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1. Comparison of two plasma p-tau217 assays to detect and monitor Alzheimer’s pathology

3. Blood phosphorylated tau 181 as a biomarker for Alzheimer's disease: a diagnostic performance and prediction modelling study using data from four prospective cohorts

4. Sex modulates the role of astrocyte reactivity in preclinical Alzheimer’s disease

5. Synaptic dysfunction, Tau pathology and Neurodegeneration

6. CSF total tau is more closely associated with synaptic dysfunction than overt neurodegeneration

7. Association of reactive astrogliosis and microglial activation with tau pathology in Alzheimer’s disease

8. Increased regional brain inflammation predicts longitudinal brain atrophy in individuals in the Alzheimer’s disease continuum

9. Plasma p‐tau and brain‐derived total tau biomarkers predict longitudinal changes in Aβ, tau, and cognition across the AD continuum

10. Neuroinflammation Exacerbates Irritability and Agitation in Alzheimer’s Disease

11. Amyloid β‐dependent tau phosphorylation is triggered by reactive astrocytes in preclinical Alzheimer’s disease

12. Plasma biomarkers as stand‐alone tests in the diagnosis of Alzheimer’s disease

13. Association between glial and synaptic fluid biomarkers and brain [18F]FDG‐PET signal

14. APOEε4 potentiates the effects of Aβ pathology on the deposition of neurofibrillary tangles via tau phosphorylation

15. Interactions of synaptic and inflammatory biomarkers in Alzheimer’s Disease

16. Amyloid and Tau Predominance Subtyping Identifies CI Patients With Different Clinical Phenotypes

17. Fast accumulators of tau have higher levels of plasma pTau and stronger associations with amyloid in later Braak regions at baseline

18. Association between brain [18F]FDG‐PET signal and genetic cell markers in the brain tissue

19. The impact of young controls in the detection of tau load in cognitively impaired and asymptomatic elderly

20. Plasma biomarkers rates of change across the preclinical stage of Alzheimer’s disease: a longitudinal study

21. Microglial activation interacts with amyloid‐β to drive longitudinal tau tangle accumulation and cognitive decline

23. Astrocyte reactivity is associated with synaptic dysfunction across the aging and Alzheimer’s disease spectrum, whereas microglial reactivity is specifically associated with synaptic dysfunction related to cognitive impairment

24. Potential utility of using both APOEε4 and Aβ positivity to enrich clinical trials of tau‐targeting therapies

25. Sex impacts the association of plasma Glial Fibrillary Acidic Protein with neurodegeneration in Alzheimer’s disease

26. Amyloid‐dependent tau phosphorylation drives faster accumulation of tau aggregates in female

27. Plasma biomarkers of tau for the differentiation between slow and fast tau‐PET accumulators

28. Microglial Activation Contributes to Neuropsychiatric Dysfunction in Alzheimer’s Disease

29. Synapse dysfunction and astrocyte reactivity are associated independently of amyloid‐β and tau pathologies

30. Exploring inflammation‐related protein expression and its relationship with TSPO PET in Alzheimer

31. Plasma p‐tau181 and nfl as surrogates biomarkers in clinical trials targeting preclinical stage of Alzheimer’s disease

32. Associations between brain TSPO and [18F]FDG‐PET signals in neurodegenerative disorders

33. Synaptic dysfunction in the presence of tau tangles is not a strong predictor of atrophy

34. Plasma p‐tau231 and p‐tau217 provides information on tau tangle deposition in symptomatic Alzheimer’s disease individuals

35. Biomarkers disclose associations between Neuroinflammation and Synaptic Depletion in AD.

36. Plasma ALZpath p‐tau217 for the identification of amyloid and tau positivity

37. Discriminative accuracy of the A/T/N scheme to identify cognitive impairment due to Alzheimer's disease

38. Plasma GFAP in presymptomatic and symptomatic familial Alzheimer’s disease: a longitudinal cohort study

39. Late‐life hypertension acts together with amyloid‐β pathology to promote early cognitive decline.

40. Acute sleep loss increases CNS health biomarkers and compromises the ability to stay awake in a sex-and weight-specific manner.

41. Amyloid processing in COVID ‐19‐associated neurological syndromes

42. Performance of plasma amyloid, tau, and astrocyte biomarkers to identify cerebral AD pathophysiology

43. Author response: Impact of long- and short-range fibre depletion on the cognitive deficits of fronto-temporal dementia

44. Plasma GFAP in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study.

46. Associations between brain TSPO and [18F]FDG‐PET signals in neurodegenerative disorders.

47. Association between brain [18F]FDG‐PET signal and genetic cell markers in the brain tissue.

48. P2-346: TAU AND AMYLOID PATHOLOGY SUPPRESS GLOBAL BRAIN FUNCTIONAL CONNECTIVITY

49. IC-P-070: ASSOCIATIONS BETWEEN PLASMA NFL AND BRAIN PET IN THE ALZHEIMER'S DISEASE

50. IC‐P‐182: AMYLOID‐DEPENDENT AND AMYLOID‐INDEPENDENT EFFECTS OF TAU ON CLINICAL STATUS

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