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1. Biallelic variants in GTF3C3 result in an autosomal recessive disorder with intellectual disability

2. Narrowing the diagnostic gap: Genomes, episignatures, long-read sequencing, and health economic analyses in an exome-negative intellectual disability cohort

3. Dominant-negative variants in CBX1 cause a neurodevelopmental disorder

4. The clinical and molecular spectrum of the KDM6B-related neurodevelopmental disorder

5. Bi-allelic TTI1 variants cause an autosomal-recessive neurodevelopmental disorder with microcephaly

6. Whole exome and genome sequencing in mendelian disorders: a diagnostic and health economic analysis

7. De Novo ZMYND8 variants result in an autosomal dominant neurodevelopmental disorder with cardiac malformations

8. Standardized practices for RNA diagnostics using clinically accessible specimens reclassifies 75% of putative splicing variants

9. Multisystem inflammation and susceptibility to viral infections in human ZNFX1 deficiency

10. Biallelic variants in GTF3C3 result in an autosomal recessive disorder with intellectual disability

11. Whole-exome sequencing reanalysis at 12 months boosts diagnosis and is cost-effective when applied early in Mendelian disorders

12. Mutations in the Epithelial Cadherin-p120-Catenin Complex Cause Mendelian Non-Syndromic Cleft Lip with or without Cleft Palate

13. A genome-wide association study identifies susceptibility loci for nonsyndromic sagittal craniosynostosis near BMP2 and within BBS9

14. Feasibility of Ultra-Rapid Exome Sequencing in Critically Ill Infants and Children With Suspected Monogenic Conditions in the Australian Public Health Care System

15. Integration of EpiSign, facial phenotyping, and likelihood ratio interpretation of clinical abnormalities in the re‐classification of an ARID1B missense variant

16. Biallelic variants inTUBGCP6result in microcephaly and chorioretinopathy 1: Report of four cases and a literature review

18. A mutational hotspot in AMOTL1 defines a new syndrome of orofacial clefting, cardiac anomalies, and tall stature

20. Biallelic variants in TUBGCP6 result in microcephaly and chorioretinopathy 1: Report of four cases and a literature review.

21. Clinical, molecular, and cellular immunologic findings in patients with SP110-associated veno-occlusive disease with immunodeficiency syndrome

24. Standardized practices for RNA diagnostics using clinically accessible specimens reclassifies 75% of putative splicing variants

27. Feasibility of Ultra-Rapid Exome Sequencing in Critically Ill Infants and Children With Suspected Monogenic Conditions in the Australian Public Health Care System

30. Next-generation genetic testing for retinitis pigmentosa

32. Dominant-negative mutations inCBX1cause a neurodevelopmental disorder

33. CDH1 Mutation Distribution and Type Suggests Genetic Differences between the Etiology of Orofacial Clefting and Gastric Cancer

35. SLC29A3 gene is mutated in pigmented hypertrichosis with insulin-dependent diabetes mellitus syndrome and interacts with the insulin signaling pathway

38. Expanding the Spectrum of BAF-Related Disorders: De Novo Variants in SMARCC2 Cause a Syndrome with Intellectual Disability and Developmental Delay

39. De Novo ZMYND8variants result in an autosomal dominant neurodevelopmental disorder with cardiac malformations

41. Expanding the Spectrum of BAF-Related Disorders : De Novo Variants in SMARCC2 Cause a Syndrome with Intellectual Disability and Developmental Delay

43. Front Cover, Volume 40, Issue 10

45. Mutations in GDF11 and the extracellular antagonist, Follistatin, as a likely cause of Mendelian forms of orofacial clefting in humans

48. Expanding the Spectrum of BAF-Related Disorders: De Novo Variants in SMARCC2 Cause a Syndrome with Intellectual Disability and Developmental Delay

50. Reanalysis and optimisation of bioinformatic pipelines is critical for mutation detection

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