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1. Reprogramming of endothelial gene expression by tamoxifen inhibits angiogenesis and ERα-negative tumor growth

2. Author Correction: Dimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptor

3. Dimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptor

4. Supplementary Figure 3 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

5. Data from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

6. Supplementary Figure 5 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

7. Supplementary Figure 6 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

8. Supplementary Figure 2 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

9. Supplementary Figure 4 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

10. Supplementary Figure 1 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

11. Supplementary Methods, Table 1 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

12. Supplementary Figure 7 from Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

13. Mutation of the palmitoylation site of estrogen receptor α in vivo reveals tissue-specific roles for membrane versus nuclear actions

17. Comparative investigation of the pathogenicity of three Mycobacterium tuberculosis mutants defective in the synthesis of p-hydroxybenzoic acid derivatives

18. Vers une optimisation de la modulation du récepteur des œstrogènes dans le traitement hormonal de la ménopause

19. Additional file 1: Figure S1. of The AF-1-deficient estrogen receptor ERα46 isoform is frequently expressed in human breast tumors

20. The AF-1-deficient estrogen receptor ERα46 isoform is frequently expressed in human breast tumors

21. Mycobacterium tuberculosis exploits the formation of new blood vessels for its dissemination

22. Effets membranaires du récepteur alpha des œstrogènes

23. 0053: The estrogen receptor alpha C451 palmitoylation site is absolutely required for vascular membrane-initiated action of estrogens in mice

24. Mutation of the palmitoylation site of estrogen receptor α in vivo reveals tissue-specific roles for membrane versus nuclear actions

25. Stromal Estrogen Receptor-α Promotes Tumor Growth by Normalizing an Increased Angiogenesis

26. Functional characterization of the Mycobacterium tuberculosis serine/threonine kinase PknJ

27. A murine DC-SIGN homologue contributes to early host defense against Mycobacterium tuberculosis

28. A Point Mutation in the Two-Component Regulator PhoP-PhoR Accounts for the Absence of Polyketide-Derived Acyltrehaloses but Not That of Phthiocerol Dimycocerosates in Mycobacterium tuberculosis H37Ra

29. Signature-Tagged Transposon Mutagenesis Identifies Novel Mycobacterium tuberculosis Genes Involved in the Parasitism of Human Macrophages

31. Signature-Tagged Transposon Mutagenesis Identifies Novel Mycobacterium tuberculosisGenes Involved in the Parasitism of Human Macrophages

32. Signature-tagged transposon mutagenesis identifies novel mycobacterium tuberculosis genes involved in the parasitism of human macrophages

33. Functional characterization of the Mycobacterium tuberculosis serine/threonine kinase PknJ.

34. [Towards an optimization of the modulation of the estrogen receptor during menopausal hormonal therapy].

35. [Tissue-specific membrane effects of estrogen receptor alpha].

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