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1. Incomplete-penetrant hypertrophic cardiomyopathy MYH7 G256E mutation causes hypercontractility and elevated mitochondrial respiration.

2. Far-red and sensitive sensor for monitoring real time H 2 O 2 dynamics with subcellular resolution and in multi-parametric imaging applications.

3. Structure-guided engineering of a fast genetically encoded sensor for real-time H 2 O 2 monitoring.

4. Engineered Heart Tissues for Standard 96-Well Tissue Culture Plates.

5. Multi-scale models reveal hypertrophic cardiomyopathy MYH7 G256E mutation drives hypercontractility and elevated mitochondrial respiration.

6. Full-length dystrophin deficiency leads to contractile and calcium transient defects in human engineered heart tissues.

7. A Change of Heart: Human Cardiac Tissue Engineering as a Platform for Drug Development.

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