Search

This research investigates the viability of high-strength Recycled Concrete Aggregate (RCA) sourced from demolished structures containing high-strength concrete as a substitute for natural fine aggregates (NA) in cementitious mortar applications. Concrete specimens (40 × 40 × 160 mm) were prepared in a controlled environment with varying percentages of RCA replacing NA, ranging from 0% to 100% in 10% increments. The resulting RCA aggregates exhibited lower weight for sizes from 0.01 to 1 mm compared to NA, and for 1 to 3 mm sizes, RCA weights were 145% to 177% higher than SS aggregates. After curing for 28 days, flexural and compressive strength tests were conducted on the batches. The average compressive strength for the 0% RCA batch was 66.26 MPa, while the 50% RCA batch showed the closest average compressive strength at 63.10 MPa. Batches with varying RCA levels displayed compressive strengths between 49.52 and 58.18 MPa. The highest flexural strength was observed in the 0% RCA batch, with the closest result for a batch containing RCA being the 50% RCA batch. [ABSTRACT FROM AUTHOR]

This paper presents a second-order sensitivity analysis for a smart structural system formulated in terms of multiobjective optimization where structural design variables and control parameters are equally treated as design variables. The analytical derivatives of various objectives and constraints are obtained. These expressions provide a complete sensitivity package for the multidisciplinary analysis and optimization problem. An example truss regulated using four actuators is presented as verification of the derivations. This includes the optimal designs achieved using sequential linear and quadratic programming methods. Numerical results show the advantages of using high-order derivatives for the solution of a problem of such complexity and confirm that a systematic sensitivity analysis is essential, as the performance of the controller relies heavily on the structural design variables as well as feedback control logic.

Monetary policy rarely accomplished stabilization if sound fiscal policy had not already been established. Distinctions between different nominal anchors can be exaggerated. Any credit crunch owed more to micro distortions [...]

Spectrin has been implicated in a variety of different processes during late embryogenesis, after transcription of the zygotic genome has been activated. However, relatively little is known about the role of maternally derived spectrin during the early cleavage divisions that give rise to a multicellular embryo. To investigate the role of spectrin in early development, we have microinjected anti-spectrin antibodies into Patiria miniata starfish embryos to inhibit the activity of the maternal pool of spectrin. Microinjection of affinity-purified anti-spectrin antibody, or low to moderate doses of [F.sub.ab] fragments, into one blastomere of a two-cell-stage embryo caused a dose-dependent, progressive increase in the length of the cell cycle compared to the uninjected control blastomere. The progeny of injected blastomeres were unable to participate in the formation of a blastula epithelium, instead forming a loose aggregate of cells that eventually stopped dividing. When division stopped, the cells formed surface protrusions and became motile. At high doses of either whole antibody or [F.sub.ab] fragments, cells initiated, but failed to complete, cytokinesis. Blastomeres injected with high doses of [F.sub.ab] fragments also failed to reform nuclei and underwent variable periods of cell cycle arrest up to 12 hr. Injected embryos stained with BODIPY-phallacidin exhibited extensive disruption of the cortical actin cytoskeleton. These results support previous studies implicating spectrin in stabilizing the cell surface and maintaining the organization of the cortical cytoskeleton. They further suggest that spectrin is not required for the initiation or contraction of the cleavage furrow, but functions in the completion of cytokinesis. Most surprisingly, however, the results demonstrate that inhibition of spectrin function alters cell cycle timing, suggesting that disruption of the actin cytoskeleton inhibits progression through the cell cycle.