21 results on '"Bassi, Rekha"'
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2. HEME OXYGENASE-1 IS PARTIALLY RESPONSIBLE FOR DECREASED VASCULAR CONTRACTILITY MEDIATED BY ENDOTOXIN
3. The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules
4. Redox-dependent dimerization of p38α mitogen-activated protein kinase with mitogen-activated protein kinase kinase 3
5. The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules
6. TAB1-Induced Autoactivation of p38α Mitogen-Activated Protein Kinase Is Crucially Dependent on Threonine 185
7. Redox-dependent dimerization of p38α mitogen-activated protein kinase with mitogen-activated protein kinase kinase 3
8. TAB1-Induced Autoactivation of p38α Mitogen-Activated Protein Kinase Is Crucially Dependent on nThreonine 185.
9. Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1
10. Abstract 32: Investigating the Autoactivation of p38α Mitogen-Activated Protein Kinase Mediated by Transforming Growth Factor-β-Activated Protein Kinase Binding Protein 1
11. Abstract 336: TAB1 Initiates p38α Autophosphorylation During Myocardial Ischemia
12. A Chemical Genetic Approach Reveals That p38α MAPK Activation by Diphosphorylation Aggravates Myocardial Infarction and Is Prevented by the Direct Binding of SB203580
13. Mechanisms of MAPK kinase-independent p38-MAPK activation
14. Diverse Mechanisms of Myocardial p38 Mitogen-Activated Protein Kinase Activation
15. Endothelial Heme Oxygenase-1 Induction by Hypoxia
16. Curcumin, an antioxidant and anti-inflammatory agent, induces heme oxygenase-1 and protects endothelial cells against oxidative stress
17. Varying susceptibility to myocardial infarction among C57BL/6 mice of different genetic background
18. Inhibition of p38 MAPK activity fails to attenuate contractile dysfunction in a mouse model of low-flow ischemia
19. Abstract 336.
20. Abstract 32.
21. Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1.
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