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2. Impact of concomitant fibrates on immunotherapy outcomes for advanced non-small cell lung cancer.

9. The Integrated Genomic Landscape of Thymic Epithelial Tumors

11. Comprehensive and Integrated Genomic Characterization of Adult Soft Tissue Sarcomas

15. Treatment Guidance for Patients With Lung Cancer During the Coronavirus 2019 Pandemic

17. Genomic Classification of Cutaneous Melanoma

22. MERTK activation drives osimertinib resistance in EGFR-mutant non-small cell lung cancer

23. Class I HDACs Are Mediators of Smoke Carcinogen–Induced Stabilization of DNMT1 and Serve as Promising Targets for Chemoprevention of Lung Cancer

25. P-171 The changing landscape of peri-transplant adverse events with HDT/ASCT and the impact on long term outcomes for myeloma patients

27. The Cancer Genome Atlas Comprehensive Molecular Characterization of Renal Cell Carcinoma

28. Spatial Organization and Molecular Correlation of Tumor-Infiltrating Lymphocytes Using Deep Learning on Pathology Images

29. Molecular Characterization and Clinical Relevance of Metabolic Expression Subtypes in Human Cancers

30. Pan-Cancer Analysis of lncRNA Regulation Supports Their Targeting of Cancer Genes in Each Tumor Context

31. Genomic, Pathway Network, and Immunologic Features Distinguishing Squamous Carcinomas

32. Integrated Genomic Analysis of the Ubiquitin Pathway across Cancer Types

33. Machine Learning Detects Pan-cancer Ras Pathway Activation in The Cancer Genome Atlas

34. Systematic Analysis of Splice-Site-Creating Mutations in Cancer

35. Driver Fusions and Their Implications in the Development and Treatment of Human Cancers

36. Genomic and Molecular Landscape of DNA Damage Repair Deficiency across The Cancer Genome Atlas

37. Somatic Mutational Landscape of Splicing Factor Genes and Their Functional Consequences across 33 Cancer Types

46. Shifting Sociodemographic Characteristics of a Phase I Clinical Trial Population at an NCI-Designated Comprehensive Cancer Center in the Southeast.

49. Supplementary Figures 1 - 10 from Class I HDACs Are Mediators of Smoke Carcinogen–Induced Stabilization of DNMT1 and Serve as Promising Targets for Chemoprevention of Lung Cancer

50. Data from Class I HDACs Are Mediators of Smoke Carcinogen–Induced Stabilization of DNMT1 and Serve as Promising Targets for Chemoprevention of Lung Cancer

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