655 results on '"Bodmer, Walter F."'
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2. Acid-adapted cancer cells alkalinize their cytoplasm by degrading the acid-loading membrane transporter anion exchanger 2, SLC4A2
3. Sequencing of autosomal, mitochondrial and Y-chromosomal forensic markers in the People of the British Isles cohort detects population structure dominated by patrilineages
4. CRISPR-Cas9 screen identifies oxidative phosphorylation as essential for cancer cell survival at low extracellular pH
5. Genomic landscape of colorectal carcinogenesis
6. GMMchi: gene expression clustering using Gaussian mixture modeling
7. Polygenic inheritance, GWAS, polygenic risk scores, and the search for functional variants
8. Somatic selection of poorly differentiating variant stem cell clones could be a key to human ageing
9. Phenotypic screen of sixty-eight colorectal cancer cell lines identifies CEACAM6 and CEACAM5 as markers of acid resistance
10. Evolutionary perspectives on cancer and aging
11. List of contributors
12. Single-molecule DNA-mapping and whole-genome sequencing of individual cells
13. CRISPR-Cas9 screen identifies oxidative phosphorylation as essential for cancer cell survival at low extracellular pH
14. Effects of Threshold Choice on Biological Conclusions Reached during Analysis of Gene Expression by DNA Microarrays
15. Genetics of the human face : Identification of large-effect single gene variants
16. Multiple Rare Variants in Different Genes Account for Multifactorial Inherited Susceptibility to Colorectal Adenomas
17. The Eurasian Heartland: A Continental Perspective on Y-Chromosome Diversity
18. Antibody Targeting Studies in a Transgenic Murine Model of Spontaneous Colorectal Tumors
19. Alkaline-Mediated Differential Interaction (AMDI): A Simple Automatable Single-Nucleotide Polymorphism Assay
20. Spectral Karyotyping Suggests Additional Subsets of Colorectal Cancers Characterized by Pattern of Chromosome Rearrangement
21. Insulin-Like Growth Factor 1 Regulates the Location, Stability, and Transcriptional Activity of β -Catenin
22. Tumor Burden and Clonality in Multiple Intestinal Neoplasia Mouse/Normal Mouse Aggregation Chimeras
23. Transforming Growth Factor β Stimulation of Colorectal Cancer Cell Lines: Type II Receptor Bypass and Changes in Adhesion Molecule Expression
24. The APC Variants I1307K and E1317Q are Associated with Colorectal Tumors, but not Always with a Family History
25. Genetic Diversity and Disease Susceptibility
26. Dietary Fat Influences on Polyp Phenotype in Multiple Intestinal Neoplasia Mice
27. Expression of a Single-Chain HLA class I Molecule in a Human Cell Line: Presentation of Exogenous Peptide and Processed Antigen to Cytotoxic T Lymphocytes
28. Stromal uptake and transmission of acid is a pathway for venting cancer cell-generated acid
29. Myofibroblasts are distinguished from activated skin fibroblasts by the expression of AOC3 and other associated markers
30. Supplementary Table 5 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
31. Supplementary Table S3 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
32. Supplementary Figure 1 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
33. Supplementary Table 8 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
34. Supplementary Figure 3 from Stem Cell Differentiation and Lumen Formation in Colorectal Cancer Cell Lines and Primary Tumors
35. Data from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
36. Supplementary Figure 1 from Stem Cell Differentiation and Lumen Formation in Colorectal Cancer Cell Lines and Primary Tumors
37. Supplementary Figure Legends and Methods from Stem Cell Differentiation and Lumen Formation in Colorectal Cancer Cell Lines and Primary Tumors
38. Supplementary Table 9 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
39. Supplementary Figure 4 from Stem Cell Differentiation and Lumen Formation in Colorectal Cancer Cell Lines and Primary Tumors
40. Supplementary Data and Methods from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
41. Supplementary Figures S1-S6 from Dsh Homolog DVL3 Mediates Resistance to IGFIR Inhibition by Regulating IGF-RAS Signaling
42. Supplementary Figure Legend from Dsh Homolog DVL3 Mediates Resistance to IGFIR Inhibition by Regulating IGF-RAS Signaling
43. Supplementary Methods from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
44. Supplementary Methods and References from Dsh Homolog DVL3 Mediates Resistance to IGFIR Inhibition by Regulating IGF-RAS Signaling
45. Supplementary Table 4 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
46. Supplementary Figure 2 from Stem Cell Differentiation and Lumen Formation in Colorectal Cancer Cell Lines and Primary Tumors
47. Supplementary Tables S1-S8 from Dsh Homolog DVL3 Mediates Resistance to IGFIR Inhibition by Regulating IGF-RAS Signaling
48. Supplementary Table 7 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
49. Supplementary Table 6 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
50. Supplementary Table 3 from Colorectal Cancer Cell Lines Are Representative Models of the Main Molecular Subtypes of Primary Cancer
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