21 results on '"Bennett, Stuart J."'
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2. Regulation of T-Cell Functions by Oxidative Stress
3. Healthy ageing and depletion of intracellular glutathione influences T cell membrane thioredoxin-1 levels and cytokine secretion
4. A Role for Epigenetic Modulation of the Innate Immune Response During Aging
5. Does metabolic reprogramming underpin age-associated changes in T cell phenotype and function?
6. Regulation of T-Cell Functions by Oxidative Stress
7. Revealing 4-hydroxynonenal-histidine adducts as qualitative and quantitative biomarker of oxidative stress, lipid peroxidation and oxidative homeostasis
8. Intracellular redox state modulates the T cell surface proteome – relevance to ageing
9. Age‐associated changes in long‐chain fatty acid profile during healthy aging promote pro‐inflammatory monocyte polarization via PPARγ
10. Age-associated changes in long-chain fatty acid profile during healthy aging promote pro-inflammatory monocyte polarization via PPARγ
11. CD4+ T cell surface alpha enolase is lower in older adults
12. Ankyrin is the major oxidised protein in erythrocyte membranes from end-stage renal disease patients on chronic haemodialysis and oxidation is decreased by dialysis and vitamin C supplementation
13. CD4+ T cell surface alpha enolase is lower in older adults
14. Thioredoxin as a putative biomarker and candidate target in age-related immune decline
15. Ultra-endurance exercise: unanswered questions in redox biology and immunology
16. Measurement of HNE-protein adducts in human plasma and serum by ELISA-Comparison of two primary antibodies
17. Healthy ageing and depletion of intracellular glutathione influences T cell membrane thioredoxin-1 levels and cytokine secretion
18. The antioxidant enzyme peroxiredoxin-2 is depleted in lymphocytes seven days after ultra-endurance exercise
19. Measurement of HNE-protein adducts in human plasma and serum by ELISA—Comparison of two primary antibodies
20. Free radicals and redox signalling in T-cells during chronic inflammation and ageing
21. Age-associated changes in long-chain fatty acid profile during healthy aging promote pro-inflammatory monocyte polarization via PPARγ.
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