269 results on '"Baukrowitz, Thomas"'
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2. Atomistic mechanism of coupling between cytosolic sensor domain and selectivity filter in TREK K2P channels
3. Extracellular modulation of TREK-2 activity with nanobodies provides insight into the mechanisms of K2P channel regulation
4. Gain-of-function mutations in KCNK3 cause a developmental disorder with sleep apnea
5. An otopetrin family proton channel promotes cellular acid efflux critical for biomineralization in a marine calcifier
6. Ion occupancy of the selectivity filter controls opening of a cytoplasmic gate in the K2P channel TALK-2.
7. Selectivity filter instability dominates the low intrinsic activity of the TWIK-1 K2P K+ channel
8. Ion occupancy of the selectivity filter controls opening of a cytoplasmic gate in the K2P channel TALK-2
9. Extracellular modulation of TREK-2 activity with nanobodies provides insight into the mechanisms of K2P channel regulation
10. From head to tail - Atomistic mechanism of long-range coupling from the cytosolic sensor domain to the selectivity filter in TREK K2Pchannels
11. Functional Conversion between A-Type and Delayed Rectifier K + Channels by Membrane Lipids
12. Bilayer-Mediated Structural Transitions Control Mechanosensitivity of the TREK-2 K2P Channel
13. PIP$_2$ and PIP as Determinants for ATP Inhibition of K$_{ATP}$ Channels
14. Two Functionally Distinct Subsites for the Binding of Internal Blockers to the Pore of Voltage-Activated K + Channels
15. Use-Dependent Blockers and Exit Rate of the Last Ion from the Multi-Ion Pore of a K$^+$ Channel
16. A Non-canonical Voltage-Sensing Mechanism Controls Gating in K2P K+ Channels
17. Validation of TREK1 ion channel activators as an immunomodulatory and neuroprotective strategy in neuroinflammation
18. Sodium permeable and “hypersensitive” TREK‐1 channels cause ventricular tachycardia
19. State-Dependent Network Connectivity Determines Gating in a K+ Channel
20. KCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel function
21. A Specific Two-pore Domain Potassium Channel Blocker Defines the Structure of the TASK-1 Open Pore
22. From head to tail—atomistic mechanism of long-range coupling from the cytosolic sensor domain to the selectivity filter in TREK K2P channels
23. Dynamic interplay of the proximal C-terminus and selectivity filter reveals gating and pharmacological modulation mechanisms in TREK K2P channels
24. 5-Hydroxydecanoate and coenzyme A are inhibitors of native sarcolemmal KATP channels in inside-out patches
25. The versatile regulation of K2P channels by polyanionic lipids of the phosphoinositide and fatty acid metabolism
26. Side pockets provide the basis for a new mechanism of Kv channel–specific inhibition
27. Defective X-gating caused byde novogain-of-function mutations inKCNK3underlies a developmental disorder with sleep apnea
28. The versatile regulation of K2P channels by polyanionic lipids of the phosphoinositide (PIP2) and fatty acid metabolism (LC-CoA)
29. Identification of the muscarinic pathway underlying cessation of sleep-related burst activity in rat thalamocortical relay neurons
30. The pore structure and gating mechanism of K2P channels
31. RNA editing modulates the binding of drugs and highly unsaturated fatty acids to the open pore of Kv potassium channels
32. Norfluoxetine inhibits TREK-2 K2P channels by multiple mechanisms including state-independent effects on the selectivity filter gate
33. H Bonding at the Helix-Bundle Crossing Controls Gating in Kir Potassium Channels
34. Structural and functional analysis of the putative pH sensor in the Kir1.1 (ROMK) potassium channel
35. Structural Basis for Gating of the Two-Pore Domain K+ (K2P) Channels TASK-1 and TALK-2
36. Molecular basis of inward rectification: polyamine interaction sites located by combined channel and ligand mutagenesis
37. Functional conversion between A-type and delayed rectifier [K.sup.+] channels by membrane lipids
38. Activation of the alkaline-activated K2P K+ channel talk-2 requires opening of two functionally coupled gates
39. Inward rectification in KATP channels: a pH switch in the pore
40. Multiple Mechanisms Underlie State-Independent Inhibitory Effects of Norfluoxetine on TREK-2 K2P Channels
41. Characterisation of the Versatile Gating Behaviour in Talk-2 K2P Channels
42. Random mutagenesis screening indicates the absence of a separate H+-sensor in the pH-sensitive Kir channels
43. Long Chain CoA Esters as Competitive Antagonists of Phosphatidylinositol 4,5-Bisphosphate Activation in Kir Channels
44. Cytoplasmic accumulation of long-chain coenzyme A esters activates KATP and inhibits Kir2.1 channels
45. Two functionally distinct subsites for the binding of internal blockers to the pore of voltage-activated K+ channels
46. The inward rectification mechanism of the HERG cardiac potassium channel
47. Phosphatidylinositol 4,5-Bisphosphate (PIP2) Modulation of ATP and pH Sensitivity in Kir Channels: A TALE OF AN ACTIVE AND A SILENT PIP2 SITE IN THE N TERMINUS
48. Selectivity filter instability dominates the low intrinsic activity of the TWIK-1 K2P K+ Channel
49. The molecular basis for an allosteric inhibition of K+-flux gating in K2P channels
50. A pharmacological master key mechanism that unlocks the selectivity filter gate in K + channels
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