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2. Mutant TP53 switches therapeutic vulnerability during gastric cancer progression within interleukin-6 family cytokines

4. A tumor focused approach to resolving the etiology of DNA mismatch repair deficient tumors classified as suspected Lynch syndrome

9. DCLK1 induces a pro-tumorigenic phenotype to drive gastric cancer progression.

10. Associations between pathological features and risk of metachronous colorectal cancer.

13. Marginal zone lymphomas: a consensus practice statement from the Australasian Lymphoma Alliance.

14. Hereditary diffuse gastric cancer: updated clinical practice guidelines

18. Contributors

20. Identifying primary and secondary MLH1 epimutation carriers displaying low-level constitutional MLH1 methylation using droplet digital PCR and genome-wide DNA methylation profiling of colorectal cancers

21. Comprehensive molecular characterization of gastric adenocarcinoma

23. The Cancer Genome Atlas Comprehensive Molecular Characterization of Renal Cell Carcinoma

24. Spatial Organization and Molecular Correlation of Tumor-Infiltrating Lymphocytes Using Deep Learning on Pathology Images

25. Molecular Characterization and Clinical Relevance of Metabolic Expression Subtypes in Human Cancers

26. Pan-Cancer Analysis of lncRNA Regulation Supports Their Targeting of Cancer Genes in Each Tumor Context

27. Genomic, Pathway Network, and Immunologic Features Distinguishing Squamous Carcinomas

28. Integrated Genomic Analysis of the Ubiquitin Pathway across Cancer Types

29. Machine Learning Detects Pan-cancer Ras Pathway Activation in The Cancer Genome Atlas

30. Systematic Analysis of Splice-Site-Creating Mutations in Cancer

31. Driver Fusions and Their Implications in the Development and Treatment of Human Cancers

32. Genomic and Molecular Landscape of DNA Damage Repair Deficiency across The Cancer Genome Atlas

33. Somatic Mutational Landscape of Splicing Factor Genes and Their Functional Consequences across 33 Cancer Types

36. Supplementary Tables from DNA Methylation in Peripheral Blood and Risk of Gastric Cancer: A Prospective Nested Case–control Study

37. Figure S5 from Rapid Resistance of FGFR-driven Gastric Cancers to Regorafenib and Targeted FGFR Inhibitors can be Overcome by Parallel Inhibition of MEK

38. Table S1 from Rapid Resistance of FGFR-driven Gastric Cancers to Regorafenib and Targeted FGFR Inhibitors can be Overcome by Parallel Inhibition of MEK

39. Data from Rapid Resistance of FGFR-driven Gastric Cancers to Regorafenib and Targeted FGFR Inhibitors can be Overcome by Parallel Inhibition of MEK

40. Data from DNA Methylation in Peripheral Blood and Risk of Gastric Cancer: A Prospective Nested Case–control Study

44. Supp Legends and Table S1 and S2 from Frizzled-7 Is Required for Wnt Signaling in Gastric Tumors with and Without Apc Mutations

45. Data from Frizzled-7 Is Required for Wnt Signaling in Gastric Tumors with and Without Apc Mutations

47. Fig S7 from Frizzled-7 Is Required for Wnt Signaling in Gastric Tumors with and Without Apc Mutations

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