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1. Liver cell circuits and therapeutic discovery for advanced liver disease and cancer

2. An atlas of the human liver diurnal transcriptome and its perturbation by hepatitis C virus infection

11. Targeting the liver clock improves fibrosis by restoring TGF-β signaling

13. Hepatitis D virus interferes with hepatitis B virus RNA production via interferon-dependent and -independent mechanisms

14. Treatment of HCC with claudin-1-specific antibodies suppresses carcinogenic signaling and reprograms the tumor microenvironment

16. IL-22 regulates MASTL expression in intestinal epithelial cells.

17. Early hepatocellular carcinoma detection using magnetic resonance imaging is cost-effective in high-risk patients with cirrhosis

18. Comparative analysis of human, rodent and snake deltavirus replication

20. Erratum to ‘JAK1 promotes HDV replication and is a potential target for antiviral therapy’ [J Hepatol 80 (2024) 220-231]

22. The circadian clock component BMAL1 regulates SARS-CoV-2 entry and replication in lung epithelial cells

24. A blood-based prognostic liver secretome signature and long-term hepatocellular carcinoma risk in advanced liver fibrosis

25. Tight Junction Proteins as Therapeutic Targets to Treat Liver Fibrosis and Hepatocellular Carcinoma.

26. Stromal and Immune Drivers of Hepatocarcinogenesis

33. Joint statement in support of hepatitis C human challenge studies

34. JAK1 promotes HDV replication and is a potential target for antiviral therapy

35. Circadian control of hepatitis B virus replication

36. A human liver cell-based system modeling a clinical prognostic liver signature for therapeutic discovery

38. Follicular T helper cells shape the HCV-specific [CD4.sup.+] T cell repertoire after virus elimination

40. Combined Analysis of Metabolomes, Proteomes, and Transcriptomes of Hepatitis C Virus–Infected Cells and Liver to Identify Pathways Associated With Disease Development

44. Novel non-HAP class A HBV capsid assembly modulators have distinct in vitro and in vivo profiles

45. HBV Bypasses the Innate Immune Response and Does Not Protect HCV From Antiviral Activity of Interferon

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