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130 results on '"Bourdon, J. -C."'

8. Scotin, a novel p53-inducible proapoptotic protein located in the ER and the nuclear membrane

Author

Bourdon, J.-C., Renzing, J., Robertson, P.L., Fernandes, K.N., and Lane, D.P.

Subjects
Cytochemistry -- Research, Tumor suppressor genes -- Physiological aspects, Endoplasmic reticulum -- Physiological aspects, Cell death -- Genetic aspects, Nuclear membranes -- Physiological aspects, Biological sciences
Abstract

p53 is a transcription factor that induces growth arrest or apoptosis in response to cellular stress. To identify new p53-inducible proapoptotic genes, we compared, by differential display, the expression of genes in spleen or thymus of normal and p53 nullizygote mice after [gamma]-irradiation of whole animals. We report the identification and characterization of human and mouse Scotin homologues, a novel gene directly transactivated by p53. The Scotin protein is localized to the ER and the nuclear membrane. Scotin can induce apoptosis in a caspase-dependent manner. Inhibition of endogenous Scotin expression increases resistance to p53-dependent apoptosis induced by DNA damage, suggesting that Scotin plays a role in p53-dependent apoptosis. The discovery of Scotin brings to light a role of the ER in p53-dependent apoptosis.

Published
2002

10. Photo-fission for the production of radioactive beams ALTO project

Author

Essabaa, S., Arianer, J., Ausset, P., Bajeat, O., Baronick, J.P., Clapier, F., Coacolo, L., Donzaud, C., Ducourtieux, M., Galès, S., Gardès, D., Grialou, D., Hosni, F., Guillemaud-Mueller, D., Ibrahim, F., Junquera, T., Lau, C., Le Blanc, F., Lefort, H., Le Scornet, J.C., Lesrel, J., Mueller, A.C., Obert, J., Perru, O., Potier, J.C., Proust, J., Pougheon, F., Roussière, B., Rouvière, N., Sauvage, J., Sorlin, O., Tkatchenko, A., Verney, D., Waast, B., Rinolfi, L., Rossat, G., Forkel-Wirth, D., Muller, A., Bienvenu, G., Bourdon, J.-C., Garvey, T., Jacquemard, B., and Omeich, M.

Published
2003
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11. Modulation of p53β and p53γ expression by regulating the alternative splicing of TP53 gene modifies cellular response

Author

Marcel, V, Fernandes, K, Terrier, O, Lane, D P, and Bourdon, J-C

Subjects
Original Paper, Alternative Splicing, Humans, Tumor Suppressor Protein p53
Abstract

In addition to the tumor suppressor p53 protein, also termed p53α, the TP53 gene produces p53β and p53γ through alternative splicing of exons 9β and 9γ located within TP53 intron 9. Here we report that both TG003, a specific inhibitor of Cdc2-like kinases (Clk) that regulates the alternative splicing pre-mRNA pathway, and knockdown of SFRS1 increase expression of endogenous p53β and p53γ at mRNA and protein levels. Development of a TP53 intron 9 minigene shows that TG003 treatment and knockdown of SFRS1 promote inclusion of TP53 exons 9β/9γ. In a series of 85 primary breast tumors, a significant association was observed between expression of SFRS1 and α variant, supporting our experimental data. Using siRNA specifically targeting exons 9β/9γ, we demonstrate that cell growth can be driven by modulating p53β and p53γ expression in an opposite manner, depending on the cellular context. In MCF7 cells, p53β and p53γ promote apoptosis, thus inhibiting cell growth. By transient transfection, we show that p53β enhanced p53α transcriptional activity on the p21 and Bax promoters, while p53γ increased p53α transcriptional activity on the Bax promoter only. Moreover, p53β and p53γ co-immunoprecipitate with p53α only in the presence of p53-responsive promoter. Interestingly, although p53β and p53γ promote apoptosis in MCF7 cells, p53β and p53γ maintain cell growth in response to TG003 in a p53α-dependent manner. The dual activities of p53β and p53γ isoforms observed in non-treated and TG003-treated cells may result from the impact of TG003 on both expression and activities of p53 isoforms. Overall, our data suggest that p53β and p53γ regulate cellular response to modulation of alternative splicing pre-mRNA pathway by a small drug inhibitor. The development of novel drugs targeting alternative splicing process could be used as a novel therapeutic approach in human cancers.

Published
2014

13. The study of a new PARRNe experimental area using an electron linac close to the Orsay tandem

Author

Essabaa, S., Arianer, J., Ausset, P., Baronick, J P., Bergot, J P., Boulot, A., Clapier, F., Coacolo, J L., Curaudeau, J M., Dupont, F., Gales, S., Gardes, D., Grialou, D., Ibrahim, F., Junquera, T., Kandry-Rody, S., Lefort, H., Le Scornet, J C., Lesrel, J., M'Garrech, S., Mueller, a C., Rouviere, N., Tkatchenko, A., Waast, B., Rinolfi, L., Rossat, G., Bienvenu, G., Bourdon, J C., Garvey, T., Jacquemard, B., Omeich, M., Institut de Physique Nucléaire d'Orsay (IPNO), Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Paris-Sud - Paris 11 (UP11), Laboratoire de l'Accélérateur Linéaire (LAL), Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), and Kerec, Hélène

Subjects
[PHYS.PHYS.PHYS-INS-DET] Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det], Astrophysics::High Energy Astrophysical Phenomena, Nuclear Theory, Physics::Accelerator Physics, [PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det], Nuclear Experiment, Accelerators and Storage Rings
Abstract

The Production of neutron-rich radioactive nuclei through fission is currently prime of research interest for the future radioactive beam facilities. For example in the EURISOL[1] project, photo-fission and fast neutron induced fission are proposed. The photo-fission cross-section for 238U is about 0.16 barn (against 1.6 barn for fast neutrons of 40 MeV) but the conversion electrons/gammas is much more efficient than that of deuterons/neutrons. It was necessary, to test this new method of production, to carry out, in equivalent conditions, an experiment of the type PARRNe-1 using a 50 MeV electron beam. In April 2001, production of fission fragments induced by gammas proved to be successful. Bremsstrahlung gamma rays were produced by the few nA-50 MeV electron beam delivered by the CERN LEP Injector Linac (LIL). This promising alternative has stimulated the study of a new experimental area at IPNO based on an electron Linac close to the Tandem, through a collaboration with LAL and CERN PS groups.

Published
2002

14. ELYSE - an intense linac for pulsed radiolysis research

Author

Garvey, T., Bernard, M., Borie, H., Bourdon, J-C., Jacquemard, B., Leblond, B., Lepercq, P., Omeich, M., Roch, M., Rodier, J., Roux, R., Gobert, FF., Monard, H., Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Subjects
[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]
Published
2002

15. Generation of GW radiation pulses from a VUV free- electron laser operating in the femtosecond regime

Author

Ayvazyan, V., Baboi, N., Bakker, R., Balakin, V., Barni, D., Bazhan, A., Bernard, M., Bosotti, A., Bourdon, J C., Brefeld, W., Brinkmann, R., Buhler, S., Carneiro, J P., Castellano, M G., Castro, P., Catani, L., Chel, S., Cho, Y., Choroba, S., Colby, E R., Decking, W., Den Hartog, P., Desmons, M., Dohlus, M., Edwards, D., Edwards, H T., Faatz, B., Feldhaus, J., Ferrario, M., Fitch, M J., Flottmann, K., Fouaidy, M., Gamp, A., Garvey, T., Geitz, M., Gluskin, E., Gretchko, V., Hahn, U., Hartung, W H., Hubert, D., Huning, M., Ischebek, R., Jablonka, M., Joly, J M., Juillard, M., Junquera, T., Jurkiewicz, P., Kabel, A., Kahl, J., Kaiser, H., Kamps, T., Katelev, V V., Kirchgessner, J L., Korfer, M., Kravchuk, L., Kreps, G., Krzywinski, J., Lokajczyk, T., Lange, R., Leblond, B., Leenen, M., Lesrel, J., Liepe, M., Liero, A., Limberg, T., Lorenz, R., Lu, H H., Lu, F H., Magne, C., Maslov, M., Materlik, G., Matheisen, A., Menzel, J., Michelato, P., Moller, W D., Mosnier, A., Muller, U C., Napoly, O., Novokhatski, A V., Omeich, M., Padamsee, H S., Pagani, C., Peters, F., Petersen, B., Pierini, P., Pfluger, J., Piot, P., Phung Ngoc, B., Plucinski, L., Proch, D., Rehlich, K., Reiche, S., Reschke, D., Reyzl, I., Rosenzweig, J., Rossbach, J., Roth, S., Saldin, E L., Sandner, W., Sanok, Z., Schlarb, H., Schmidt, G., Schmuser, P., Schneider, J R., Schneidmiller, E A., Schreiber, H J., Schreiber, S., Schutt, P., Sekutowicz, J., Serafini, L., Sertore, D., Setzer, S., Simrock, S., Sonntag, B F., Sparr, B., Stephan, F., Sytchev, V A., Tazzari, S., Tazzioli, F., Tigner, M., Timm, M., Tonutti, M., Trakhtenberg, E., Treusch, R., Trines, D., Verzilov, V., Vielitz, T., Vogel, V., Von Walter, G., Wanzenberg, R., Weiland, T., Weise, H., Weisend, J., Wendt, M., Werner, M., White, M M., Will, I., Wolff, S., Yurkov, M V., Zapfe, K., Zhogolev, P., Zhou, F., Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Subjects
[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]
Published
2002

16. L'accelerateur du projet ELYSE

Author

Marignier, J.-L., Belloni, J., Gaillard, M., Fuchs, Alain H., Bourdon, J.-C., Monard, H., Lampre, I., Remita, H., Mostafavi, M., Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Subjects
[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]
Published
2000

17. New picosecond electron accelerator ELYSE at Orsay Paris-Sud

Author

Belloni, J., Gaillard, M., Bourdon, J.-C., Monard, H., Mostafavi, M., Marignier, J.-L., Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Subjects
[PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]
Published
1999

18. Communications aux journees accelerateurs : - L'accelerateur lineaire du projet ELYSE - Caracterisation du photoinjecteur - TIF : l'accelerateur et son programme experimental

Author

Bourdon, J.-C., Devanz, G., Garvey, T., Kerec, Hélène, Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Subjects
[PHYS.PHYS.PHYS-INS-DET] Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det], [PHYS.PHYS.PHYS-INS-DET]Physics [physics]/Physics [physics]/Instrumentation and Detectors [physics.ins-det]
Published
1998

19. Status of the TTF Linac Injector

Author

Garvey, Terence, Bernard, M Y, Bourdon, J C, Chehab, R, Dufresne, P, Jacquemard, B, Mencik, M, Mouton, B, Omeich, M, Roch, M, Rodier, J, Roudier, P, Saury, J L, Solyak, N A, Taurigna-Quere, M, Thiery, Y, Aune, B, Desmons, M, Fusellier, J, Gastebois, J, Gougnaud, F, Gournay, J F, Jablonka, M, Joly, J M, Juillard, M, Long, H, Lussignol, Y, Mosnier, A, Phung Ngoc, B, Bühler, S, and Junquera, T

Subjects
Accelerators and Storage Rings
Published
1996

37. First Observation of Self-Amplified Spontaneous Emission in a Free-Electron Laser at 109 nm Wavelength

Author

Andruszkow, J., primary, Aune, B., additional, Ayvazyan, V., additional, Baboi, N., additional, Bakker, R., additional, Balakin, V., additional, Barni, D., additional, Bazhan, A., additional, Bernard, M., additional, Bosotti, A., additional, Bourdon, J. C., additional, Brefeld, W., additional, Brinkmann, R., additional, Buhler, S., additional, Carneiro, J.-P., additional, Castellano, M., additional, Castro, P., additional, Catani, L., additional, Chel, S., additional, Cho, Y., additional, Choroba, S., additional, Colby, E. R., additional, Decking, W., additional, Den Hartog, P., additional, Desmons, M., additional, Dohlus, M., additional, Edwards, D., additional, Edwards, H. T., additional, Faatz, B., additional, Feldhaus, J., additional, Ferrario, M., additional, Fitch, M. J., additional, Flöttmann, K., additional, Fouaidy, M., additional, Gamp, A., additional, Garvey, T., additional, Gerth, C., additional, Geitz, M., additional, Gluskin, E., additional, Gretchko, V., additional, Hahn, U., additional, Hartung, W. H., additional, Hubert, D., additional, Hüning, M., additional, Ischebek, R., additional, Jablonka, M., additional, Joly, J. M., additional, Juillard, M., additional, Junquera, T., additional, Jurkiewicz, P., additional, Kabel, A., additional, Kahl, J., additional, Kaiser, H., additional, Kamps, T., additional, Katelev, V. V., additional, Kirchgessner, J. L., additional, Körfer, M., additional, Kravchuk, L., additional, Kreps, G., additional, Krzywinski, J., additional, Lokajczyk, T., additional, Lange, R., additional, Leblond, B., additional, Leenen, M., additional, Lesrel, J., additional, Liepe, M., additional, Liero, A., additional, Limberg, T., additional, Lorenz, R., additional, Hua, Lu Hui, additional, Hai, Lu Fu, additional, Magne, C., additional, Maslov, M., additional, Materlik, G., additional, Matheisen, A., additional, Menzel, J., additional, Michelato, P., additional, Möller, W.-D., additional, Mosnier, A., additional, Müller, U.-C., additional, Napoly, O., additional, Novokhatski, A., additional, Omeich, M., additional, Padamsee, H. S., additional, Pagani, C., additional, Peters, F., additional, Petersen, B., additional, Pierini, P., additional, Pflüger, J., additional, Piot, P., additional, Phung Ngoc, B., additional, Plucinski, L., additional, Proch, D., additional, Rehlich, K., additional, Reiche, S., additional, Reschke, D., additional, Reyzl, I., additional, Rosenzweig, J., additional, Rossbach, J., additional, Roth, S., additional, Saldin, E. L., additional, Sandner, W., additional, Sanok, Z., additional, Schlarb, H., additional, Schmidt, G., additional, Schmüser, P., additional, Schneider, J. R., additional, Schneidmiller, E. A., additional, Schreiber, H.-J., additional, Schreiber, S., additional, Schütt, P., additional, Sekutowicz, J., additional, Serafini, L., additional, Sertore, D., additional, Setzer, S., additional, Simrock, S., additional, Sonntag, B., additional, Sparr, B., additional, Stephan, F., additional, Sytchev, V. A., additional, Tazzari, S., additional, Tazzioli, F., additional, Tigner, M., additional, Timm, M., additional, Tonutti, M., additional, Trakhtenberg, E., additional, Treusch, R., additional, Trines, D., additional, Verzilov, V., additional, Vielitz, T., additional, Vogel, V., additional, Walter, G. v., additional, Wanzenberg, R., additional, Weiland, T., additional, Weise, H., additional, Weisend, J., additional, Wendt, M., additional, Werner, M., additional, White, M. M., additional, Will, I., additional, Wolff, S., additional, Yurkov, M. V., additional, Zapfe, K., additional, Zhogolev, P., additional, and Zhou, F., additional

Published
2000
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38. Drosophila p53 isoforms differentially regulate apoptosis and apoptosis-induced proliferation.

Author

Dichtel-Danjoy, M-L, Ma, D, Dourlen, P, Chatelain, G, Napoletano, F, Robin, M, Corbet, M, Levet, C, Hafsi, H, Hainaut, P, Ryoo, H D, Bourdon, J-C, and Mollereau, B

Subjects
DROSOPHILA, APOPTOSIS, CELL proliferation, AUTOPHAGY, ADAPTOR proteins, DISEASE risk factors
Abstract

Irradiated or injured cells enter apoptosis, and in turn, promote proliferation of surrounding unaffected cells. In Drosophila, apoptotic cells have an active role in proliferation, where the caspase Dronc and p53 induce mitogen expression and growth in the surrounding tissues. The Drosophila p53 gene structure is conserved and encodes at least two protein isoforms: a full-length isoform (Dp53) and an N-terminally truncated isoform (DΔNp53). Historically, DΔNp53 was the first p53 isoform identified and was thought to be responsible for all p53 biological activities. It was shown that DΔNp53 induces apoptosis by inducing the expression of IAP antagonists, such as Reaper. Here we investigated the roles of Dp53 and DΔNp53 in apoptosis and apoptosis-induced proliferation. We found that both isoforms were capable of activating apoptosis, but that they each induced distinct IAP antagonists. Expression of DΔNp53 induced Wingless (Wg) expression and enhanced proliferation in both 'undead cells' and in 'genuine' apoptotic cells. In contrast to DΔNp53, Dp53 did not induce Wg expression in the absence of the endogenous p53 gene. Thus, we propose that DΔNp53 is the main isoform that regulates apoptosis-induced proliferation. Understanding the roles of Drosophila p53 isoforms in apoptosis and in apoptosis-induced proliferation may shed new light on the roles of p53 isoforms in humans, with important implications in cancer biology. [ABSTRACT FROM AUTHOR]

Published
2013
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39. Operation of the CLIO accelerator

Author

Chaput, R., primary, Glotin, F., additional, Humbert, G., additional, Kergosien, B., additional, Lesrel, J., additional, Ortega, J.-M., additional, Serin, L., additional, Bourdon, J.-C., additional, Bernard, M., additional, Coulle, H., additional, Dehamme, M., additional, Garvey, T., additional, Mencik, M., additional, Mouton, B., additional, Rodier, J., additional, Roudier, P., additional, and Taurigna, M., additional

Published
1993
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40. Diverse p63 and p73 isoforms regulate Δ133p53 expression through modulation of the internal TP53 promoter activity.

Author

Marcel, V, Petit, I, Murray-Zmijewski, F, Goullet de Rugy, T, Fernandes, K, Meuray, V, Diot, A, Lane, D P, Aberdam, D, and Bourdon, J-C

Subjects
GENE expression, CHROMATIN, DNA-binding proteins, KERATINOCYTES, ECTODERMAL dysplasia, PATIENTS
Abstract

In response to stress, p53 binds and transactivates the internal TP53 promoter, thus regulating the expression of its own isoform, Δ133p53α. Here, we report that, in addition to p53, at least four p63/p73 isoforms regulate Δ133p53 expression at transcriptional level: p63β, ΔNp63α, ΔNp63β and ΔNp73γ. This regulation occurs through direct DNA-binding to the internal TP53 promoter as demonstrated by chromatin immunoprecipitation and the use of DNA-binding mutant p63. The promoter regions involved in the p63/p73-mediated transactivation were identified using deleted, mutant and polymorphic luciferase reporter constructs. In addition, we observed that transient expression of p53 family members modulates endogenous Δ133p53α expression at both mRNA and protein levels. We also report concomitant variation of p63 and Δ133p53 expression during keratinocyte differentiation of HaCat cells and induced pluripotent stem cells derived from mutated p63 ectodermal dysplasia patients. Finally, proliferation assays indicated that Δ133p53α isoform regulates the anti-proliferative activities of p63β, ΔNp63α, ΔNp63β and ΔNp73γ. Overall, this study shows a strong interplay between p53, p63 and p73 isoforms to orchestrate cell fate outcome. [ABSTRACT FROM AUTHOR]

Published
2012
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41. p53 directly transactivates Δ133p53α, regulating cell fate outcome in response to DNA damage.

Author

Aoubala, M., Murray-Zmijewski, F., Khoury, M. P., Fernandes, K., Perrier, S., Bernard, H., Prats, A.-C., Lane, D. P., and Bourdon, J.-C.

Subjects
P53 antioncogene, DNA damage, CELL cycle, APOPTOSIS, DOXORUBICIN
Abstract

We have previously reported that the human p53 gene encodes at least nine different p53 isoforms, including Δ133p53α, which can modulate p53 transcriptional activity and apoptosis. In this study, we aimed to investigate the regulation of Δ133p53α isoform expression and its physiological role in modulating cell cycle arrest and apoptosis. We report here that in response to a low dose of doxorubicin (which induces cell cycle arrest without promoting apoptosis), p53 directly transactivates the human p53 internal promoter, inducing Δ133p53α protein expression. The induced Δ133p53α then inhibits p53-dependent apoptosis and G1 arrest without inhibiting p53-dependent G2 arrest. Therefore, endogenous Δ133p53α does not exclusively function in a dominant-negative manner toward p53, but differentially regulates cell cycle arrest and apoptosis. [ABSTRACT FROM AUTHOR]

Published
2011
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42. Human and mouse Fas (APO-1/CD95) death receptor genes each contain a p53-responsive element that is activated by p53 mutants unable to induce apoptosis.

Author

Munsch, D, Watanabe-Fukunaga, R, Bourdon, J C, Nagata, S, May, E, Yonish-Rouach, E, and Reisdorf, P

Abstract

p53 is a tumor suppressor protein that induces apoptosis at least in part through its ability to act as a sequence-specific transactivator. This work reports that intron 1 of the mouse Fas death receptor gene contains a p53-responsive element (p53RE) that matches the p53 consensus sequence and that is located between nucleotides +1704 and +1723 from the transcription initiation site. This element is specifically bound by p53 and functions as a p53-dependent enhancer in mammalian or in yeast reporter gene assays. Contrary to bax, another known pro-apoptotic p53-target gene, both mouse and human FAS p53REs are still activated by the discriminatory p53 mutants Pro-175 and Ala-143, a class of mutants unable to induce apoptosis. We propose that p53-dependent up-regulation of Fas does not induce apoptosis per se but sensitizes the cell to other pro-apoptotic signal(s). The functional conservation of p53-dependent Fas up-regulation argues strongly in favor of its biological importance and suggests that murine models may be used to study further the in vivo role of Fas in the p53 response.

Published
2000

44. First experience with the LEP Pre-Injector (LPI)

Author

Madsen, J H B, Battisti, S, Blechschmidt, Diether, Delahaye, J P, Frammery, Bertrand, Hübner, K, Kugler, H, Krusche, A, Poncet, Alain, Potier, J P, Riche, A J, Warner, D J, Bourdon, J C, and Brunet, P

Subjects
Accelerators and Storage Rings
Published
1987

45. Rapport d'études sur le projet des linacs injecteur de LEP (LIL)

Author

Belbéoch, R, Bienvenu, G, Bourdon, J C, Brunet, P, Chaput, R, Chehab, R, Costa, S, Dabin, Y, Danon, G, Dardenne, G, Dupont, F, Hasselsweiller, J M, Le Duff, J, Le Meur, G, Mélard, L, Michaud, G, Mossetti, M, Mouton, B, Nalin, G, Nguyen-Ngoc, C, Perrine, J P, Renard, M, Roch, M, Rodier, J, Roudier, P, and Villain, J L

Subjects
Accelerators and Storage Rings
Published
1982

46. Report on the running-in of the electron injection system of LIL-W

Author

Bourdon, J C, Brunet, P, Mouton, B, Plouviez, E, Battisti, S, Bellanger, A, Bertolotto, R, Blechschmidt, D, Delahaye, J P, Frammery, B, Hübner, K, Madsen, J H B, Marti, P, Riche, A, and Warner, D J

Subjects
Accelerators and Storage Rings
Published
1986

47. Contributed paper to the 1984 Linac conference

Author

Dupont, F., Bienvenu, G., Bourdon, J.-C., Brunet, P., Rodier, J., Plouviez, E., Chaput, R., Laboratoire de l'Accélérateur Linéaire (LAL), and Université Paris-Sud - Paris 11 (UP11)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)

Published
1984

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