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4. Urinary complement proteins are increased in children with IgA vasculitis (Henoch-Schönlein purpura) nephritis

5. P2RX7 gene variants associate with altered inflammasome assembly and reduced pyroptosis in chronic nonbacterial osteomyelitis (CNO)

6. Towards stratified treatment of JIA: machine learning identifies subtypes in response to methotrexate from four UK cohorts

7. Real world treatment of juvenile-onset systemic lupus erythematosus: Data from the UK JSLE cohort study

11. Towards stratified treatment of JIA: machine learning identifies subtypes in response to methotrexate from four UK cohorts

13. Kidney outcomes for children with lupus nephritis

14. Patient-reported wellbeing and clinical disease measures over time captured by multivariate trajectories of disease activity in individuals with juvenile idiopathic arthritis in the UK: a multicentre prospective longitudinal study

18. Contribution of rare and predicted pathogenic gene variants to childhood-onset lupus: a large, genetic panel analysis of British and French cohorts

28. OA39 Development of a Childhood Lupus Low Disease Activity State definition: recommendations from the International Childhood Lupus Treat-to-Target Task Force

31. Stuck in transit: A qualitative study of the transitional care needs of young people with epilepsy and juvenile idiopathic arthritis.

32. Management and treatment of children, young people and adults with systemic lupus erythematosus: British Society for Rheumatology guideline scope.

43. Development of a consensus core dataset in juvenile dermatomyositis for clinical use to inform research

44. European evidence-based recommendations for the diagnosis and treatment of childhood-onset lupus nephritis: the SHARE initiative

45. European evidence-based recommendations for diagnosis and treatment of childhood-onset systemic lupus erythematosus: the SHARE initiative

46. Successful stopping of biologic therapy for remission in children and young people with juvenile idiopathic arthritis.

50. Panel sequencing links rare, likely damaging gene variants with distinct clinical phenotypes and outcomes in juvenile-onset SLE

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