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44 results on '"Boscaro, Elisa"'

2. Features, reason for testing, and changes with time of 583 paroxysmal nocturnal hemoglobinuria clones from 529 patients: a multicenter Italian study

Author

Cannizzo, Elisa, Raia, Maddalena, De Propris, Maria Stefania, Triolo, Anna, Scarpati, Barbara, Marfia, Anna, Stacchini, Alessandra, Buccisano, Francesco, Lanza, Francesco, Regazzoli, Antonio, Michelutti, Angela, Cesaro, Simone, Conte, Cinzia Armentano, Vanelli, Laura, Tedone, Elisabetta, Omedè, Paola, Ciriello, Maria Matilde, Caporale, Roberto, Catinella, Virginia, Pantano, Giorgia, De Rosa, Clorinda, Lo Pardo, Catia, Poletti, Giovanni, Ulbar, Francesca, Pavanelli, Maria Cristina, Del Pup, Laura, Ottaviano, Virginia, Santonocito, Anna Maria, Bartocci, Chiara, Boscaro, Elisa, Arras, Marcella, Amodeo, Rachele, Mestice, Anna, Oliva, Bianca, Ferrari, Luisa, Statuto, Teodora, D’Auria, Fiorella, Pianezze, Graziano, Tanca, Donatella, Visconte, Feliciano, Rubba, Fabiana, Musto, Pellegrino, Geuna, Massimo, Gatti, Arianna, Brando, Bruno, and Del Vecchio, Luigi

Published
2019
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9. Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression

Author

Albiero, Mattia, Rattazzi, Marcello, Menegazzo, Lisa, Boscaro, Elisa, Cappellari, Roberta, Pagnin, Elisa, Bertacco, Elisa, Poncina, Nicol, Dyar, Kenneth, Ciciliot, Stefano, Iwabuchi, Kazuya, Millioni, Renato, Arrigoni, Giorgio, Kraenkel, Nicolle, Landmesser, Ulf, Agostini, Carlo, Avogaro, Angelo, and Fadini, Gian Paolo

Published
2013
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19. STAT3 mutation impacts biological and clinical features of T-LGL leukemia

Author

Teramo, Antonella, primary, Barilà, Gregorio, additional, Calabretto, Giulia, additional, Ercolin, Chiara, additional, Lamy, Thierry, additional, Moignet, Aline, additional, Roussel, Mikael, additional, Pastoret, Cédric, additional, Leoncin, Matteo, additional, Gattazzo, Cristina, additional, Cabrelle, Anna, additional, Boscaro, Elisa, additional, Teolato, Sara, additional, Pagnin, Elisa, additional, Berno, Tamara, additional, De March, Elena, additional, Facco, Monica, additional, Piazza, Francesco, additional, Trentin, Livio, additional, Semenzato, Gianpietro, additional, and Zambello, Renato, additional

Published
2017
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20. Phenotypic Heterogeneity of Chronic Lymphoproliferative Disorder of NK Cells

Author

Gregorio, Barila', primary, Teramo, Antonella, additional, Calabretto, Giulia, additional, Ercolin, Chiara, additional, Lico, Albana, additional, Branca, Antonio, additional, Berno, Tamara, additional, Cabrelle, Anna, additional, Boscaro, Elisa, additional, Facco, Monica, additional, Trentin, Livio, additional, Piazza, Francesco, additional, Semenzato, Gianpietro, additional, and Zambello, Renato, additional

Published
2015
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21. Integrated CLL Scoring System, a New and Simple Index to Predict Time to Treatment and Overall Survival in Patients With Chronic Lymphocytic Leukemia

Author

Visentin, Andrea, primary, Facco, Monica, additional, Frezzato, Federica, additional, Castelli, Monica, additional, Trimarco, Valentina, additional, Martini, Veronica, additional, Gattazzo, Cristina, additional, Severin, Filippo, additional, Chiodin, Giorgia, additional, Martines, Annalisa, additional, Bonaldi, Laura, additional, Gianesello, Ilaria, additional, Pagnin, Elisa, additional, Boscaro, Elisa, additional, Piazza, Francesco, additional, Zambello, Renato, additional, Semenzato, Gianpietro, additional, and Trentin, Livio, additional

Published
2015
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26. Comparative Analysis of NK Receptor and T-Cell Receptor Repertoires in Patients with Chronic Myeloid Leukemia Treated with Different Tyrosine Kinase Inhibitors

Author

Binotto, Gianni, primary, Frison, Luca, additional, Boscaro, Elisa, additional, Zambello, Renato, additional, Lessi, Federica, additional, Parolo, Anna, additional, Piazza, Francesco, additional, Bertorelle, Roberta, additional, Bonaldi, Laura, additional, and Semenzato, Gianpietro, additional

Published
2014
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29. LGL Disorders: From An Inflammatory-Mediated To a Self-Maintaining Proliferation

Author

Teramo, Antonella, primary, Gattazzo, Cristina, additional, Passeri, Francesca, additional, Barilà, Gregorio, additional, Bovo, Enrica, additional, Nardin, Chiara, additional, Campagnaro, Alberto, additional, Cabrelle, Anna, additional, Martini, Veronica, additional, Boscaro, Elisa, additional, Branca, Antonio, additional, Facco, Monica, additional, Trentin, Livio, additional, Semenzato, Gianpietro, additional, and Zambello, Renato, additional

Published
2013
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30. Stem cell compartmentalization in diabetes and high cardiovascular risk reveals the role of DPP-4 in diabetic stem cell mobilopathy

Author

Fadini, Gian Paolo, primary, Albiero, Mattia, additional, Seeger, Florian, additional, Poncina, Nicol, additional, Menegazzo, Lisa, additional, Angelini, Annalisa, additional, Castellani, Chiara, additional, Thiene, Gaetano, additional, Agostini, Carlo, additional, Cappellari, Roberta, additional, Boscaro, Elisa, additional, Zeiher, Andreas, additional, Dimmeler, Stefanie, additional, and Avogaro, Angelo, additional

Published
2012
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32. Intrinsic and Estrinsic Mechanism Contributes to Maintain the JAK/STAT Pathway Aberrantly Activated in T-Type Large Granular Lymphocyte Leukemia

Author

Zambello, Renato, primary, Teramo, Antonella, additional, Gattazzo, Cristina, additional, Passeri, Francesca, additional, Lico, Albana, additional, Tasca, Giulia, additional, Cabrelle, Anna, additional, Frezzato, Federica, additional, Martini, Veronica, additional, Trimarco, Valentina, additional, Berno, Tamara, additional, Boscaro, Elisa, additional, Facco, Monica, additional, Trentin, Livio, additional, and Semenzato, Gianpietro, additional

Published
2011
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34. Widespread Increase in Myeloid Calcifying Cells Contributes to Ectopic Vascular Calcification in Type 2 Diabetes

Author

Fadini, Gian Paolo, primary, Albiero, Mattia, additional, Menegazzo, Lisa, additional, Boscaro, Elisa, additional, Vigili de Kreutzenberg, Saula, additional, Agostini, Carlo, additional, Cabrelle, Anna, additional, Binotto, Gianni, additional, Rattazzi, Marcello, additional, Bertacco, Elisa, additional, Bertorelle, Roberta, additional, Biasini, Lorena, additional, Mion, Monica, additional, Plebani, Mario, additional, Ceolotto, Giulio, additional, Angelini, Annalisa, additional, Castellani, Chiara, additional, Menegolo, Mirko, additional, Grego, Franco, additional, Dimmeler, Stefanie, additional, Seeger, Florian, additional, Zeiher, Andreas, additional, Tiengo, Antonio, additional, and Avogaro, Angelo, additional

Published
2011
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36. Selective estrogen receptor‐α agonist provides widespread heart and vascular protection with enhanced endothelial progenitor cell mobilization in the absence of uterotrophic action

Author

Bolego, Chiara, primary, Rossoni, Giuseppe, additional, Fadini, Gian Paolo, additional, Vegeto, Elisabetta, additional, Pinna, Christian, additional, Albiero, Mattia, additional, Boscaro, Elisa, additional, Agostini, Carlo, additional, Avogaro, Angelo, additional, Gaion, Rosa Maria, additional, and Cignarella, Andrea, additional

Published
2010
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37. Effects of androgens on endothelial progenitor cellsin vitroandin vivo

Author

Fadini, Gian Paolo, primary, Albiero, Mattia, additional, Cignarella, Andrea, additional, Bolego, Chiara, additional, Pinna, Christian, additional, Boscaro, Elisa, additional, Pagnin, Elisa, additional, De Toni, Renzo, additional, De Kreutzenberg, Saula, additional, Agostini, Carlo, additional, and Avogaro, Angelo, additional

Published
2009
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39. KIR/HLA-I Mismatching Predicts Risk of Relapse in Pediatric Patients Undergoing Allogeneic Hematopoietic Stem Cell Transplantation

Author

Scquizzato, Elisa, primary, Zambello, Renato, additional, Baesso, Ilenia, additional, Varotto, Stefania, additional, Albergoni, Maria Paola, additional, Teramo, Antonella, additional, Boscaro, Elisa, additional, Cesaro, Simone, additional, Pillon, Marta, additional, Calore, Elisabetta, additional, Gazzola, Maria Vittoria, additional, Semenzato, Gianpietro, additional, Messina, Chiara, additional, and Trentin, Livio, additional

Published
2008
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40. Expression and role of CCR6/CCL20 chemokine axis in pulmonary sarcoidosis

Author

Facco, Monica, primary, Baesso, Ilenia, additional, Miorin, Marta, additional, Bortoli, Michela, additional, Cabrelle, Anna, additional, Boscaro, Elisa, additional, Gurrieri, Carmela, additional, Trentin, Livio, additional, Zambello, Renato, additional, Calabrese, Fiorella, additional, Cassatella, Marco Antonio, additional, Semenzato, Gianpietro, additional, and Agostini, Carlo, additional

Published
2007
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41. A Review on the Association between Glucagon-Like Peptide-1 Receptor Agonists and Thyroid Cancer.

Author

Fadini, Gian Paolo, Albiero, Mattia, Menegazzo, Lisa, Boscaro, Elisa, Agostini, Carlo, de Kreutzenberg, Saula Vigili, Rattazzi, Marcello, and Avogaro, Angelo

Subjects
DIABETES, CORONARY disease, PROGENITOR cells, OSTEOCALCIN genetics, PEOPLE with diabetes
Abstract

There is a concern on the risk of thyroid cancer associated with glucagon-like peptide-1 (GLP-1) analogs including liraglutide and exenatide. In this article, we review related experimental studies, clinical trials and observational human studies currently available. In rodents, liraglutide activated the GLP-1 receptors on C-cells, causing an increased incidence of C-cell neoplasia. Animal experiments with monkeys demonstrated no increase in calcitonin release and no C-cell proliferation after long-term liraglutide administration. Longitudinal 2-year data from clinical trials do not support any significant risk for the activation or growth of C-cell cancer in humans in response to liraglutide. However, an analysis of the FDA adverse event reporting system database suggested an increased risk for thyroid cancer associated with exenatide after its marketing. Noticeably, a recent study discovered that GLP-1 receptor could also be expressed in human papillary thyroid carcinomas (PTC), but the impact of GLP-1 analogs on PTC is not known. Therefore, GLP-1 analogs might increase the risk of thyroid C-cell pathology in rodents, but its risk in humans awaits confirmation. Since GLP-1 receptor is also expressed in PTC besides C-cells, it is important to investigate the actions of GLP-1 on different subtypes of thyroid cancer in the future. [ABSTRACT FROM AUTHOR]

Published
2012
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42. Low levels of endothelial progenitor cells correlate with disease duration and activity in patients with Behçet's disease

Author

GIAN PAOLO FADINI, Tognon, S., Rodriguez, L., Boscaro, Elisa, Baesso, I., ANGELO AVOGARO, Gianpietro Semenzato, Carlo Agostini, and Marcolongo, R.

Subjects
Adult, Male, Behcet Syndrome, Stem Cells, Endothelial Cells, Antigens, CD34, Flow Cytometry, Severity of Illness Index, Vascular Endothelial Growth Factor Receptor-2, Blood Cell Count, C-Reactive Protein, Antigens, CD, Case-Control Studies, Humans, AC133 Antigen, Peptides, Glycoproteins
Abstract

We tested whether Behçet's disease (BD) is characterized by alterations of circulating endothelial progenitor cells (EPCs),which are involved in vascular homeostasis and repair.We enrolled 30 BD patients and 27 matched healthy controls. EPCs were defined and measured by flow cytometry according to the expression of CD34, CD133 and KDR.We show that BD patients had significantly lower levels of CD34+KDR+ and CD34+CD133+KDR+ EPCs than controls. We found significant negative correlations between EPC phenotypes and BD duration, while there were positive correlations between CD34+KDR+ EPCs and both BD activity scores and C-reactive protein. The lower EPC levels with increasing disease duration was shown in univariate analysis and in multivariable analysis adjusted for possible confounders.This is the first report that BD is associated with progressive EPC decline. Reduction of EPCs may represent a mechanism of induction and/or progression of vascular injury in these patients.

43. Clinical significance of LAIR1 (CD305) as assessed by flow cytometry in a prospective series of patients with chronic lymphocytic leukemia.

Author

Perbellini O, Falisi E, Giaretta I, Boscaro E, Novella E, Facco M, Fortuna S, Finotto S, Amati E, Maniscalco F, Montaldi A, Alghisi A, Aprili F, Bonaldi L, Paolini R, Scupoli MT, Trentin L, Ambrosetti A, Semenzato G, Pizzolo G, Rodeghiero F, and Visco C

Subjects
Adult, Aged, Aged, 80 and over, Chromosome Aberrations, Disease Progression, Female, Flow Cytometry, Follow-Up Studies, Gene Expression, Humans, Immunoglobulin Heavy Chains genetics, Immunophenotyping, Leukemia, Lymphocytic, Chronic, B-Cell diagnosis, Leukemia, Lymphocytic, Chronic, B-Cell drug therapy, Leukemia, Lymphocytic, Chronic, B-Cell mortality, Male, Middle Aged, Mutation, Neoplasm Staging, Patient Outcome Assessment, Prognosis, Prospective Studies, Receptors, Immunologic genetics, Leukemia, Lymphocytic, Chronic, B-Cell metabolism, Receptors, Immunologic metabolism
Abstract

Most patients affected by chronic lymphocytic leukemia are diagnosed by flow cytometry. Several immunophenotypic markers have been identified as significant and independent prognostic variables, especially from retrospective cohorts. However, while attractive because their detection is inexpensive and feasible in most laboratories, only few have been validated by independent series. The expression of leukocyte-associated immunoglobulin-like receptor-1 (also known as LAIR1, LAIR-1 or CD305), an inhibitor of B-cell receptor-mediated signaling, has been reported to be lacking in high-risk chronic lymphocytic leukemia. However, its correlation with biological variables and its prognostic significance remain unknown. We investigated 311 consecutive patients, prospectively enrolled since 2007. Methods for studying patients were standardized and included clinical assessment, immunophenotype, fluorescence in situ hybridization, and status of immunoglobulin heavy chain variable region genes. Overall, 22.1% of patients had Binet stage B or C disease, 38.5% had unmutated immunoglobulin genes, 15.1% had high-risk cytogenetic abnormalities, 23.4% were CD38(+), 37.8% CD49d(+), and 59.8% LAIR1(+). Expression of LAIR1 was inversely related to that of CD38 (P=0.0005), but was not associated with CD49d expression (P=0.96). A significantly lower expression of LAIR1 was observed in patients with Binet stage B or C disease (P=0.023), and in the presence of high-risk cytogenetic abnormalities (P=0.048) or unmutated immunoglobulin heavy chain variable region genes (P<0.0001). At univariate analysis LAIR1(+) was significantly associated with longer time to first treatment (P=0.0002). This favorable effect of LAIR1(+) was confirmed by multivariate analysis (hazard ratio=2.1, P=0.03 for LAIR1). Our results indicate that LAIR1 expression is a reliable and inexpensive marker capable of independently predicting time to first treatment in newly diagnosed unselected patients with chronic lymphocytic leukemia.

Published
2014
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44. Effects of androgens on endothelial progenitor cells in vitro and in vivo.

Author

Fadini GP, Albiero M, Cignarella A, Bolego C, Pinna C, Boscaro E, Pagnin E, De Toni R, de Kreutzenberg S, Agostini C, and Avogaro A

Subjects
Adult, Animals, Aromatase metabolism, Blood Cell Count, Cell Adhesion drug effects, Cell Proliferation drug effects, Cells, Cultured, Dihydrotestosterone pharmacology, Dose-Response Relationship, Drug, Endothelial Cells physiology, Estradiol blood, Estradiol pharmacology, Humans, Male, Middle Aged, Orchiectomy, Phenotype, Rats, Rats, Sprague-Dawley, Receptors, Androgen metabolism, Stem Cells physiology, Testosterone blood, Testosterone pharmacology, Androgens pharmacology, Endothelial Cells drug effects, Stem Cells drug effects
Abstract

The beneficial or detrimental effects of androgens on the cardiovascular system are debated. Endothelial progenitor cells are bone-marrow-derived cells involved in endothelial healing and angiogenesis, which promote cardiovascular health. Oestrogens are potent stimulators of endothelial progenitor cells, and previous findings have indicated that androgens may improve the biology of these cells as well. In the present study, we show that testosterone and its active metabolite dihydrotestosterone exert no effects on the expansion and function of late endothelial progenitors isolated from the peripheral blood of healthy human adult males, whereas they positively modulate early 'monocytic' endothelial progenitor cells. In parallel, we show that castration in rats is followed by a decrease in circulating endothelial progenitor cells, but that testosterone and dihydrotestosterone replacement fails to restore endothelial progenitor cells towards normal levels. This is associated with persistently low oestrogen levels after androgen replacement in castrated rats. In a sample of 62 healthy middle-aged men, we show that circulating endothelial progenitor cell levels are more directly associated with oestradiol, rather than with testosterone, concentrations. In conclusion, our results collectively demonstrate that androgens exert no direct effects on endothelial progenitor cell biology in vitro and in vivo.

Published
2009
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