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1. Pathogenic proteotoxicity of cryptic splicing is alleviated by ubiquitination and ER-phagy.

2. Nuclear eNOS interacts with and S-nitrosates ADAR1 to modulate type I interferon signaling and endothelial function.

3. Cytosolic N6AMT1-dependent translation supports mitochondrial RNA processing

4. Cytosolic N6AMT1- dependent translation supports mitochondrial RNA processing.

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