor receptor: Structure-function informing the design of anticancer therapeutics
10. Preclinical small molecule WEHI-7326 overcomes drug resistance and elicits response in patient-derived xenograft models of human treatment-refractory tumors
11. Antitumor Efficacy of Cytotoxic Drugs and the Monoclonal Antibody 806 Is Enhanced by the EGF Receptor Inhibitor AG1478
12. Colorectal Cancer Cell Line Proteomes Are Representative of Primary Tumors and Predict Drug Sensitivity
13. Evolution of anti-HER2 therapies for cancer treatment
14. Growth Control Mechanisms in Normal and Transformed Intestinal Cells
15. The Human A33 Antigen is a Transmembrane Glycoprotein and a Novel Member of the Immunoglobulin Superfamily
16. Figure S3 from Combined Treatment with a WNT Inhibitor and the NSAID Sulindac Reduces Colon Adenoma Burden in Mice with Truncated APC
17. Data from Combined Treatment with a WNT Inhibitor and the NSAID Sulindac Reduces Colon Adenoma Burden in Mice with Truncated APC
18. Supplementary Table 5 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
19. Supplementary Table S3 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
20. Supplementary Figure 1 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
21. Supplementary Table 8 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
22. Data from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
23. Supplementary Table 9 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
24. Supplementary Data and Methods from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
25. Supplementary Methods from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
26. Supplementary Table 4 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
27. Supplementary Table 7 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
28. Supplementary Table 6 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
29. Supplementary Table 3 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
30. Supplementary Figure S1 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
31. Supplementary Table 2 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
32. Supplementary Table 1 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
33. LRIG1 Extracellular Domain: Structure and Function Analysis
34. Dual role of Src kinase in governing neuronal survival
35. EGFR Receptor Family Extracellular Domain Structures and Functions
36. Mammals I: Regulation of RAS Activation
37. Correction to “Regulation of Signaling from the Epidermal Growth Factor Family”
38. Regulation of Signaling from the Epidermal Growth Factor Family
39. Applications of Optical Biosensors to Structure-Function Studies on the EGF/EGF Receptor System
40. The PI(3)P interactome from a colon cancer cell
41. Identification of a Wnt-induced protein complex by affinity proteomics using an antibody that recognizes a sub-population of β-catenin
42. Incentives: Encouraging Adventurous Ideas
43. Antibodies Specifically Targeting a Locally Misfolded Region of Tumor Associated EGFR
44. Wnt signaling and colon tumorigenesis — A view from the periphery
45. A Phase I Clinical Trial with Monoclonal Antibody ch806 Targeting Transitional State and Mutant Epidermal Growth Factor Receptors
46. Dual drug targeting to kill colon cancers
47. TGF-beta1 induced S100 family protein expression is associated with epithelial to mesenchymal transition states and poor survival in pancreatic cancer
48. Combined Treatment with a WNT Inhibitor and the NSAID Sulindac Reduces Colon Adenoma Burden in Mice with Truncated APC
49. Quantitative intratumoural microdistribution and kinetics of 131I-huA33 antibody in patients with colorectal carcinoma
50. Murine fecal proteomics: A model system for the detection of potential biomarkers for colorectal cancer
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