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1. MCT1 Deletion in Oligodendrocyte Lineage Cells Causes Late-Onset Hypomyelination and Axonal Degeneration

2. Lactate Transporters Mediate Glia-Neuron Metabolic Crosstalk in Homeostasis and Disease

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3. Metabolic Transporters in the Peripheral Nerve—What, Where, and Why?

4. MCT1 Deletion in Oligodendrocyte Lineage Cells Causes Late-Onset Hypomyelination and Axonal Degeneration

5. 517 Regulatory T cell functional identity is sustained by a glucose:lactate axis that is exploited in the tumor microenvironment

6. Macrophage monocarboxylate transporter 1 promotes peripheral nerve regeneration after injury in mice

7. Surprising New Players in Glia-Neuron Crosstalk: Role in CNS Regeneration

8. Reducing monocarboxylate transporter MCT1 worsens experimental diabetic peripheral neuropathy

9. Monocarboxylate transporter 1 in Schwann cells contributes to maintenance of sensory nerve myelination during aging

10. Monocarboxylate transporter 1 in Schwann cells is critical for maintenance of sensory nerve myelination during aging

11. ALSUntangled No. 36: Accilion

12. Absence of Survival and Motor Deficits in 500 Repeat C9ORF72 BAC Mice

13. ALSUntangled No. 35: Hyperbaric Oxygen Therapy*

14. ALSUntangled: Introducing The Table of Evidence

15. ALSUntangled No. 29: MitoQ

16. Oligodendroglia: metabolic supporters of axons

17. ALSUntangled 38: L-serine

18. Motor neuron disease, TDP-43 pathology, and memory deficits in mice expressing ALS-FTD-linked UBQLN2 mutations

19. ALSUntangled No. 34: GM604

20. Expanding the spectrum of monoclonal light chain deposition disease in muscle

21. Magnetic resonance imaging of mouse skeletal muscle to measure denervation atrophy

22. Genetically Decreased Spinal Cord Copper Concentration Prolongs Life in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis

23. Amyotrophic Lateral Sclerosis and Novel Therapeutic Strategies

24. ALSUntangled No. 26: lunasin

25. Early and Selective Pathology of Light Chain Neurofilament in the Spinal Cord and Sciatic Nerve of G86R Mutant Superoxide Dismutase Transgenic Mice

26. Amyotrophic lateral sclerosis associated with mutations in superoxide dismutase: a putative mechanism of degeneration

27. Determinants of neuronal vulnerability in neurodegenerative diseases

28. Superoxide dismutase and neurofilament transgenic models of amyotrophic lateral sclerosis

29. Light and electron microscopic distribution of the AMPA receptor subunit, GluR2, in the spinal cord of control and G86R mutant superoxide dismutase transgenic mice

30. Time course of neuropathology in the spinal cord of G86R superoxide dismutase transgenic mice

31. Quantitative immunocytochemical analysis of the spinal cord in G86R superoxide dismutase transgenic mice: Neurochemical correlates of selective vulnerability

32. Medication, toxic, and vitamin-related neuropathies

33. Oligodendroglia metabolically support axons and contribute to neurodegeneration

37. A soluble activin type IIB receptor improves function in a mouse model of amyotrophic lateral sclerosis

39. Deficiency in monocarboxylate transporter 1 (MCT1) in mice delays regeneration of peripheral nerves following sciatic nerve crush