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By calcination, a sulfur vacancy rich CdS based composite photocatalyst with graphitic carbon nitride (g-C3N4) as a matrix has been synthesized successfully from a tetranuclear Cd–S cluster assembled supramolecular network. In this photocatalyst (CdS@g-C3N4), CdS nanoparticles with a size of about 5 to 8 nm disperse homogenously in the g-C3N4 matrix. During calcination, some coordinated nitrogen atoms dope in the lattice of CdS and replace sulfur atoms, which generates a large number of sulfur vacancies. Under visible light irradiation, CdS@g-C3N4 exhibits excellent H2 production activity with a rate achieving as high as 19.88 mmol g−1 h−1 in the absence of a Pt cocatalyst. Its H2 production ability remains stable for 30 h, which does not decay. Besides H2 production, CdS@g-C3N4 also shows excellent photocatalytic activity for Volatile Organic Compound (VOC) degradation. For a photocatalyst, chemical content plays an important role in its performance. Here, the influence of sulfur vacancies on H2 production and VOC degradation is discussed in detail. We expect that the sulfur vacancy rich CdS@g-C3N4 can act as an efficient material for H2 production and indoor air purification. [ABSTRACT FROM AUTHOR]

Background: Age is the most important risk factor for cancer, but aging rates are heterogeneous across individuals. We explored a new measure of aging-Phenotypic Age (PhenoAge)-in the risk prediction of site-specific and overall cancer., Methods: Using Cox regression models, we examined the association of Phenotypic Age Acceleration (PhenoAgeAccel) with cancer incidence by genetic risk group among 374,463 participants from the UK Biobank. We generated PhenoAge using chronological age and nine biomarkers, PhenoAgeAccel after subtracting the effect of chronological age by regression residual, and an incidence-weighted overall cancer polygenic risk score (CPRS) based on 20 cancer site-specific polygenic risk scores (PRSs)., Results: Compared with biologically younger participants, those older had a significantly higher risk of overall cancer, with hazard ratios (HRs) of 1.22 (95% confidence interval, 1.18-1.27) in men, and 1.26 (1.22-1.31) in women, respectively. A joint effect of genetic risk and PhenoAgeAccel was observed on overall cancer risk, with HRs of 2.29 (2.10-2.51) for men and 1.94 (1.78-2.11) for women with high genetic risk and older PhenoAge compared with those with low genetic risk and younger PhenoAge. PhenoAgeAccel was negatively associated with the number of healthy lifestyle factors (Beta = -1.01 in men, p<0.001; Beta = -0.98 in women, p<0.001)., Conclusions: Within and across genetic risk groups, older PhenoAge was consistently related to an increased risk of incident cancer with adjustment for chronological age and the aging process could be retarded by adherence to a healthy lifestyle., Funding: This work was supported by the National Natural Science Foundation of China (82230110, 82125033, 82388102 to GJ; 82273714 to MZ); and the Excellent Youth Foundation of Jiangsu Province (BK20220100 to MZ)., Competing Interests: LB, ZM, XF, CJ, TW, CY, JD, HM, ZH, HS, LW, MZ, GJ No competing interests declared, (© 2024, Bian, Ma et al.)