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32 results on '"Bichovsky, Yoav"'

24. Missile Embolism to the Pulmonary Artery

Author

Frenkel, Amit, primary, Shaked, Gad, additional, Shelef, Ian, additional, Sebbag, Gilbert, additional, Bichovsky, Yoav, additional, Brotfain, Evgeni, additional, Perry, Zvi H., additional, Koyfman, Leonid, additional, and Klein, Moti, additional

Published
2017
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26. Blood Glutamate Reducing Effect of Hemofiltration in Critically Ill Patients.

Author

Brotfain, Evgeni, Kutz, Ruslan, Grinshpun, Julia, Gruenbaum, Benjamin F., Gruenbaum, Shaun E., Frenkel, Amit, Zhumadilov, Agzam, Zeldetz, Vladimir, Bichovsky, Yoav, Boyko, Matthew, Klein, Moti, and Zlotnik, Alexander

Subjects
GLUTAMIC acid, NEUROTOXICOLOGY, BLOOD filtration, HEMODYNAMICS, CRITICALLY ill
Abstract

Glutamate toxicity plays a well-established role in secondary brain damage following acute and chronic brain insults. Previous studies have demonstrated the efficacy of hemodialysis and peritoneal dialysis in reducing blood glutamate levels. However, these methods are not viable options for hemodynamically unstable patients. Given more favorable hemodynamics, longer treatment, and less needed anticoagulation, we investigated whether hemofiltration could be effective in lowering blood glutamate levels. Blood samples were taken from 10 critically ill patients immediately before initiation of hemofiltration and after 1, 2, 4, 6, and 12 h, for a total of 6 blood samples. Samples were sent for determination of glutamate, glutamate oxaloacetate transaminase (GOT), glutamate pyruvate transaminase (GPT), hemoglobin, hematocrit, urea, creatinine, glucose, sodium, potassium, platelet, and white blood cell (WBC) levels. There was a statistically significant reduction in blood glutamate levels at all time points compared to baseline levels. There was no difference in levels of GOT or GPT. Hemofiltration can be a promising method of reducing blood glutamate levels, especially in critically ill patients where hemodialysis and peritoneal dialysis may be contraindicated. [ABSTRACT FROM AUTHOR]

Published
2018
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28. The Effects of Insulin, Glucagon, Glutamate, and Glucose Infusion on Blood Glutamate and Plasma Glucose Levels in Naive Rats

Author

Zlotnik, Alexander, primary, Gruenbaum, Benjamin Fredrick, additional, Klin, Yael, additional, Gruenbaum, Shaun Evan, additional, Ohayon, Sharon, additional, Sheiner, Eyal, additional, Kuts, Ruslan, additional, Boyko, Matthew, additional, Bichovsky, Yoav, additional, Shapira, Yoram, additional, and Teichberg, Vivian I., additional

Published
2011
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30. Timing of sugammadex administration: a case report.

Author

Frenkel, Amit, Roy-Shapira, Aviel, Zlotnik, Alexander, Brotfain, Evgeni, Koyfman, Leonid, Bichovsky, Yoav, and Klein, Moti

Subjects
*SUGAMMADEX, *DRUG administration, *MUSCLE relaxants, *ALLERGY treatment
Abstract

Sugammadex is a relatively new drug used to reverse the effects of rocuronium, a nondepolarizing muscle relaxing agent to hasten emergence from general anesthesia. Unlike neostigmine and atropine, its use is not associated with re-curarization or cardiac arrhythmias. Sugammadex carries a small risk of allergic reactions including anaphylactic shock. We present a case report of a 67 years old woman who underwent an urgent operation for small bowel obstruction. Due to atrial fibrillation (AF) the anesthesiologist administered Sugammadex just before skin closure. Soon after the injection, peak inspiratory pressures (PIP) increased precipitously followed by hypotension and increasing tachycardia. For anticipated cardioversion, the chest was exposed and it revealed urticarial. There was severe bronchospasm on auscultation. Treatment of anaphylactic shock was initiated, the patient improved dramatically and fully recovered. This case is presented to alert practitioners to the importance of a sudden rise in PIP after Sugammadex administration in the early diagnosis of an anaphylactic reaction, and to suggest that due to the risk of anaphylaxis, it may be advisable to initiate sugammadex only when the patient can be fully exposed without compromising the sterility of the operating field. [ABSTRACT FROM AUTHOR]

Published
2015

31. Ivabradine for Uncontrolled Sinus Tachycardia in Thyrotoxic Cardiomyopathy - Case Report.

Author

Frenkel A, Bichovsky Y, Arotsker N, Besser L, Joshua BZ, Fraenkel M, Zahger D, and Klein M

Subjects
Humans, Female, Adult, Ivabradine therapeutic use, Tachycardia, Sinus drug therapy, Tachycardia, Sinus etiology, Adrenergic beta-Antagonists therapeutic use, Adrenergic beta-Antagonists pharmacology, Heart Failure, Cardiomyopathies, Thyrotoxicosis
Abstract

Background: Beta-blockers, mainly propranalol, are usually administered to control heart rate in patients with thyrotoxicosis, especially when congestive heart failure presents. However, when thyrotoxicosis is not controlled, heart rate may be difficult to control even with maximal doses of propranolol. This presentation alerts physicians to the possibility of using ivabradine, a selective inhibitor of the sinoatrial pacemaker, for the control of heart rate., Case Presentation: We present a 37-year-old woman with thyrotoxicosis and congestive heart failure whose heart rate was not controlled with a maximal dose of beta blockers during a thyroid storm. The addition of ivabradine, a selective inhibitor of the sinoatrial pacemaker, controlled her heart rate within 48 hours., Conclusion: Ivabradine should be considered in patients with thyrotoxicosis, including those with heart failure, in whom beta blockers are insufficient to control heart rate., (Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.)

Published
2023
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32. WITHDRAWN: Ivabradine for Uncontrolled Sinus Tachycardia in Thyrotoxic Cardiomyopathy – Case Report

Author

Frenkel A, Bichovsky Y, Arotsker N, Besser L, Joshua BZ, Fraenkel M, Zahger D, and Klein M

Abstract

Since the authors are not responding to the editor’s requests to fulfill the editorial requirement, therefore, the article has been withdrawn by the publisher., Bentham Science apologizes to the readers of the journal for any inconvenience this may have caused., The Bentham Editorial Policy on Article Withdrawal can be found at https://benthamscience.com/editorial-policies-main.php, Bentham Science Disclaimer: It is a condition of publication that manuscripts submitted to this journal have not been published and will not be simultaneously submitted or published elsewhere. Furthermore, any data, illustration, structure or table that has been published elsewhere must be reported, and copyright permission for reproduction must be obtained. Plagiarism is strictly forbidden, and by submitting the article for publication the authors agree that the publishers have the legal right to take appropriate action against the authors, if plagiarism or fabricated information is discovered. By submitting a manuscript the authors agree that the copyright of their article is transferred to the publishers if and when the article is accepted for publication., (Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net)

Published
2021
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