32 results on '"Borraz, Y."'
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2. CAN AST VALUES BE USED INSTEAD OF ALT TO CALCULATE THE PATTERN OF DRUG INDUCED LIVER INJURY (DILI)?: 16
3. Evaluation of Naranjo Adverse Drug Reactions Probability Scale in causality assessment of drug-induced liver injury
4. Hepatotoxicidad secundaria a 'productos naturales': análisis de los casos notificados al Registro Español de Hepatotoxicidad
5. Análisis de las causas, características y consecuencias de la reexposición al fármaco o compuesto responsable de un episodio de hepatotoxicidad
6. 1370 CAN AST VALUES BE USED INSTEAD OF ALT TO CALCULATE THE PATTERN OF DRUG INDUCED LIVER INJURY (DILI)?
7. 1378 EFFECT OF ABCC2 HAPLOTYPES ON INDIOSYNCRATIC DRUG-INDUCED LIVER INJURY (DILI) SUSCEPTIBILITY
8. 505 IBUPROFEN-ASSOCIATED HEPATOTOXICITY: ANALYSIS OF A CASE SERIES INCLUDED IN THE SPANISH DILI REGISTRY
9. 486 IDIOSYNCRATIC DRUG-INDUCED LIVER INJURY (DILI) IN PATIENTS WITH PRE-EXISTING LIVER DISEASE: AN ANALYSIS OF THE CASES INCLUDED IN THE SPANISH DILI REGISTRY
10. 1138 IDIOSYNCRATIC DRUG-INDUCED LIVER INJURY (DILI): FOLLOW-UP IN A COHORT OF PATIENTS IDENTIFIED IN 24 TERTIARY CARE CENTRES IN SPAIN
11. 1137 THE HLA CLASS I B*1801 ALLELE INFLUENCES HEPATOCELLULAR EXPRESSION OF AMOXICILLIN-CLAVULANATE LIVER DAMAGE AND OUTCOME IN SPANISH PATIENTS
12. 312 GENOTYPE DISTRIBUTION OF THE HEPATIC CANALICULAR TRANSPORTER ABCC2 – 24C>T POLYMORPHISM IS NOT RELATED TO DRUG-INDUCED IDIOSYNCRATIC LIVER INJURY (DILI). RELEVANCE OF ETHNIC VARIANTS IN EUROPEANS
13. 306 ASSOCIATION OF MANGANESE SUPEROXIDE DISMUTASE FUNCTIONAL POLYMORPHISM (SOD2 V16A) WITH IDIOSYNCRATIC DRUG-INDUCED LIVER INJURY
14. Hepatotoxicidad secundaria a "productos naturales": análisis de los casos notificados al Registro Español de Hepatotoxicidad
15. Análisis de las causas, características y consecuencias de la reexposición al fármaco o compuesto responsable de un episodio de hepatotoxicidad
16. 895 THE INFLUENCE OF AGE AND GENDER IN THE BIOCHEMICAL EXPRESSION OF IDIOSYNCRATIC LIVER INJURY
17. 896 THE NATURAL HISTORY OF IDIOSYNCRATIC DRUG-INDUCED LIVER INJURY (DILI): FOLLOW-UP IN A COHORT OF PATIENTS IDENTIFIED IN A SINGLE TERTIARY CARE CENTRE
18. 894 COMBINED GLUTATHIONE-S-TRANSFERASE M1 AND T1 GENETIC POLYMORPHISM AND SUSCEPTIBILITY TO DEVELOP DRUG-INDUCED IDIOSYNCRATIC LIVER INJURY (DILI)
19. 93 Amoxicillin-clavulanate (AC) associated hepatotoxicity: Analysis of a single country large cohort prospectively identified
20. Liver injuries induced by 'natural remedies': an analysis of cases submitted to the Spanish Liver Toxicity Registry | Hepatotoxicidad secundaria a 'productos naturales': Análisis de los casos notificados al Registro Español de Hepatotoxicidad
21. An Analysis of the causes, characteristics, and consequences of reexposure to a drug or compound responsible for a hepatotoxicity event | Análisis de las causas, características y consecuencias de la reexposición al fármaco o compuesto responsable de un episodio de hepatotoxicidad
22. An Analysis of the causes, characteristics, and consequences of reexposure to a drug or compound responsible for a hepatotoxicity event,Análisis de las causas, características y consecuencias de la reexposición al fármaco o compuesto responsable de un episodio de hepatotoxicidad
23. 684 EFFECTS OF CHEMICAL MOIETIES AND POLYMORPHISMS IN THE HUMAN ABC TRANSPORTER ABCB1 (P-GLYCOPROTEIN/MDR1) IN DRUG-INDUCED LIVER INJURY
24. 669 Chronic liver damage after an episode of idiosyncratic drug induced liver injury (DILI)
25. [Hepatotoxicity, a global problem with local features: toward the creation of a Pan-American Hepatotoxicity Network].
26. Mitochondrial superoxide dismutase and glutathione peroxidase in idiosyncratic drug-induced liver injury.
27. Phenotypic characterization of idiosyncratic drug-induced liver injury: the influence of age and sex.
28. [Liver injury induced by "natural remedies": an analysis of cases submitted to the Spanish Liver Toxicity Registry].
29. Glutathione S-transferase m1 and t1 null genotypes increase susceptibility to idiosyncratic drug-induced liver injury.
30. Analysis of IL-10, IL-4 and TNF-alpha polymorphisms in drug-induced liver injury (DILI) and its outcome.
31. [An analysis of the causes, characteristics, and consequences of reexposure to a drug or compound responsible for a hepatotoxicity event].
32. Outcome of acute idiosyncratic drug-induced liver injury: Long-term follow-up in a hepatotoxicity registry.
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